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From: "Remy C." <[EMAIL PROTECTED]>
To: "endsecrecy list" <[EMAIL PROTECTED]>
Subject: [endsecrecy] Lyme Disease & Aspartame
Date: Saturday, December 16, 2000 6:08 PM

From:
http://www.rense.com/general5/lyme2.htm

Lyme Disease Is Sexually Transmitted -
Reactivated By Aspartame
>From Betty Martini Mission-Possible-USA@ altavista.net
12-4-00

Lyme Disease Is Sexually Transmitted,
Produces AutoImmune Self-Destruction
Which Is Reactivated By Aspartame

By Dr. James Bowen, M.D.
12-4-00

Lyme Disease and Aspartame Disease are tragically conjoined Twins. Aspartame
can cause Lyme disease and the Lyme treponema can cause the same
hyperautoimmunicity and resultant hyperautoimmunity destructions that
aspartame does. American medicine unfortunately overlooks the basics that
have long been known about Lyme Disease.

Aspartame can activate Lyme Disease in people who have the infection but
would not experience Lyme Disease because unimpaired by the ill effects of
aspartame the Lyme treponema would without hazard be eliminated by a
competent immune response. In addition, I think those who have had Lyme
Disease and achieved a "cure" may experience a relapse upon exposure to
aspartame or other sensitizing agents even though the Lyme treponema has
been eliminated from their body.

Lyme Disease has been known under other names in the British Isles for about
a hundred years. The British and continental experience could also serve
well as a learning model in the US. The other reason that an apparent lack
of understanding exists on these topics in the US is that the US has
defacto, virtual full control of the press by the government and the
establishment to protect the undefendable aspartame in spite of
constitutional provisions to the contrary. Whether with respect to Aspartame
Disease or Lyme Disease "hypersensitivity" and hyperautoimmunicity can be
used interchangeably to describe the particular immunologic disorder
engendered in either case whereby the human immune system is left so
impaired that it can readily be stimulated to a severe autoimmune attack on
the human organism itself: "hyperautoimmunity". One of aspartame's
hyperautoimmunity damages is chemical hypersensitivity, poly or multiple
chemical sensitivity syndrome, "aroma sensitivity" etc.

To save words this article will use hyperautoimmunity to denote disease and
pathology caused by autoimmune attack deriving from
hypersensitivity/hyperautoimmunicity. The common ground/common immunologic
event shared by aspartame and Lyme Disease is that the hypersensitivity that
is induced is largely caused by denatured human protein interacting with the
human immune system in either case. This leads to striking interactivity
between the two diseases on one hand and similar therapeutic hardships for
the afflicted patient on the other. It is about equally hard to find a
competent medical caregiver for either condition in the US because the
available information just isn't put to the attention of most physicians in
a credible manner. To treat either condition effectively requires a large
measure of courage as well as independent expertise even though the
treatments are logical, simple, safe and avail the patient immense benefit.

It seems quite overlooked that the hyperautoimmuninicity stimulated by the
treponema and the resulting hyperautoimmunity are the major cause of the
various symptoms of Lyme Disease. This explains why, what usually might be
considered an adequate antimicrobial approach for more straight forward
infectious processes is found to be inadequate for Lyme Disease. It can also
explain why that from the earliest experiences with Lyme Disease in the US
until the present time that the patients most grievously afflicted by Lyme
Disease and who are urgently in need of treatment for it are highly likely
to be serologically negative and test negative for Lyme Disease. Experienced
centers that treat significant volumes of Lyme disease cases have coined
this phenomenon "sero negative Lyme disease" because a majority of the
patients most grieviously afflicted by Lyme disease are indeed sero
negative.

One such clinic performed a follow through search for live organisms on
their cases and were able to culture Lyme treponema in 91% of sero
negatives. Remarkable confirmation because isolating the organism is a
highly technical task.

Three known biological mechanisms explain sero negativity in active Lyme
disease.

First: Since hypersensitivity/hyper autoimmunity is the pathophysiologic
mechanism the damages can be produced rapidly and in response to a minuscule
innoculum. Second: Hypersensitivity results from production of abnormal
amounts of abnormal IGG which forms an "IGG blockade" to sero positivity.

The highly pleomorphic profile of the Lyme surface antigens can totally omit
various antigens. For example, the immunologically dominant and often tested
for Osp A has been found to be persistently lacking in some regional phases
of treponemae. If your immune system can mount an appropriate and adequate
response, you may test positive and be able to eliminate the disease without
damages and the disease will often be self limiting; completely cured
without antimicrobial therapy of any kind. If you lack that kind of
immunocompetence you likely will not produce a positive titer in any case
and your immune system may be diverted to attacking your own body with
autoimmune destruction while not affecting the Lyme treponema at all.

The hyperautoimmunicity engendered by Aspartame and/or Lyme Disease is
probably the "blocking factor" that immunologists are looking for to explain
why serologic Lyme Disease testing often does not work. Thus, for many
people with negative Lyme titers, it is a devastating disease with
intolerable consequences arising from its not being vigorously and
thoroughly treated. Of the 53 laboratory tests currently available, none
yield accurate negative results. Lyme therapy experts can only say that
hopefully we will have therapeutically significant testing available in the
future but that at present we simply do not.

The destruction arising from leaving undetected Lyme Disease untreated may
be immediately evident or may occur in the near or distant future as is also
true of its close cousin syphilis. Yes, Lyme Disease can be spread venerally
and in several other ways. So a "bite" may never have occurred and the
classic "erythema migrans" rash may never be observed. In the long term, how
does and how can a miniscule and fragile micro-organism like the Lyme
spirochete survive the attack of the human immune system? The spirochete's
microscopic, near bacterial size and wormlike proportions with relatively
large surface area should make it readily subject to immune destruction
after the 10-60 days or so it usually takes for the immune system to mount
an adequate response to a new foreign protein.

Being a matador is a good analog to the "bait and switch" games the Lyme
spirochete plays with the immune system to prevent an appropriate immune
response from destroying it. The bull fighter would be quickly destroyed if
the bull could simply get a horn in him and remain unrestrained to finish
the job. The matador achieves mastery of the situation by inciting the bull
to charge him and then by avoiding bodily contact, changing the bull's
charge into a near miss which only wears the bull down leading to more and
more destruction of the bull. The matador also has a red flag that he can
manipulate with various wiggles and waves to further confuse and
disorientate the bull and engage the bull in self destructive charges.

Because of their small size and shape and mobility the treponemae cannot
depend on thick cuticles (secretions of defensive mucoid shields) or
encapsulating themselves as other larger parasitic organisms do, to protect
themselves. They would be completely destroyed by any competent immune
attack. The treponema much like the matador can accomplish only avoidance.
They use two (2) mechanisms; "game playing" and being "fast on their feet".
They play the game of "let's you and him fight" with the human immune system
and body, getting the body itself to be the focus of the immune attack
rather than the treponemae. It accomplishes this by making the major known
immunologically dominant constant component of its surface a human protein,
only slightly denatured called Osp A. Osp A is nearly identical to a human
protein called LFA-1. The very small difference is just enough to elicit
immune system response with destruction of the treponemae in the
immunocompetent but eliciting hyper auto immunicity in those with lesser
immune competence in this regards. Aspartame likewise creates denatured
human proteins of the body's own tissues with no healthy alternative insofar
as hypersensitivity is concerned.

Lyme vaccines, presently available, are based on Osp A and it is stated the
immunoglobulins produced enter the tick's digestive tract as it bites thus
destroying the Lyme organisms before they can ever reach the human body. Not
surprisingly, some people have accused the Osp A vaccines of having caused
severe knee damage by stimulating destructive arthritis. This correlates
well with the fact that Lyme arthritis almost invariably affects the knees
but other joints with less frequency. This pattern reflects the tremendous
physical stress imposed on the knees inherent to knee kinesiology. The Osp A
is considered an "arthrogenic antigen".

The immune difficulties are further complicated by the fact the spirochete
can constantly keep changing which genetic information it expresses in its
surface proteins. This "gene surfing" is very analogous to the matador
confusing and agitating the bull with his red flag. Even as the human immune
system may attempt to self correct, different antigenic pictures demanding
immediate destruction are presented on the organism's surface - again and
again - faster than the immune system can mount an effective response
against it. Thus in the less immuno-competent the human body itself becomes
the only readily and constantly target for immune attack and destruction.

A comparable example is the situation with "flu" vaccines. If the flu virus
strain mutates, even slightly, the antibodies elicited by the vaccine just
aren't effective against the flu bug. Lyme Disease damages and some of
aspartame's damages are hyperautoimmunity destruction based on inappropriate
immune response to denatured human protein.

It is no surprise then the two diseases can, and often do, produce identical
problems. It should likewise be possible, once a pattern of autoimmune
destruction has been established in either case, that the persistent
hyperautoimmunicity (after the situation has been quieted down) will produce
the same disease pattern as at least part of the response when
hyperautoimmunity is reactivated by either agent. Exposure to aspartame can
cause the Lyme disease process to recur after the Lyme organism has been
eliminated from the body. Likewise the Lyme organism can cause recurrence of
the hyperautoimmune destruction caused by aspartame such as the Persian Gulf
Syndrome and the others already mentioned. The chemical hyper reactivity
engendered by either disease can yield recurrences of either upon exposure
to other noxious chemicals at levels undetectable to "normal" or unafflicted
individuals. Therefore, in those already immunologically deranged by
aspartame, Lyme Disease will often be atypical: lacking the usual 10 to 14
day incubation period etc because immediate autoimmune hyper reactivity
already exists and is already subject to an immediately fulminant and
virulent response to very low doses of the treponemal antigen. This mutual
synergism/activation phenomena between aspartame poisoning and Lyme Disease
persists so that the activation of Lyme destruction is possible at almost
anytime amongst the large segment of our population sensitized by aspartame
exposure. Even pollen seasons and fungal blooms with airborne spores now are
problems of severe hyperautoimmunity for the hypersensitized and can be
expected to produce full blown autoimmune destructive processes without
treponemal reinfection nor exposure to aspartame itself.

This ill considered knowledge is not new to medicine. One classic medical
case of destructive hyperautoimmunicity is lupus erythematosus known to be
stimulated to reoccur upon exposure to various chemicals, many of them so
innocuous as to be included amongst commonly used medicines.

The need for higher doses of properly selected antibiotics used for a much
longer term than in most infections reflect two aspects of Lyme infection.
The treponemae are somewhat larger and more complex organisms than most
pathogenic bacteria and are thus harder to eliminate for that reason alone.
Moreover, an appropriate immune response is an essential component of the
successful treatment of any infection. Without it there is little hope of
cure. This brings us back to the Lyme "matador" and its "switch and bait"
defense and its fast footed "antigen surfing" to further disrupt immunologic
competence. This antigenic "channel surfing" can completely prevent the
immune system from destroying the treponema on one hand while continuing to
promote aggravated hyperautoimmunologic damages on the other.

In addition to the usual anti microbial actions, the high dose long term
antibiotics needed appear to assist the immune system with two (2) important
mechanisms. The first as is stated by some experts is that it "roughs up"
the treponemal surface allowing the immune system a better chance to
differentiate Osp A from human antigens and focus an immune attack on it
since the human cells are more resistant to damage from the appropriate
antimicrobial agents. Secondly by inhibiting and slowing treponemal
metabolic processes it can slow down the "mutation" of surface genetic
expression so the immune system can "catch up" and properly identify the
pathogen for destruction and in conjunction with the antibiotic eliminate
the trepanemae. Since the treponemae are independently of each other
"mutating" their expressed surface antigens it only makes sense that it will
take some time for the immune system to catch up to all their various
expressed forms and eliminate them all.

In all hyperautoimmunity diseases, cure depends on avoidance of the inciting
stimuli as one of the keystones of achieving a measure of good health. In
Lyme Disease, that includes successful eradicating of the Lyme organism and
avoiding the other mentioned immunologic stimuli. This explains much of the
confusion surrounding the clinical response. Some patients require prolonged
antibiotics but get well only after the antibiotics are stopped. Very likely
the antibiotic itself or some other substance given with it such as a
preservative dye or capsule coating, ie methyl cellulose, was keeping the
hyperautoimmunity fully activated. Most experts who regularly treat
recurrent Lyme Disease now prefer a 60 day antibiotic regimen.

The picture is further complicated by governmental bureaucracy which often
requires a positive Lyme titer prior to treatment as a "community standard
of practice". This is highly dangerous to those afflicted with Lyme Disease
for several reasons

1. The titer is negative in two thirds of cases of active Lyme Disease. 2.
Even a larger percentage of cases are negative after antibiotic
administration. 3. Active Lyme cases who have ever once had a positive
response to antibiotic therapy almost never exhibit a positive titer
thereafter. 4. The worst cases of Lyme Disease most frequently do not have a
positive titer. 5. At best the titer will not turn positive for 10-60 days
which is too late for those already having pre-existant autoimmunity -
pre-existing hyperautoimmunicity.

The Lyme sufferer must therefore quickly find a physician who is
knowledgeable of Lyme Disease and its treatment (rare) and rarer still
possessed of the courage to defy bureauacy and proceed with adequate
therapy! Even more rare would be to find such a physician also possessed of
knowledge of hyperautoimmunicity chemical hypersensitivity and aspartame
toxicity.

The need for immunotherapy may vary and techniques are varied. A universally
applicable one is to maintain a proper mineral balance for immune system
function. I prefer kelp selenium - 200 mcg per day because in addition to
selenium it contains copper, a whole host of trace minerals from the sea.
Sea Sel is one brand with which I am familiar.

A case to illustrate the above follows. The subject is a 61 year old male,
retired physician and patient who had previously been poisoned by aspartame;
low calorie Kool-Aid in 1983. The problems encountered following the
original episode included a toxic cardiomyopathy, classic symptoms of methyl
alcohol poisoning, depression and Lou Gehrig's symptoms which cleared fairly
well after discontinuing aspartame. He was left with striking
hypersensitivity and hyperautoimmunicity. Typical of chemical
hypersensitivity, the patient, otherwise free of symptoms when in pristine
environments and thus would be relatively non-reactive to an infrequent
inadvertent exposure. If the the hypersensitivity was flared by other
exposures eg aspartame, it could cause violent pathologic reactions to even
a minimal subsequent chemical stimulus. His reactions included everything
commonly associated with hypersensitivity eg diabetes, neurodenenerative
disease or any allergic or auto immuno phenomena depending on the inciting
stimulus and the status of his hyper autoimmunicity at the moment. Sounds
confusing to some, but a situation well known to those so afflicted and
physicians who care for them. As experienced allergists say "in the
hypersensitive individual 'allergy' can produce any symptoms of any disease
process in any system in the body".

The subject was selected by two ticks as dinner while walking across a rest
area in Lincoln, Nebraska in August 1995. The ticks, undetected by him,
attached themselves to the back of one of his knees. The pain was soon
noticeable and at the end of his shift his wife noticed the ticks and
removed them. The atypical local reactions continued to be extreme, with
painful swelling and reddening. Flu like symptoms with fatigue, joint pains,
muscle aches and headache rapidly ensued - again an atypical immediate
response due to this aspartame induced hypersensitivity. The symptoms
increased in severity over the next 36 hours until his arrival in Portland,
Oregon, where the subject presented to the Veterans' Administration
Emergency Room with meningismus in addition to the other findings. He was
treated for Lyme Disease with Doxycycline,100 mg twice daily for 14 days
(most experts would give Doxycycline for 20-30 days at this juncture and for
a 260 lb man,100 mg dosage twice daily is a questionably inadequate dosage).

A Lyme titer was negative at that time. The response was very beneficial and
the subject took no further thought about the Lyme episode after this. Over
the ensuing years, the chemical hypersensitivity flare ups progressively
worsened: arthritis (especially in the knees) as well as various
neurological sequelae and dermatologic manifestations etc. When in
contaminated environments his blood sugars became progressively elevated but
fell to normal when in pristine environments, only to again elevate and
require vigorous insulin and oral agent treatments to control them when
chemical exposures occurred.

By late 1999 the arthritis took a turn for the worse and "joint mice"
manifested themselves in both knees. These are osteocartilaginous loose
bodies broken free from the articular surfaces that can slip in and out
between the knee joint and the bursae - under the shin at times. When in the
joints, these were quite damaging and painful. When the subject went to the
VA Emergency Room he was belittled by being offered only psychiatric care
even though he had properly and efficiently explained the meaning of all
terms used. All competent medical personnel should be able to understand the
term "joint mice" and shouldn't have to have it explained but, in this case,
even explanations couldn't avoid an attack on his sanity, in spite of the
fact these erosive processes are classical findings of knee arthritis from
recurrent Lyme meds and the patient had previously been treated in that same
emergency room for Lyme symptoms from tick bites. This was only one of
several such denials of real medical care and attempts at intimidation this
doctor was subjected to for blowing the whistle on aspartame.

This bold denial of any real therapy was contrary to the specifications by
the Lyme Disease Foundation and the CDC (Center For Disease Control) that
Lyme symptoms must be treated because of the tragic results of leaving them
untreated. The knees needless to say, thanks partly to their diligent
neglect continued to worsen. By Spring 2000, the now chronically stimulated
hyper auto immunity/chemical hypersensitivity had so worsened that a brief
Spring pollen bloom caused a devastating "flu" episode.

The diabetes blood picture usually resolved in the summer in Portland when
the pollen and fungal spore blooms were past and the weather was dry and
warm and the severe industrial pollution was relieved because the inversion
layer effects in the Williamette Valley had cleared allowing the atmospheric
pollutants to be rapidly carried away. This would usually allow the
discontinuation of all hypoglycemic agents for the summer - not so for the
summer of 2000. The blood sugar picture worsened.

Other symptoms rapidly ensued. His knees became severely disabled. Then the
left ankle turned to "mush" and lost ligamentous integrity. Other joints
began at times to show arthritic inflammation, tenosynovitis of the
shoulders occurred. Synovitis of the neck became a permanent feature. In
recurrent Lyme Disease this occurs in continuity with an autoimmune cervical
meningitis. The subject who never bruised at insulin injection sites began
to show strikingly symmetrical bruising in neat circular patterns centered
around each and every injection site. Polymyalgia and fibromyalgia symptoms
ensued. Then extreme weakness and fatigue was accompanied by cranial and
upper cervical nerve problems with partial numbness of the right side of the
face and partial facial nerve paralysis of the left nares with dilation and
fasiculation so rapid and persistent the subject felt compelled to check his
pulse to make sure this wasn't an aneurysmic phenomenon. The check revealed
the nasal fasiculations were just that and didn't coincide in any way with
pulse rate, rythym or timing.

Carefully reviewing his own medical history for a clue (his physicians at
the VA had not) revealed he had almost every symptom of recurrent Lyme
Disease. This included, by now, various types of radiculo neuropathies
throughout his body but particularly striking from the cervical nerve roots.
His neural paralysis of the right chest from an episode of the plague
contacted while medically serving an orphanage in Vietnam in 1968 worsened
to the point he was experiencing severe right shoulder pain, weakness and
trouble with his right hand control. His previous experience with the
apparently poorly trained and possibly improperly motivated personnel at the
Portland VA ER led the subject to go to the public library and on the
internet to do a ten year review of the medical literature on Lyme disease,
ehrlichiosis and related topics. A few hours well spent before his futile
visit to the Portland VA ER where he was denied treatment of any kind on the
basis that "they wouldn't treat Lyme Disease there" capped off with the lie
they couldn't draw a Lymes titer there - both untrue and irrelevant to the
subject's misery state and highly damaging disease status.

Going back to his medical cabinet, untreated by the VA, he luckily found a
supply of Doxycycline tablets unused from a previous facial injury and was
able to take them 200 mg twice/day for 5 days until his scheduled urgent
care visit at the VA. On Doxycycline, the blood sugar levels dramatically
dropped to normal. The brusing and platelet difficulties quickly resolved,
the arthritis improved incrementally day by day until almost his usual
functional level for the first time in months. The polymyalgia and cervical
synovitis cleared dramatically. The facial and cranial nerve problems
resolved, the fatigue lifted. During the 5-6 day period until his
appointment he was inadvertently away from the Doxycycline for one day and
Lyme symptoms began to relapse while he was unexpectedly caught out of town
on a trip.

This case history gives a striking picture:

1. getting virtually all the findings of recurrent Lyme Disease; 2. the
successful treatment by Doxycycline; 3. the temporary relapse whilst off
Doxycycline; 4. the complete resolution in response to the use of the high
dose Doxycycline and the greatly improved status of the subject at the
visit.

This was the best, most objective information on the subject's condition and
needs that the VA physicians would ever have. Instead of treating the
patient the VA physician gave the subject a stern lecture about treating
himself - totally ignoring his interim misery and pathology - only ordering
a Lyme titer and refusing to give any other treatment.

The subject's regular VA clinic physician also refused to see him until
January 2001, again boldly defying CDC regulations. The subject was off
Doxycycline for about a week until he could arrange for help from better
medical caregivers during which time all of the symptoms, except the facial
and cranial nerve problems recurred. Belatedly back on Doxycycline, the
problems began again to resolve somewhat but much more slowly. No competent
or caring physician would ever recommend interrupting partially completed
successful therapy of a complicated infectious problem nor concern of
allowing immuniologic reflaring of a destructive hyperautoimmine process as
the VA doctors did. The botched therapeutic regimen was not as rapidly
effective now. The structural physical damage to the neck, knees, ankles and
peripheral nerves were allowed to progress unabatted due to the diligent
neglect by the VA physicians and so far that is not promising happy results.
This unfortunate scenario and the associated Lyme literature research have
served as the inspirations for this article to illustrate some of the
problems generated in our medical care systems by its unwillingness to
factor in the tremendous damages from aspartame on a worldwide basis.

Only acknowledgement of such damage would allow aspartame sufferers to get
adequate treatment and avoid tremendous damages to themselves as well as to
the financial structure of our medical care and compensation/pension
systems. Juvenile rheumatoid arthritis is a classic case of destructive
hyperautoimmune disease: some cases even require the ultimate treatment of
hyper auto immunicity - immune ablation with chemotherapy and irradiation
followed by stem cell transplantation. The strong ties sometimes discovered
between this disease and Lyme Disease only serve to emphasize the auto
immune nature of the destructive processes of Lyme Disease. The name Lyme
Disease comes from an episode in the Lyme County area of Conneticut when
physicians investigating a cluster outbreak of juvenile rheumatoid arthritis
in the region discovered tick bites as a common factor in all cases and
conducted an epidemiiological search for a causitive agent and identified
the Lyme treponema as a possible agent. This unfortunately led to a "tunnel
vision" approach focussing on Lyme Disease as only a typical infectious
disease which firstly ignored that juvenile rheumatoid arthritis is a very
destructive hyper autoimmunicity disease. The hyper autoimmunicity involved
is a key to adequate understanding of the nature and therapy of Lyme
Disease. Secondly, the excellent information to be gleaned from the British
and European experience with the treponemae and their treatment were and are
by and large ignored. Thus pseudo scientific approaches treating the very
fallible lab tests instead of the very sick patients have often gained the
uppermost in physicians' minds with disastrous results for the patients.

The cardiac conduction system and the cardiac muscle are afflicted by both
aspartame and Lyme Disease. In Europe empiric pre surgical treatment of
dilated cardiomyopathy with antibiotic regimen for Lyme Disease has in may
instances resolved the problems without surgery. The single largest category
of patients awaiting cardiac transplantation in the US relates to patients
with dilated cardiomyopathy. Just think of the possibility of avoiding many
of those procedures if the issues of possible Lyme etiology and Aspartame
Disease are fully addressed in these cases. Other issues arising out of
aspartame toxicity are similarly expensive. Over 10 years ago, Dr Hyman
Roberts, a West Palm Beach Board Certified Internist identified cases that
wasted over $60,000 on unnecessary lab and medical diagnostic procedures
when aspartame ingestion was their only correctible medical problem.

James Bowen, M.D. c/o 1720 North Watts Portland, Oregon 97217 November 2000

The following references were used in research of this article. (For more
information on aspartame see www.dorway.com and the Aspartame Toxicity
Center, www.holisticmed.com/aspartame)
1. Oral Doxycycline For Facial Palsy Related to Lyme Disease, American
Family Physician, June 99, Vol 59, Issue 11, p323, Kirchner, Jeffrey T.
2. The Role of Genetic Factors In Autoimmune Disease: Implications for
Environmental Research, Environmental Health Perspective Supplements, Oct
99, Vol 107 Issue 5, p693, 8 p,4 charts, 2 diagrams, 1 graph, Cooper,
Miller, Glinda S.
3. Lyme Borreliosis: Basic Science and Clinical Aspects, Lancet, 4/23/94,
Vol 343, Issue 8904, p1013, 4p, 3 diagrams, 4c, Pfister, Hans-Walter,
Wilske, Bettina
4. The BDR Gene Families Of The Lyme Disease And Relapsing Fever
Spirochetes: Potential Influence On Biology, Pathogenesis, and Evolution,
Emerging Infectious Diseases, Mar/Apr 2000, Vol 6 Issue 2, p110, l3p, 2
diagrams, 2bw, Roberts, David M; Carolyn, Jason A.;Theisen, Michael,
Marconi, Richard T.
5. Identification of LFA-1 As A Candidate Autoantigen In
Treatment-Resistant Lyme Arthritis, Pediatrics, Part 2 of 2, Vol 104 Issue
2, p405, 2p, Gern, James E.
6. Arthritis, FDA Consumer, May/June 2000, Vol 34, Issue 3, p27, 6p, l
diagram, 2bw, Lewis, Carol
7. Differences Are Voiced By Two Lyme Camps At A Connecticut Public
Hearing On Insurance Coverage Of Lyme Disease, Pediatrics, Apr2000 Part 1
of 2, Vol 105 Issue 4, p855, 3p, Feder Jr., Henry M.
8. Lyme Disease Is Frequently Misdiagnosed, Modern Medicine, Sept 95, Vol
63 Issue 9, p 17, 2p, 2c, Feder,M.D., Henry M, Hunt, M.D., Margaret
9. Lyme Disease In The United Kingdom, BMJ British Medical Journal,
2/4/95, Vol 310 Issue6975, p303, 6p, 4 charts, 7c, O'Connell, Susan
10. Lyme Disease May Lead To Autoimmune Disease, World Disease Weekly Plus,
9/28/98, p15, 2p, by Sandra W. Key, News Editor with Daniel J. DeNoon &
Salynn Boyles
11. Juvenile Chronic Arthritis, Lancet, 3/28/98, Vol 351 Issue 9107, p969,
5p, 2 charts, 2c, 1bw, Woo, P.; Wedderburn, L.R.
12. Possible Cause Found For Lyme Arthritis, Science 7/31/98, Vol 281
Issue 5377, p631, 2p, 1c, Dickman, Steven
13. Treatment-Resistant Lyme Arthritis May Be Autoimmune Disease, Lancet
8/1/98, Vol 352, Issue 9125, p375, 1/3p, Morris, Kelly
14. Getting Lyme Disease To Take A Hike, FDA Consumer, Jun 94, Vol 28
Issue 5, p5, 4p, Ic, Lewis, Ricks
15. Lyme Disease: Frequently Asked Questions, Lyme Disease Foundation,
Brochure 1995




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