[CTRL] Sam Moser-The Shocking Truth About Psychiatric Drugs

[Amelia]

-Caveat Lector-

I ran across this article from last year posted to the list by Sam Moser
himself and thought some might want to read it.  It is excellent and rather
long.
Ake
----- Original Message -----
From: "Sam Moser" <[EMAIL PROTECTED]>
To: <[EMAIL PROTECTED]>
Sent: Thursday, April 08, 1999 2:30 AM
Subject: [CTRL] The Shocking Truth About Psychiatric Drugs


> -Caveat Lector-
>
> I have many new references that I will be adding soon to strengthen the
> arguments made.
>
> Psychiatry Kills
> Documented Proof Psychiatric Drugs Shorten Life Span
>
> Version 2.007d
>
> By Samuel Antoine Moser
>
> No longer is involuntary drugging only a concern to those locked away in
an
> institution. "Laws quietly passed in 36 US states now allow the government
> to court order you to take psychiatric drugs, even though you're law
abiding
> and living at home, in your own neighborhood. These court orders are known
> as "Involuntary Outpatient Commitment" (IOC). Typically, you'd be required
> to report to your community mental health center every few weeks for a
> "depot injection" of a "neuroleptic drug" such as Prolixin or Haldol in
your
> butt. These drugs are time released [in a base of oil and injected
> intramuscularly], so that the super-powerful impact lasts weeks until your
> next injection. The court orders can be routinely re-approved, so these
> injections can go on for years." Reports Support Coalition International
> http://www.efn.org/~dendron/ .
>
> If this report came to you incomplete then email me now at [EMAIL PROTECTED]
or
> [EMAIL PROTECTED] and I will send you the complete report and
please
> let me know if you are able to open attachments and if you have MS Word or
> Word Pad and if your E-mail program supports Rich Text. Also from this
point
> onward I will be labeling new releases of this report with a version
number
> starting with version 2. If you liked this report and would like to get
the
> latest version then E-mail me at any time at any of the E-mail addresses
> provided. This is a work in progress.
>
> Disclaimer: Many of the statements that follow in this report (Namely the
> effect that neuroleptic drugs have on the duration of life span and sense
of
> well being.) have not been investigated or evaluated by the FDA (so they
> claim) nor do they care to. The FDA does not even require animal studies
to
> determine how these drugs affect the duration of life span which is
shocking
> considering that the FDA is supposed to be charged with protecting the
> American people. The conclusions in this report are based on available
> facts, for which a positive assertion can be made by putting the
information
> together so that the true nature of these drugs can be established. This
> information is what the FDA, psychiatrists, and the pharmaceutical
companies
> do NOT WANT YOU TO KNOW. This information is both shocking and scandalous
> and reveals a probable conspiracy so be warned. The common people that are
> affected by this are not supposed to be smart enough to understand and
> figure it out, but I have.
>
> The purpose of this report is to prove conclusively that neuroleptic drugs
> (Also know as antipsychotics or antipsychotic drugs, tranquilizers,
> psychotropics or psychotrophics, or psychotropic drugs or psychotrophic
> drugs) shorten life span, destroy sexual function and fertility, take away
> sense of well being which can precipitate suicide and or violent behavior
> etc. This will be established in this discussion through a rudimentary
> explanation of some facets of biochemistry and by quoting reputable
sources.
> Also to empower the people who may be affected by this with knowledge that
> they can use to defend their rights and help overturn these laws as
> unconstitutional and as basic human rights violations. Also to help
educate
> people who have friends and or family members who are affected by this and
> to provide understandable information to the general public who are being
> denied the facts.
>
> These drugs are not just given to those labeled as "schizophrenic" or said
> to be "psychotic" or have "psychosis" or have "schizophrenia". These drugs
> are often given to people with depression, manic depression or bipolar
> disorder, mania, Alzheimer's patients or those suffering from Alzheimer's
> Disease, people with brain damage or head injuries, retarded children and
> adults, many children with Attention Deficit Disorder or ADD or ADHD and
> children and teenagers who simply have a hot temper (which ironically the
> drugs will make worse) as well as many other things. If you are unsure
> whether a certain drug is a neuroleptic or not then here is a list of many
> names for neuroleptic drugs.
>
> List of neuroleptic drug names: Chlorpromazine, (Brand names are as
follows;
> Chlor-PZ, Klorazine, Promachlor, Promapar, Sonazine, Thorazine, Chlorprom,
> Chlor-Promanyl and Largactil) Fluphenazine, (Brand names are as follows;
> Permitil, Prolixin, Modecate, Moditen) Mesoridazine Besylate, (Brand name
is
> Serentil) Perphenazine, (Brand names are as follows; Trilafon, Etrafon,
> Triavil, Phenazine and Etrafon) Prochlorperazine, (Brand names are as
> follows; Compazine and Stemetil) Promazine Hydrocloride, (Brand name
> Sparine) Thioridazine, (Brand names are Mellaril, Novoridazine and
Thioril)
> Trifuoperazine, (Brand names are as follows; Stelazine, Clinazine,
> Novaflurazine, Pentazine, Terfluzine and Triflurin) Clozapine, (Brand
named
> Clozaril, in Germany called Leponex) Haloperadol, (Brand name Haldol)
> Loxapine, (Brand name Loxitane) Pimozide (Brand name is Orap) Thiothixene,
> (Brand name Navane) Risperidone, Zyprexa (Brand name is Olanzapine),
> Sertindole, Ziprasidone, Amperozide, Remoxipride, Melperone, Zotepine,
> Isofloxythepin, Setoperone, Perospirone, Quetiapine. Also the
antidepressant
> drug Sertraline (Brand name Zoloft) is molecularly indistinguishable from
> that of the neuroleptics and has even been tried as a neuroleptic. This
list
> contains generic names and many brand names used in the USA and Canada.
This
> list is by no means comprehensive. Please help provide the names used in
> other countries by E-mailing me at: [EMAIL PROTECTED] and
> [EMAIL PROTECTED] .
>
> The compulsory administration of neuroleptic drugs by order of the state
is
> not only a violation of one's right to liberty but also the right to life
> and the pursuit of happiness. The right to life because neuroleptics
shorten
> life span and the precipitous onset of degenerative disease that these
drugs
> induce. The right to the pursuit of happiness because neuroleptics take
away
> sense of well being. The right to liberty because of not being able to
make
> ones own decision regarding the administration of these drugs and the
right
> to freedom of religious worship when the administration of these drugs
> precludes their use when it violates one's religious values and
convictions.
> For legitimate religious grounds to refuse these drugs see Footnote C
below.
>
> The compulsory administration of neuroleptics because of state order is a
> violation to one's right to life because the drugs not only shorten life
> span but also make the quality of one's life inferior due to the
precipitous
> onset of degenerative disease that these drugs induce. This is not a
> statement unsupported by fact. Let me now explain.
>
> Dopamine, norepinephrine, and epinephrine all belong to the class of
> neurotransmitters called catecholamines. Catecholamines are synthesized
from
> the amino acids L-Phenylalanine and L-Tyrosine. This is the complete step
by
> step synthesis of these substances: L-Phenylalanine --> L-Tyrosine -- >
> L-DOPA --> Dopamine --> Norepinephrine --> Epinephrine. L-Tyrosine is also
> converted to the thyroid hormone thyroxine by the thyroid and dopamine is
> also converted to the skin pigment melanin.
>
> Serotonin is a neurotransmitter that is synthesized from the amino acid
> L-Tryptophan. Melatonin the sleep-inducing hormone produced by the pineal
> gland is synthesized from Serotonin. Here is the complete step by step
> synthesis of these substances. L-Tryptophan --> 5-Hydroxy - L-Tryptophan
> (5-HTP) --> 5-Hydroxy - L-Tryptomine (Serotonin) --> Melatonin (O-methyl -
> N - Acetylserotonin).
>
> The main function of neuroleptics is blockage of catecholamines in the
> brain. They have greatest affinity for dopamine receptors and to a lessor
> extent norepinephrine and epinephrine. (A future report of mine will prove
> that these drugs destroy dopaminergic receptors. See Footnote B for a
> simplified explanation of this.) Neuroleptics block dopamine receptors in
> the brain with generally greatest affinity for D1 and D2 receptors.
Dopamine
> is a neurotransmitter and a receptor is the "keyhole" which dopamine like
a
> key, plugs into for neuronal communication. There are many other
> neurotransmitters, all with their respective "key holes" or receptors. But
> the analogy of a "keyhole" is insufficient because there are different
> receptors that fit their corresponding neurotransmitter. Different sides
of
> the neurotransmitters will fit into the different types of receptors. It
is
> like the fact that a puzzle piece has different sides with different
shapes.
> It's the different sides of the neurotransmitter that fit into their
> corresponding receptors. Since these neurotransmitter molecules are three
> dimensional in shape, the different sides of its shape fit the different
> receptors.
>
> When a dopaminergic neuron fires, it releases dopamine into the synapse
from
> vesicles within the presynaptic dendrite. The synapse is a gap or space
> between dendrites, which are branches off the neuron and a presynaptic
> dendrite is the dendrite that releases the neurotransmitter. Think of this
> gap as if it were the gap of a spark plug in an automobile except it is
not
> electricity that fires within this gap it is a chemical messenger; a
> neurotransmitter. This dopamine then binds with dopamine receptors on
> postsynaptic dendrites, which are the dendrites on the other end of the
gap
> that are the recipient of the neurotransmitter that branch off another
> neuron. The neurotransmitter then floats within the synapse and through
> quantum mechanics is drawn to the postsynaptic receptors on the end of the
> other dendrite where they bind activating ion pumps. Ion pumps are tubular
> molecular machines made up of amino acid building blocks, which is a
> protein. Ion pumps are also called channels. These receptors surrounding
the
> circumference of the ion pump cause this ion pump to dilate when activated
> by the stimulating neurotransmitter dopamine which allows electrolytes to
> pass through the cell membrane causing an electrical shift in polarity
which
> causes the neuron to fire an electrical charge down its axon. When the
> electrical charge reaches the other end it causes dopamine to be released
> from that neuron's presynaptic dendrites and the process continues in a
> cascade.
>
> The neurotransmitter Serotonin is an inhibiting neurotransmitter that
> mediates the expression of the stimulating neurotransmitter Dopamine.
There
> are third dendrites from neuron's of the serotoninergic system utilizing
> this inhibiting neurotransmitter serotonin that act as variable switches
at
> different points within the dopaminergic system. If serotonin is being
> released into a synapse from a third dendrite where dopamine had just been
> released, this inhibiting neurotransmitter then acts as a chemical
straight
> jacket by binding to serotoninergic receptors on these same ion pumps
which
> mediates the degree to which dopamine will cause this neuron to fire its
> electrical charge. This is the simplified relationship dopamine has with
> serotonin. There are many other neurotransmitters both stimulatory and
> inhibitory that act in many complex ways that have yet to be understood by
> science up until this point in time. (For instance the neurotransmitter
GABA
> (4-AminoButyric Acid) is another inhibiting neurotransmitter that has a
> mediating effect on the stimulating neurotransmitter Glutamate and also
has
> a mediating affect on the stimulating neurotransmitter dopamine in some
> dopaminergic pathways. The seizure drug Valproic acid (brand name Depakote
> or Depakane) is often prescribed for "mania" or "manic psychosis" and Bi
> Polar disorder also known as manic depression. Depakote causes an increase
> in GABA in the brain by inhibiting the two enzymes that are involved in
> breaking down this neurotransmitter.) Many newer neuroleptic drugs bind
with
> both dopamine and serotoninergic receptors. These serotoninergic receptors
> are activated continuously by these drugs while at the same time blocking
> normal serotoninergic function, greatly inhibiting dopaminergic
expression.
>
> Neuroleptic drugs block dopamine receptors in all areas of the brain for
the
> types of receptors they have affinity for. Antagonism of a type of
receptor
> is not neurological pathway specific and antagonizes all receptors of the
> type it has affinity for without regard for the actual function of the
> neurological pathways they affect. Most dopaminergic function takes place
> within the limbic system of the brain. (See the article in the magazine
> called "Scientific American" March-April 1996 Volume 84 called "Reward
> Deficiency Syndrome" on pages 134 and 135 for an explanation of the limbic
> system and how it relates to the "reward cascade" which I will discuss
later
> in this report.)
>
> The limbic system of the brain contains a structure called the
hypothalamus.
> This area of the brain called the hypothalamus is responsible for the
> regulation of pituitary hormones by the release of controlling hormones.
The
> pituitary is a small gland located at the base of the brain just under the
> hypothalamus. The pituitary in turn regulates all other bodily hormones.
See
> the book "Facts and Comparisons" III W. Port Plaza, Suite 300 St. Louis
MO.
> USA 63146-3098 (telephone 314-216-2100 or 1-800-223-0554). (Note this book
> is currently used by Rite Aid Pharmacies in the USA as a reference aid and
> it is a loose bound updatable book. The updatable section called
> "Antipsychotic Agents" is (c) 1990) This book states under "antipsychotic
> agents" that "Antipsychotics block postsynaptic dopamine receptors in the
> basal ganglia, hypothalamus, limbic system, brain stem and medulla... The
> phenothiazines appear to act at both D1 and D2 receptors, whereas
> haloperidol appears to act primarily at D2 receptors." ("D" here stands
for
> dopamine and each different receptor is numbered.) Also see the book "Drug
> Info for the Health Care Professional 17th edition volume I 1997" by
> Authority of the United States Pharmacopeial Convention, Inc. (c) 1997 by
> The United States Pharmacopeial Convention Inc. printed by Rand McNally,
> Taunton, Massachusetts. Distributed by USPC 12601 Twinbrook Parkway,
> Rockville, Maryland USA. This book states on page 2321 in the section on
> phenothiazines under the subheading "Mechanism of action/Effect" that
> neuroleptics "block postsynaptic mesolimbic dopaminergic receptors in the
> brain... and depress the release of hypothalamic and hypophyseal
hormones."
> Hypophyseal hormones are pituitary hormones.
>
> One hormone the hypothalamus produces is TRH (thyrotropin releasing
hormone
> or thyrotrophin releasing hormone). This hormone when released by the
> hypothalamus stimulates the release of the pituitary hormone TSH (thyroid
> stimulating hormone also called thyroidea stimulating hormone,
thyreotropic
> hormone, threotrophin hormone, TTH, thyrotropin, thyrotrophin). TSH in
turn
> when released by the pituitary stimulates the production or release of the
> thyroid hormone thyroxine which is abbreviated T4 (four because this is
the
> number of iodine atoms the hormone contains). Thyroxine is the hormone
that
> regulates bodily metabolism. Lowering metabolism by lowering the body's
> capacity to produce thyroxine shortens life span. See the book "Handbook
of
> Vitamins, Minerals and Hormones 2nd Edition" by Roman J. Kutsky, Ph.D.
> published by Van Nostrand Reinhold Company New York (c)1973. On page 369
in
> this book under "Essentially for Life" it states "Deficiency" of thyroxine
> "in adult shortens life span". Also on page 367 of this book in paragraph
> three it states that the capacity to produce this hormone is associated
with
> aging. And on page 371 under "Deficiency Symptoms" where it states among
> other things that deficiency of thyroxine causes "Deceased BMR" (BMR is
the
> abbreviation of Basal Metabolic Rate), "Increase in blood lipid and
> cholesterol" (lipid is fat). Also see the book "Fats that Heal Fats that
> Kill" (c) 1986,1993 by Udo Erasmus Ph.D. and published by Alive Books,
> Fraser Park Drive, Burnaby BC Canada V5J 5B9. On page 37 of this book at
the
> top of the page it states that "Deceased metabolic rate is also involved
in
> aging, arthritic diseases, cancer, and cardiovascular disorders, and is
> another general symptom of degenerative diseases." Published by Alive
Books,
> 7436 Fraser Park Drive, Burnaby BC Canada V5J 5B9. (c) 1986,1993.
>
> Exposure to cold temperatures will stimulate the production of TRH by
means
> of dopaminergic neurons within the hypothalamus which stimulates the
> production of TSH by the pituitary which in turn stimulates the production
> of T4 by the thyroid which raises metabolism and body temperature to keep
> the body warm and to regulate the burning of fat and carbohydrates as
fuel.
> The increased body temperature from a good metabolism stimulates the
> production of the anabolic hormone testosterone and anabolism keeps the
body
> rejuvenated and fights aging. As I have shown interfering with this
process
> lowers metabolism and shortens life span. Lowered metabolism causes weight
> gain and according to the American Medical Association's own statistics
the
> more overweight a person is the more likely that person will suffer a
> premature death. See the chapter called "Drugs Used in Obesity" starting o
n
> page 2439 of the book "Drug Evaluations Annual 1995" by the American
Medical
> Association. Neuroleptic drugs suppress the production of T4 thereby
> lowering metabolism by suppressing the release of the hypothalamic hormone
> TRH. Metabolism is the main mechanism that promotes thermogenesis.
> Thermogenesis is the production of body heat from the burning of fatty
acids
> and glucose when body heat is lost due to exposure to cold temperatures
and
> simply the continuous normal loss of body heat. It is T4 in the end that
is
> responsible for helping to generate body heat that is lost and to keep the
> bodies metabolism going which includes both catabolism the burning of fat
> and glucose as fuel and anabolism the building up of the body through the
> maintenance and manufacture of replacement biochemical substances and
> structure which fights aging. Metabolism is both catabolic and anabolic.
> Although T4 technically is a catabolic hormone catabolism and anabolism
are
> interconnected in a continues cycle so catabolism through T4 promotes
> anabolism. A healthy well nourished body will not burn protein as fuel.
>
> Because of the neuroleptics or antipsychotics actions on the hypothalamus
> suppressing the release of TRH the pituitary doesn't produce as much TSH
and
> in turn the Thyroid doesn't produce as much T4. See the book again "Facts
> and Comparisons". This book states under "Antipsychotic Agents" that
> neuroleptics "depress various components of the reticular activating
system
> which is involved in the control of basal metabolic rate and body
> temperature, wakefulness, vasomotor tone, emesis, and hormonal balance."
> Also see the book "Cecil Textbook of Medicine 19th edition" edited by
James
> B. Wyngaarden, MD, Lloyd H. Smith, Jr., MD and J. Claude Bennett, MD
> published by W.B. Saunders Company, Harcourt Brace Jovanovich, Inc.
> Philadelphia London, Toronto, Montreal, Sydney, Tokyo. This book states on
> page 1569 under "The Pathogenesis of Fever" under the subheading
"Initiation
> of Fever" that "...thermoregulation originate[s] in the hypothalamus" and
> "... neuroleptic drugs are capable of disrupting the hypothalamic response
> and may interfere with the development of fever. Among these, [the
> neuroleptic] phenothiazines are the best known for their poikilothermic
> effect. These agents are not specifically active in febrile states;
rather,
> they act to disable thermoregulatory mechanisms at all times following
their
> administration." Also see the book called "AHFS 96 Drug Information"
> published by American Hospital Formulary Service and "published by the
> authority of the board of directors of the American Hospital Formulary
> Service. This book states on page 1617 at the bottom right of the page;
> "Phenothiazines have a poikilothermic effect, interfering with temperature
> regulation in the hypothalamus: depending on environmental conditions,
> hypothermia or hyperthermia can occur." Hypothermia is under heating of
the
> body and hyperthermia is overheating of the body. Many people forced to
take
> neuroleptics have died of heatstroke. See footnote A at the end of this
> thesis.
>
> Again see the book "Drug Info for the Health Care Professional 17th
edition
> volume I 1997" on page 2321 in the section on phenothiazines under the
> subheading "Mechanism of action/Effect" that neuroleptics "block
> postsynaptic mesolimbic dopaminergic receptors in the brain... and depress
> the release of hypothalamic and hypophyseal hormones." These effects are
the
> result of blockage or destruction of dopamine receptors within the
> hypothalamus. Suppression of this hormonal system is one of the main
> mechanisms in which neuroleptics shorten life span.
>
> Many of the quotes are obtained from statements concerning the class of
> neuroleptics called phenothiazines but since all neuroleptics block or
> destroy dopamine receptors they all produce the same undesirable symptoms.
> For instance this quote from the book "Drug Info for the Health Care
> Professional" page 1564 under the heading of "haloperidol" a neuroleptic
in
> a class by its self; "[The] Pharmacological effects of haloperidol are
> similar to the effects of... phenothiazines". All neuroleptics block or
> destroy dopamine receptors so therefore all suppress hypothalamic hormone
> secretion. Now again see the book "Fats that Heal Fats that Kill" on page
37
> at the top of the page where it states that "Decreased metabolic rate is
> also involved in aging, arthritic diseases, cancer, and cardiovascular
> disorders, and is another general symptom of degenerative disease." On
page
> 191 of the same book at the top of the page it states; "The brightness of
> the fire is the rate at which our body produces energy our metabolic rate.
> For continued good health, it is vital that the fire of life burns
> brightly." Decreased metabolic rate not only leads to obesity but also to
> degenerative disease. Not only do neuroleptic drugs shorten life span but
> also make life inferior due to inducing the onset of degenerative disease.
>
> See the book "Facts and Comparisons" again under "antipsychotic agents"
and
> note that one of the neuroleptic drugs is sarcastically named
"thioridazine"
> (Brand names are Mellaril, Novoridazine and Thioril) denoting the fact
that
> these drugs suppress the production of the thyroid hormone thyroxine or
T4.
> "Thio" being a grammatical construct from the beginning of the word
> thyroxine and also the beginning of the word iodine, which is the mineral
> that this hormone contains, followed by the word rid in the middle of this
> construct denoting the fact that these drugs get rid of or suppress the
> release of this hormone. In my opinion this reveals the utter contempt the
> designers of these drugs have for those labeled "mentally ill". Also the
> question must be asked, why would a drug be named after a life span
> shortening "side effect" unless that was its sole intended purpose? This
> also reveals that the designers of these drugs knew the biological effects
> of these drugs even before they were pushed on the public. Also notice
that
> one of the neuroleptics is called "mesoridazine besylate" (Brand name is
> Serentil). Meso means middle which is where the limbic system is located
in
> the brain. Since neuroleptic drugs shorten life span, the compulsory
> administration of these drugs by court order of the state is a violation
of
> one's right to life.
>
> Again see the book "Drug Info for the Health Care Professional 17th
edition
> volume I 1997" on page 2321 which states that neuroleptics "block
> postsynaptic mesolimbic dopaminergic receptors in the brain ... [and] ...
> depress the release of hypothalamic and hypophyseal hormones." Again
> hypophyseal hormones are hormones produced by the pituitary gland. Because
> hypothalamic hormones control the release of pituitary hormones this is
the
> reason pituitary hormones are suppressed. Most hypothalamic hormones are
> releasing hormones. One hypothalamic hormone which is an inhibiting
hormone
> is prolactin release-inhibiting hormone (abbreviated PRIH also called PIF,
> RIH, prolactin inhibiting factor or prolactin inhibiting hormone, PIH,
> prolactostatin).
>
> Since neuroleptic drugs suppress the release of this inhibiting hormone
then
> the pituitary is free to secrete abnormal amounts of this female hormone
> that regulates lactation into the blood stream of both males and females.
> Most other bodily hormones are suppressed by neuroleptics except for
> prolactin for this reason. Since phenothiazines were the first
neuroleptics
> used starting with Thorazine and the fact that phenothiazines have been
used
> on more people then all other neuroleptics combined, the affect on hormone
> production caused by these drugs has been well established. All the other
> neuroleptics have been designed after what has been learned from the
> phenothiazines.
>
> The level of hypothalamic hormone production is in direct correlation with
> dopaminergic activity or the ability of the dopaminergic system to
function
> normally without interference. For this reason all neuroleptics that block
> or destroy dopamine receptors or utilize the serotoninergic system or both
> will suppress the production of all hypothalamic hormones which in turn
> suppresses pituitary hormone production and in turn all other bodily
> hormones.
>
> Serotonin is an inhibiting neurotransmitter that mediates the function of
> the dopaminergic system. Excess serotonin stimulation has the effect of
> suppressing the dopaminergic system. One neuroleptic called Molindone
(brand
> names Lidone and Moban) utilizes this function by mimicking serotonin.
Also
> this is why antidepressant drugs that raise levels of serotonin cause
sexual
> dysfunction. One "antidepressant" drug Sertraline (Brand name Zoloft) is
> molecularly indistinguishable from that of the neuroleptics. In fact
Zoloft
> has even been tried as a neuroleptic. Perhaps it's labeled as an
> antidepressant just for people who insist that their "problems are just
> depression". A man I talked to on the telephone at the FDA told me that
> Zoloft was so powerful at destroying sexual function that if just one pill
> is taken by someone with premature ejaculation it will slow him down. But
> don't take this man's word for it. Even the PDR says Zoloft causes sexual
> dysfunction. Percentage rates of impotence listed in the PDR for various
> drugs is misleading as only people who were sexually active and not
> embarrassed to speak about it would even report impotence or sexual
> dysfunction as a side effect. Every male I have questioned who has been on
> these drugs has confided in me that these drugs have caused them some type
> of major sexual problem.
>
> See the book "Biochemistry A Case-Oriented Approach fifth edition" by the
> Department of Biochemistry, The University of Iowa College of Medicine,
Iowa
> City, Iowa. Montgomery Rex Ph.D., Thomas Conway Ph.D. and Arthur A.
Spector
> MD (c) 1990 The C.V. Mosby Company. This book states on page 761 that
> prolactin secretion is increased by "serotonin". See the book "Facts and
> Comparisons" again under "antipsychotic agents" under "Carcinogenicity /
> prolactin secretion:" which states "Neuroleptic drugs elevate prolactin
> levels which persist during chronic administration." This is a reference
to
> all neuroleptics, not just phenothiazines. Also see the book again called
> "Biochemistry A Case-Oriented Approach fifth edition" on page 761 in the
> seventh paragraph where it states that "Some medications, acting as
dopamine
> antagonist, increase prolactin secretion. Although many drugs of this type
> exist, the most common are the antipsychotic phenothiazines, such as
> thorazine." On page 778 of this same book it states in the first
paragraph;
> "Dopamine is an active compound believed to inhibit prolactin secretion".
>
> Since neuroleptics block or destroy dopamine receptors there is no
> dopaminergic activity to inhibit prolactin secretion. Going back to page
761
> of this same book it sates in the fifth paragraph under "Disorders
> associated with prolactin" that "Prolactin secretion has a dramatic effect
> in blocking the pituitary gonadotrophs, and elevated prolactin levels are
> associated with sexual dysfunction. Hyperprolactinemia before puberty
blocks
> sexual maturation and the pubertal growth spurt. After puberty,
> hyperprolactinemia is associated with loss of libido and impotence in
males
> and amenorrhea in females." The gonads are testicles in men and ovaries in
> women and amenorrea is the abnormal suspension or suppression of
> menstruation. Hyperprolactinemia is the abnormal excessive production of
the
> female hormone prolactin.
>
> One pituitary gonadotroph is luteinizing hormone (abbreviated LH and also
> called luteotrophin or luteotropin, interstitial cell-stimulating hormone,
> ICSH, Prolan B, gonadotrophin II or gonadotropin II, metakentrin, corpus
> luteum-ripening hormone). See the book again "Handbook of Vitamins,
Minerals
> and Hormones" on page 323 under "Deficiency Diseases, Disorders" where it
> states that deficiency of this hormone causes "Hypogonadism".
(Hypogonadism
> is the inability of the gonads to perform their function of remaining
> fertile and producing sex hormones.) Also on this page it states that this
> hormone is necessary for "reproduction". Now reference page 327 of this
book
> under "Mode of Action" where it states that this hormone "increases
> synthesis of steroid hormones (sex hormones) ... estradiol (estrogen) ...
> [and] testosterone". Also here it also states that this hormone
"stimulates
> rupture of follicles in ovary". (This is so the ova or egg can be released
> from the ovary.)
>
> The other pituitary gonadotroph is follicle stimulating hormone
(abbreviated
> FSH, and also called Follotropin or Follotrophin, Thylakentrin, Prolan A,
> gonadotrophin I or gonadotrophin I, gametogenic hormone, follicle ripening
> hormone, gametokinetic hormone). See the book again "Handbook of Vitamins,
> Minerals and Hormones" on page 330 where it states that this hormone is
> required for "reproduction". Also see page 332 under "Deficiency Symptoms"
> where it states that deficiency of this hormone causes "Decreased
> gametogenic function and development (nonfunctional)". (This is just a
fancy
> way of saying that deficiency of this hormone will cause any sperm or ova
> produced to be genetically incapable of producing offspring or rendering
any
> sperm or ova (eggs) produced "nonfunctional") Also on this page it states
> that deficiency of this hormone also causes "Atrophy of the gonads"
(atrophy
> means to waste away), "No maturation of ova, sperm", "Obesity", "Decreased
> libido, potency, hair growth" and "decreased blood levels of estrogen".
> (Estrogen is the female sex hormone.) Pituitary gonadotrophs stimulate the
> production of sex hormones by the gonads.
>
> The hypothalamic hormone, luteinizing hormone-releasing hormone regulates
> these pituitary gonadotrophs. (Abbreviated LRH also called LRF,
LH-releasing
> factor or LH-releasing hormone, (LH-RH/FSH-RH), Gonadotropin releasing
> hormone (abbreviated Gon-RH or GnRH)) This hormone is suppressed by
> neuroleptics so here is another mechanism by which sex hormone production
is
> inhibited besides the inhibition due to excessive prolactin secretion.
>
> Going further on to the sex hormones which neuroleptic drugs inhibit we
will
> learn more about what these drugs do to the body. Estradiol also called
> estrogen (some other names are female hormone, dihydrotheelin,
> dihydrofollicular hormone, dihydrofolliculin) "is essential for
reproduction
> and female characteristics. Its chief functions are to maintain and
regulate
> female sex characteristics and behavior. Its chief importance is as the
> major female sex hormone with its command of female sex development and
> maintenance of female body characteristics and behavior. ...Deficiency
> conditions include menopause and delayed maturation." As stated in the
book
> "Hand Book of Vitamins, Minerals and Hormones" on page 415. (See footnote
D)
> On page 419 of the book "Handbook of Vitamins, Minerals and Hormones" it
> states that deficiency of estrogen will cause "Delayed maturation",
"Female
> accessory and reproductive organs recess" (recess means not to function),
> "Decreased female behavior pattern", "Senescence" (means Growing old,
> aging), and "Menopause". Here is further proof that neuroleptic drugs
> program the body to self-destruct, grow old and die and proof that these
> drugs prevent reproduction.
>
> Another female hormone that is stimulated by the hypothalamic hormone,
> luteinizing hormone-releasing hormone through the pituitary hormone
> luteinizing hormone is progesterone. So therefore neuroleptics also
inhibit
> the production of this hormone as well. On page 423 of the book "Handbook
of
> Vitamins, Minerals and Hormones" it states that progesterone "is
indirectly
> essential for life, since it is a precursor to aldosterone and cortisol,
> which is essential. Its chief functions are to synergize the actions of
> estradiol (estrogen) in the female organs, especially during pregnancy.
Its
> importance stems from the fact that it is a precursor to all the steroid
> hormones and that estradiol (estrogen) requires its presence for many of
its
> actions. ...Deficiency conditions [include]... dysfunctional uterine
> bleeding." On page 427 under "Deficiency Symptoms" this book states that
> deficiency of progesterone causes "Termination of pregnancy", "Decreased
> production of steroids", Decreased ovulation", "Loss of normal cyclic
> changes" and "Decreased development for implantation and gestation". This
is
> shocking because neuroleptics are routinely given to pregnant women who
> commonly have miscarriages and then the prescribing psychiatrists will
deny
> that the drugs were the cause!
>
> Now onto the male sex hormone testosterone; Testosterone's "chief
functions
> are: development and maintenance of the male organs, male sex
> characteristics, and behavior, as well as stimulation of growth
(anabolic),
> and metabolism of muscles, liver; and kidney. The chief importance of
> testosterone lies in its major command of male sex development, body
> characteristics, and behavior. Deficiencies include eunuchoidism, and male
> hypogonadism. ...Testosterone is formed mainly in the testes (interstitial
> cells) but also in small amounts in the adrenal cortex and the ovary[s]."
> (From page 431 of the book "Handbook of Vitamins, Minerals and Hormones")
On
> page 433 of this book it states that testosterone is "essential for
> reproduction in all (male) vertebrates". On page 435 of this book it
states
> that deficiency of testosterone will cause, "Involution of accessory
organs
> (prostate, seminal vesicles)", (involution is the progressive decline or
> degeneration of normal physiological functioning occurring as a result of
> the aging process and or decrease in size of an organ.) "Decreased male
> behavior patterns and libido", "Decreased secondary sex traits", "Poor
> muscle development and function". Neuroleptic drugs will destroy sexual
> function in men and produce sterility and will cause musculature to waste
> away since this is another hormone that neuroleptic drugs suppress the
> production of. See the web address below that states that risperidone
(which
> is a neuroleptic in a class by itself) will cause "testicular atrophy"
> because of its "antidopaminergic activity". (This web-site is open to the
> public but requires a password that can be obtained immediately.) All
> neuroleptics though will cause testicular atrophy do to hormonal
> suppression.
>
http://www.medscape.com/Medscape/MentalHealth/1997/v02.n10/mh3145.wirshing/m
> h3145.wirshing.html This link can no longer be accessed directly. You must
> enter their site now and do a search using their search engine under
> "testicular atrophy". It's clear they did this because I linked them. Just
> use their search engine and do a search for "testicular atrophy" and
> reference the article called "Atypical Antipsychotics: A Practical
Review".
> Then use your browser to find the phrase "testicular atrophy" on that
page.
> Remember you must first get your password before you can access this page.
> Passwords are open to the public and all you have to do is fill out their
> questionnaire.
>
> I have received this argument from someone who presented himself as a
> medical student. He said that dopamine and Prolactin Release-Inhibiting
> Hormone is one and the same. He also said of course a drug acting as a
> dopamine antagonist is going to affect this hormone. I had to set this man
> straight. I told him this: "That is not true. Prolactin Release
> Inhibiting-Hormone (also known as PRIH, Prolactostatin, RIH, Prolactin
> Inhibiting Factor or hormone, PIF, PIH, PRIF), which is produced by the
> hypothalamus is a chain of amino acids similar in structure to Luteinizing
> Hormone-Releasing Hormone. This can be found in the book "The Handbook of
> Vitamins Minerals and Hormones" by Roman J. Kutsky, Ph.D. on page 303,
which
> is a very good biochemistry fact book written in outline form. At the time
> of publication of this book the exact structure of Prolactin
> Release-Inhibiting Hormone had not been determined but they knew enough
> about it that they knew it was similar in structure to Luteinizing
> Hormone-Releasing Hormone which is a chain of amino acids called a peptide
> which is a small protein. Dopamine is a monoamine derived from the amino
> acids L-Tyrosine and or L-Phenylalanine. It has been called "Prolactin
> Inhibiting Factor" but should never be called "hormone" since it is a
> neurotransmitter. Apparently there is much confusion in the medical field
> regarding this. I also told this man this; "I can understand your
confusion
> because it has been known for years that Dopamine affected Prolactin, but
by
> the mechanisms pointed out in this report. Dopamine affects the release of
> all hypothalamic hormones not just Prolactin Release Inhibiting Hormone or
> Prolactostatin. Here is the exact amino acid sequence of PRIH; Asp - Ala -
> Glu - Asn - Leu - Ile - Asp - Ser - Phe - Gln - Glu - Ile -Val - Lys -
Glu -
> Val - Gly - Gln - Leu - Ala - Glu - Thr - Gln - Arg - Phe - Glu - Cys -
> Thr - Thr - His - Gln - Pro - Arg - Ser - Pro - Leu - Arg - Asp - Leu -
> Lys - Gly - Ala - Leu - Glu - Ser - Leu - Ile - Glu - Glu - Glu - Thr -
> Gly - Gln - Lys - Lys - Ile". I also told him this: "Dopamine is not a
> hormone but a neurotransmitter. No doubt there are many independent
> neurological pathways that utilize this stimulating neurotransmitter in
the
> control of the release of these hypothalamic hormones. That is why the
> release of these hormones operate independently of each other and that
> dopamine antagonism inhibits all hypothalamic hormone production because
> antagonism is not neurological pathway specific and never will be. Which
is
> why the drug chemical approach to treating "mental illness" is
fundamentally
> flawed and will never be the answer.
>
> Go to this web-site where the hypothalamic peptide prolactostatin or
> prolactin release-inhibiting hormone is available for sale for research
> purposes. http://www.penlabs.com/biopro/biopeptides3_32.html It is called
> Prolactin Release Inhibiting Factor or (PIF) on this Web-site. Go here to
> this German language web-site where it is identified as a hypothalamic
> hormone. http://www-stud.uni-essen.de/~st0184/Infos/eseldiv.htm . The name
> Prolactostatin is used on this web-site. There are three hypothalamic
> "statins" and they are all inhibiting peptides. The idea that PRIH is
> dopamine is outmoded and erroneous. Dopamine is the controlling
> neurotransmitter involved in the release of PRIH. There have been studies
in
> the past that seemed to suggest that dopamine acts directly on the
pituitary
> to inhibit prolactin, but such studies are flawed in that infusing
dopamine
> into the area of the pituitary to observe the affect on Prolactin release
> could very well be effecting real PRIH release from the hypothalamus
simply
> because of the close proximity of the pituitary to the hypothalamus. The
> pituitary is after all at the base of the hypothalamus and this dopamine
> infusion could easily be crossing over into the hypothalamus and
stimulating
> the dopaminergic system and therefore releasing real PRIH which in turn
> would inhibit prolactin thus giving the appearance that dopamine was the
> sole causative factor acting directly on the pituitary.
>
> As has already been established neuroleptic drugs inhibit the production
of
> the hormone progesterone and it is a precursor to the hormone aldosterone,
> (Precursor means the body uses one substance to synthesis or to produce
> another substance. In simpler words the body makes the hormone aldosterone
> from the hormone progesterone) so therefore aldosterone is also inhibited
by
> neuroleptic drugs. See the book "Handbook of Vitamins, Minerals and
> Hormones" again on page 406 where many deficiency symptoms are cited which
> include "Muscular weakness" and "Stress intolerance". See the book again
on
> page 401 where it states at the bottom of the page that aldosterone is
"One
> of the most essential of all hormones; absence can be fatal in [a] short
> time period". Now look at the book "Facts and Comparisons" again under
> "antipsychotic agents" under "adverse reactions" where it states that
after
> the administration of neuroleptics that "Sudden Death has occasionally
been
> reported." Could this perhaps at least be partly due to the inhibition of
> aldosterone secretion by these neuroleptic drugs? Aldosterone secretion is
> partly governed by the pituitary hormone ACTH (the abbreviation for
> adrenocorticotropic hormone or adrenocorticotrophic hormone and also
called
> adrenocorticotropin or adrenocorticotrophin, corticotropic hormone or
> corticotrophin hormone) which is in turn governed by release of the
> hypothalamic hormone corticotropin-releasing hormone or
> corticotrophin-releasing hormone. (Abbreviated CRH and also called CRF,
> cortical-releasing factor or cortical-releasing hormone,
> adrenocorticotropin-releasing factor or adrenocorticotrophin-releasing
> factor, corticotropin-releasing factor or corticotrophin-releasing
factor.)
> See the book "Handbook of Vitamins Minerals and Hormones" again on page
342
> at the bottom of the page where it states that ACTH is "one of the most
> essential hormones-Absence causes notable shortening of normal life span."
> Now see page 344 of this same book where it states that deficiency of ACTH
> causes "Decreased weight of adrenal (atrophy)", "Decreased mobilization of
> free fatty acids" (so fat can be burned as fuel). It also states here that
> deficiency of ACTH causes "Decreased steroids in blood, urine (17-hydroxy
> and 17-keto)" (ketones are produced as a byproduct of the burning of fat
as
> fuel. Since deficiency of ACTH decreases ketones it means that fat is not
> being utilized as fuel which will lead to weight gain), "Fasting
> hypoglycemia" and "Increased insulin sensitivity" Which explains why it is
> common for people on these neuroleptic drugs to be glucose intolerant.
Since
> neuroleptic drugs inhibit the production of all these hormones described
> above, they also cause all the problems associated with deficiency of
these
> hormones. Here is unquestionable proof that neuroleptic drugs not only
> shorten life span but also destroy sexual function and fertility or the
> ability to procreate offspring or have children.
>
> Aldosterone is not the only hormone that is controlled by the ACTH, CRH
> hormonal cascade. ACTH and CRH also control the release of the adrenal
> hormone cortisol (Also called hydrocortisone, Compound F,
> 17-hydroxycorticosterone. Substance M, glucocorticoid) (also cortisol is
> converted into cortisone in the body). On page 407 of the book "Handbook
of
> Vitamins, Minerals and Hormones" it states that the "chief functions" of
> cortisol "are to maintain stress reactions, capillary permeability,
release
> of other hormones, liver anabolism, and extrahepatic catabolism. Its chief
> importance is maintenance of stress reactions". ("liver anabolism" is the
> rejuvenation of the liver and liver synthesis of substances that keep the
> body rejuvenated and "extrahepatic catabolism" is the burning of fat as
fuel
> in all parts of the body except for the liver.) Also on this page it
states
> that "Factors inhibiting the release" of cortisol "are: high
> glucocorticoids, low ACTH, and pituitary hormones." On page 408 of this
book
> it states at the bottom of the page that "Absence" of cortisol "causes
> shortening of life span due to inability to respond to stress situations."
> On page 411 of this book it states that deficiency of cortisol causes
> decreases in "Kidney function, leading to death", "Liver glycogen,
> gluconeogenesis", "Intestinal absorption, blood sugar" and "Stress
> response-Ultimately death". Also on page 411 it states that deficiency of
> cortisol will cause increases in "Fat anabolism, hemoconcentration" (Fat
> anabolism is the depositing of new fat tissue), "Muscular weakness" and
> others. Here is further proof that neuroleptic drugs shorten life span.
>
> Now on to another hormone which neuroleptic drugs suppress the release of
> which is the pituitary hormone growth hormone. (Abbreviated GH and also
> called somatotropin or somatotrophin, phyone, anterior pituitary growth
> hormone, adenohypophyseal growth hormone, somatotrophic hormone, STH) You
> may think that this is not a problem because most of the people these
drugs
> are given to are already adults or in their teens. (I have been recently
> discovering that a growing number of children with ADD or similar "conduct
> disorder" are being given these drugs.) But growth hormone has other
> functions in adults. See the book again called "Handbook of Vitamins,
> Minerals and Hormones" on page 307 where it states that "Because growth
> hormone controls the nitrogen balance of an organism, it is thought to be
> involved in the aging process." On page 309 of this same book it states
that
> absence of growth hormone will result in a "decrease in normal life span."
> On page 311 it states that deficiency will result in "Increased fat
> deposition." The hypothalamic hormone growth hormone-releasing hormone
> (abbreviated GRH and also called GHRH, GRF, somatotropin-releasing factor
or
> somatotrophin-releasing factor or somatotropin-releasing hormone or
> somatotrophin-releasing hormone, growth hormone releasing-releasing factor
> or growth hormone releasing-releasing hormone, SRF, GHRF) stimulates the
> secretion of growth hormone by the pituitary. As we have already learned
> neuroleptics suppress the release of both hypothalamic and pituitary
> hormones. Here is further evidence that neuroleptic drugs shorten life
span.
> Therefore the compulsory administration of neuroleptic drugs by court
order
> of the state is a violation of one's right to life.
>
> Neuroleptic drugs also possess "adrenergic blocking effects". Again
> reference the book "Facts and Comparisons" under the section
"Antipsychotic
> agents". The book states on the first page of this section "In addition,
the
> drugs exert anticholinergic and alpha-adrenergic blocking effects." Also
see
> the book "Drug Info for the Health Care Professional" again on page 2321
> under the section on phenothiazines where it states under the subheading
> "Mechanism of action/effect" that "Phenothiazines also produce an
> alpha-adrenergic blocking effect." (Phenothiazines being just one of the
> classes of neuroleptic drugs all of which block dopamine receptors and
cause
> the same effects due to blockage of these receptors.) This is so because
of
> the chemical similarity of dopamine to the adrenergic neurotransmitters,
all
> of which belong to a family of neurotransmitters called catecholamines.
The
> adrenergic neurotransmitters are adrenaline and noradrenaline. (Also
called
> epinephrine and norepinephrine) These two neurotransmitters are
synthesized
> or created from dopamine; thus the similarity in their molecular
structure.
> Both these neurotransmitters double as hormones. Since neuroleptics block
or
> destroy alpha-adrenergic postsynaptic receptors this would simulate a
> deficiency.
>
> See the book again "Handbook of Vitamins, Minerals and Hormones" this time
> under "Epinephrine". This book states on page 445 concerning epinephrine
> that "It is not essential for life, but it is indirectly essential, since
it
> is involved in stress responses via cortisol, which is essential". (We
have
> already learned the importance of cortisol.) On this same page it states
> that one of the functions of epinephrine or adrenaline is increasing
> metabolic rate in time of need to respond to stress or emergency. Also see
> page 447 under the subheading "Essentiality for Life" where it states that
> deficiency of this hormone and neurotransmitter will cause "possible
> shortening of life span due to decreased response to emergencies." So if
> threatened by a life threatening situation neuroleptic drugs make one
> physically ill equipped to face the threat. Also on page 448 under
> "Deficiency Symptoms", "Not fatal, but organism cannot respond to
emergency,
> hard work, temperature extreme, emotional disturbance". Not fatal of
course
> unless one fails to respond adequately to an emergency or dies of
> heatstroke. Again see the book "Facts and Comparisons" under
"Antipsychotic
> Agents" under "Adverse Reactions" where it states that
> "Heatstroke/Hyperpyrexia induced by neuroleptics has occurred. They may
act
> in several ways including disrupting the hypothalamic thermoregulator
> center, alpha-adrenergic blockage and autonomic mechanisms." (Hyperpyrexia
> is overheating of the body.) And of course not being able to respond to
> emotional disturbance or stress blocking the stress response making one
> incapable of dealing with stress thus precipitating agitation. This is
> destructive to the body in it's own right so here is another way these
> neuroleptic drugs shorten life span. Also appetite is controlled in part
by
> noradrenaline within the hypothalamus. Lowered metabolism and the
compulsion
> to consume more food due to increased appetite from the blockage of
> noradrenaline receptors within the hypothalamus equals weight gain. It can
> not be avoided. See the book again called "Facts and Comparisons" under
> "antipsychotic agents" under "Adverse Reactions" under "Miscellaneous"
which
> states that neuroleptics will cause "increases in appetite and weight".
>
> Neuroleptics also have "anticholinergic blocking effects". Acetylcholine
is
> a neurotransmitter. The brain produces an enzyme called
acetylcholinesterase
> to break down excess acetylcholine to prevent it from accumulating to
> abnormal levels. This break down of acetylcholine by this enzyme comprises
> the anticholinergic system. Neuroleptic drugs have "anticholinergic
blocking
> effects" meaning they cause the accumulation of acetylcholine to abnormal
> levels. Nerve gas's method of causing death is by its anticholinergic
> blocking activity. Also insecticide kills insects by this same method
> causing an abnormal build up of acetylcholine in the brain and nervous
> system of the insect.
>
> See the book again called "Facts and Comparisons" under "antipsychotic
> agents" at the bottom of the first page in this section where it states
that
> "In addition, the drugs exert anticholinergic and alpha-adrenergic
blocking
> effects." Now see the book called "Brainscapes" by Richard M. Restak, MD
> published by NY Herperion (c) 1995. On page 57 of this book it states; "As
> we have discussed earlier, after a neurotransmitter and it's receptor have
> reacted, the process must be brought to a halt, which is accomplished
either
> by the destruction of the neurotransmitter and recycling of it's
> constituents, or by a so-called reuptake system, whereby the
> neurotransmitter is recaptured and stored once again within the vesicle.
In
> the case of acetylcholine, the process involves a breakdown brought about
by
> the enzyme acetylcholinesterase, which cleaves the neurotransmitter back
to
> its original chemical building blocks. This process can be interfered with
> by compounds responsible for some of the worst horrors of
twentieth-century
> warfare. Nerve gases, such as the deadly Sarin released into the subway
> system in Tokyo in March 1995, form irreversible bonds with
> anticholinesterase [acetylcholinesterase], thus inhibiting the enzymes'
> ability to break down acetylcholine in the synapse." Then on page 121 of
> this same book it states; "In the section on neurotransmitters we
mentioned
> another class of neurotoxins, inhibitors of the enzyme
acetylcholinesterase,
> which breaks down the neurotransmitter acetylcholine. Many pesticides are
> designed to attack the nervous system of insects by altering the breakdown
> of acetylcholine. Not surprisingly, these agents also act on our brains
and
> nervous systems to produce symptoms like weakness, difficulty in
breathing,
> visual disturbances, and in some cases explosive violence. With low rates
of
> exposure the problems are more subtle, a prevailing sense of tension,
> disturbed sleep, restlessness, chronic anxiety, and nervousness when
> standing in lines."
>
> All of these symptoms can be observed in people on neuroleptics. For
> instance "visual disturbances", see the book again "Facts and Comparisons"
> under "Ocular" in Adverse Reactions which states that neuroleptics will
> cause "Glaucoma; photophobia; blurred vision; miosis; mydriasis; ptosis;
> star-shaped lenticular opacities; epithelial keratopathies; pigmentary
> retinopathy. Eye lesions may regress after drug withdrawal." Also as
Richard
> Restak MD reports insecticide exposure will cause "restlessness" and or
> "nervousness when standing in lines." This has been given a name
> "Akathisia", see the book again "Facts and Comparisons" under
> "Extrapyramidal" under "Akathisia" which states that neuroleptics cause "a
> condition of constant motor restlessness [called akathisia] and may
include
> feelings of muscle quivering, an inability to sit still and an urge to
> constantly move about." Also he reports that insecticide exposure will
cause
> "difficulty breathing", again see the book "Facts and Comparisons" in the
> same section under "Respiratory" which states that neuroleptics cause
> "Laryngospasm; bronchospasm; increased depth of respiration; dyspnea."
> (Dyspnea is difficulty breathing or shortness of breath.) Also he states
> that insecticide exposure will cause "weakness", again see the book "Facts
> and Comparisons" in the same section under "Other CNS effects:" that
> neuroleptic drugs will cause "Cerebral edema, headache, weakness, tremor,
> staggering gait; twitching; tension; jitteriness; akinesia; ataxia;
fatigue;
> slurring [of speech]; abnormal cerebrospinal fluid proteins;" etc. Also
> Richard Restak MD reports that insecticide exposure will cause "disturbed
> sleep". Again see the book "Facts and Comparisons" in the same section
under
> "Other CNS effects" where it states that these drugs cause "insomnia" and
> under "Adverse behavioral effects:" where it states that neuroleptic drugs
> cause "nocturnal confusion" and "bizarre dreams". Richard Restak MD also
> reports that insecticide exposure will cause "a prevailing sense of
> tension", again see the book "Facts and Comparisons" under "Other CNS
> effects" where it states that neuroleptics cause "tension". Also he
reports
> that insecticide exposure will cause "anxiety". Now see the book called
"The
> Essential Guide to Prescription Drugs Revised Edition" (c) 1980 by James
W.
> Long MD and published by Harper & Row on page 344 under haloperadol (a
> neuroleptic drug) that this drug can cause "anxiety". Also Richard Restak
MD
> reports in his book that insecticide exposure causes "in some cases,
> explosive violence." Now see the book "Facts and Comparisons" in the same
> section under "Adverse behavioral effects:" where it states that
> neuroleptics can cause "hyperactivity" and "agitation". (See the
commentary
> coming up concerning a Star Trek Voyager episode entitled "The Chute"
where
> the story centered on this very effect.)
>
> Of course nerve gas and insecticide are poisons and neuroleptics have
> blatantly poisonous properties in that part of their function is the same
as
> that of nerve gas and insecticide in causing an abnormal build up of
> acetylcholine. In fact the very molecular base of one class of
neuroleptics
> called phenothiazines is used as an insecticide! See the book again
entitled
> "AHFS 96 Drug Information American Hospital Formulary Service" in the
second
> paragraph in the right column, which states that "Phenothiazine [a class
of
> neuroleptics] is still used as an anthelmintic in veterinary medicine and
as
> an insecticide." The very insecticides that Richard Restak MD is referring
> to in his book "Brainscapes" are being given to those labeled mentally ill
> as a claimed "beneficial medical treatment"! Here is further evidence that
> neuroleptics shorten life span. Again remember that all neuroleptics
> interfere with the breakdown of acetylcholine so don't assume that because
> the drug isn't a phenothiazine that it will not have these effects. Other
> neuroleptics may even be worse at interfering with the break down of
> acetylcholine then phenothiazine.
>
> Personally every time I have been on neuroleptics I have been extremely
> agitated sometimes breaking things. One time I even assaulted my father
> because of these drugs. These drugs can cause extreme feelings of rage.
This
> is precipitated by the horrible torturous feelings that these drugs
induce.
> This well-known effect of excess acetylcholine was even the theme for an
> entertaining Star Trek Voyager episode called "The Chute" (this is an
> American science fiction television show) in which two of the members of
the
> crew were abducted and placed aboard a space station prison. In order to
> keep the prison population down the prisoners where unknowingly exposed to
a
> chemical that caused the build up of acetylcholine by interfering with its
> breakdown. This caused the prisoners to be violent and enraged often
killing
> each other. After the crew members were rescued the "holographic" doctor
on
> Voyager revealed that it was interference with the breakdown of
> acetylcholine that was causing the violence and that the prison's
operators
> must be using the chemical to keep the prison's population down. (Could
this
> be why many prisoners in the USA are forced or coerced into taking
> neuroleptic drugs?) Based on this fact of neuroleptics, not only is
> compulsory administration of neuroleptic drugs by court order of the state
a
> violation of the right to life but also the pursuit of happiness because
> these drugs take away one's sense of well being causing "agitation".
> Ironically these drugs are often prescribed under the pretense that they
> will prevent violence when in reality they can actually promote it.
>
> This leads to another problem with neuroleptic drugs and how they take
away
> sense of well being. Within the limbic system of the brain is an
> electrochemical cascade called the "reward system". This system is
> responsible for giving a person their sense of well being. Disruption of
> this electrochemical neuronal cascade results in ones sense of well being,
> being supplanted with negative emotions such as anxiety, depression, anger
> and generally a very negative outlook on things. The end result of the
> reward system's neuronal cascade is stimulation of dopamine D2 receptors
> within the nucleus accumbens and the hippocampus, which are located within
> the limbic system of the brain.
>
> See a problem yet? Remember neuroleptic drugs as you have already learned
> block or destroy dopamine D2 receptors preventing their stimulation by the
> neurotransmitter dopamine. Again see the book "Facts and Comparisons"
under
> "Antipsychotic Agents" where it states on the first page of this section;
> "Antipsychotics block postsynaptic dopamine receptors in the basal
ganglia,
> hypothalamus, limbic system, brain stem and medulla. ...The phenothiazines
> appear to act at both D1 and D2 receptors, whereas haloperadol appears to
> act primarily at D2 receptors."
>
> Neuroleptics do indeed take away one's sense of well being by utilizing
more
> then one mechanism. This is very self destructive to the body and will
> result in a premature death if one doesn't commit suicide first.
Ironically
> these drugs are prescribed to people to prevent suicide when in reality
they
> actually promote it. Perhaps the designers of these drugs want to give the
> person that extra amount to push them over the edge and actually do it.
> Personally two times I was on these drugs for extended periods I attempted
> suicide because of them.
>
> So here is further proof that the compulsory administration of neuroleptic
> drugs because of court order of the state is not only a violation of one's
> right to life but also one's right to the pursuit of happiness. This is so
> since the administration of these drugs take away sense of well being
making
> it very difficult to feel happiness.
>
> Read the article entitled "Reward Deficiency Syndrome" as published in
> "American Scientist" magazine March-April 1996 for a detailed and in depth
> discussion of this brain function called the "reward cascade". This
magazine
> article states on page 135 under figure 4, "If the activity of the
dopamine
> D2 receptor is deficient, the activity of neurons in the nucleus accumbens
> and the hippocampus is decreased, and the individual experiences
unpleasant
> emotions or cravings for substances that can provide temporary relief by
> releasing dopamine." On page 132 of this article it states that disruption
> of the stimulation of D2 receptors as part of the "reward cascade" will
> "supplant an individual's feeling of well being with anxiety, anger or a
> craving for a substance that can alleviate the negative emotions." In this
> magazine article it explains that "reward deficiency syndrome" is the
cause
> of behavioral disorders such as attention deficit disorder (with and
without
> hyperactivity), personality disorder, conduct disorder, antisocial
> personality, aggressive behavior, autism (autism is the abnormal
> introversion and egocentricity, acceptance of fantasy rather then
reality).
>
> After reading this article I came to understand the source of many of my
own
> problems and why neuroleptics were exasperating them. And also why I had
an
> insatiable craving for alcohol whenever I was on neuroleptics that even
> persisted for a long time after they were discontinued since the damage
> caused by neuroleptics to the dopaminergic system is long term. Again see
> the book "Facts and Comparisons" under "antipsychotic agents" under
"Adverse
> Reactions" under "Adverse behavioral effects:" where it states that
> neuroleptics will cause "...restlessness; hyperactivity; agitation; ...
> depression; ... paranoid reactions." And again see the book "The Essential
> Guide to Prescription Drugs Revised Edition" (c) 1980 where it states that
> haloperadol (a neuroleptic) causes "anxiety". Also as reported in this
> report, since these neuroleptic drugs cause adrenergic blocking and
> inhibition of the adrenal hormone cortisol, these drugs reduce capacity to
> deal with stress both emotionally and physically. So here is irrefutable
> proof that these drugs not only take away sense of well being but also
> shorten life span.
>
> The state does NOT have the right under any circumstances to order the
> compulsory administration of a substance, which shortens life span and
takes
> away one's sense of well being, a substance with poisonous properties like
> that of nerve gas and insecticide! The constitution of the United States
of
> America guarantees certain inalienable rights, namely the right to liberty
> (making one's own decision regarding personal matters), the right to life,
> and the right to the pursuit of happiness. When the state orders the
> compulsory administration of neuroleptics EVERY ONE of these inalienable
> rights is being violated.
>
> Mental health officials will first do their very best to intimidate and
> coerce a "patient" or "client" into "voluntarily" taking neuroleptics
before
> they will attempt to get a court order that will allow them to
involuntarily
> drug someone. They will use mind games or psychology on a person coming
back
> to them repeatedly telling them such things as "Don't you want to get
> better?". Many times when doing this they will invade your personal space.
> Perhaps in an attempt to provoke you into an act of violence that they
will
> then use as "evidence" that you "need" these drugs. Many times they will
> tell the person that they will not qualify for social security assistance
if
> they do not take the drugs or that they will remain locked up in an
> institution if they do not take the drugs. Any call to the Social Security
> Administration will reveal that it is not required that someone takes
> neuroleptics in order to receive or continue to receive social security.
> Perhaps what the psychiatrists are really saying is that they will not
> support a disability claim if the person doesn't take the drugs. In such
> case this is blackmail. Don't be tricked by a psychiatrist's attempts to
get
> you to try a new drug that is supposed to be "safer" then the older ones.
> All neuroleptics block or destroy dopamine receptors even the newer ones.
> Therefore the newer so called, "safer" neuroleptics will also cause what
has
> been described in this report by the very fact that they also block or
> destroy dopamine receptors. Risperidone and Zyprexa (Brand name is
> Olanzapine) affect higher reasoning centers and make it almost impossible
to
> form complex thoughts. This is do to its effects on the neurotransmitter
> Serotonin. If I were on either of these two drugs it would have been
> impossible for me to do the research and write this report. I speak form
> experience because I have temporarily taken both Zyprexa and Risperidone.
On
> just two pills of Zyprexa I could not even access my memories of research
on
> these drugs. On Risperidone I had slurred speech, word substitutions,
> difficulty forming complex thoughts pages of text appeared as blank piece
of
> paper when previously I could rapidly digest the material.
>
> Resist psychiatric drugging. Use the evidence in this report to argue in
> probate court that the involuntary administration of these drugs is a
> violation of all your fundamental constitutional rights, most importantly
> the right to life and the pursuit of happiness. They will not be able to
get
> around these two rights. They take away liberty from the "mentally ill"
> under the guise that they are supposedly not competent to make their own
> decisions regarding their own medical care, but they will not be able to
get
> around these other two rights. If possible get your own attorney rather
then
> allowing the court to appoint one for you because it is my experience that
> these court appointed attorneys are not really motivated to defend you and
> may even be secretly serving the interest of the mental health officials
and
> the state. If you do not get a favorable decision in the probate court
then
> insist on an appeal and remember to attempt to maintain your determination
> to follow through with the appeal realizing that these drugs take away
your
> will power and ability to resist domination. Use this knowledge to fight
> this effect of the drugs. If necessary appeal these constitutional issues
> all the way to the Supreme Court. Only when we as a people fight and
defend
> our rights will changes be made, not only for our personal protection but
> also for that of our loved ones and our children and our children's
> children. It is not just the "mentally ill" that are affected by this.
> Routinely these drugs are administered to the retarded (including
children)
> and the elderly in nursing homes as well as Alzheimer's patients, people
> with head injuries or brain damage etc.
>
> It is very clear that the motivation behind the administration of
> antipsychotics, antipsychotic drugs, neuroleptics, or neuroleptic drugs to
> the "mentally ill" and others is eugenic in nature. For those who do not
> know what eugenics is, it is idea that the human race can be improved by
> preventing "inferior stock" from reproducing and by inducing an early
death.
> The eugenic idea got its start with psychiatrists in the USA. Later Hitler
> shocked the world with the holocaust of millions of people, basing his
> program on eugenics practices that were already being carried out in the
> USA. But the first to be killed in Nazi Germany was the "mentally ill".
> Before Hitler did the things he did, the forced sterilization of the
> "mentally ill" in the USA was common practice. But as you are already
> probably starting to see after reading the evidence centered on
neuroleptic
> drugs, the same eugenics program is being covertly carried out under the
> guise of a "beneficial medical treatment". In the 1960s, the Eugenics
> Society of England adopted what they called "Crypto-eugenics", stating in
> their official reports that they would do eugenics through means and
> instruments not labeled as eugenics. Abortion and the push of birth
control
> is one of these and as it should be clear from reading this report on the
> true nature of neuroleptic drugs that it is a eugenics conspiracy also.
>
> After World War II so many psychiatrists immigrated to the USA that some
> time after the war, one third of all psychiatrists in the USA were former
> Nazi's.
>
> The following excerpt is taken from the public service publication
entitled
> "Psychiatry Destroying Religion in the Name of Salvation" by the Citizens
> Commission on Human Rights under the chapter entitled "The Devil's Doctors

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