No wonder Amy is confused.  I  have found no data to support the figure of 25% risk of stillbirth.  The latest evidence is the RCOG (U.K) 2006.  which is very informative and exposes a few myths. MM

 

BMJ 2002;324:123-124 ( 19 January ). Editorials . Obstetric cholestasis:  Reid R, Ivey KJ, Rencoret RH, Storey B. Fetal complications of obstetric cholestasis. BMJ 1976; 1: 870-872[ISI][Medline].

Clinical studies clearly show that when obstetric cholestasis complicates pregnancies it may lead to premature births in up to 60%, fetal distress in up to 33%, and intrauterine death in up to 2% of patients.1 The cause of fetal death is acute anoxia.2

 

he risk of having a stillborn baby is 15% greater for women who have OC than for other women (Crafter, 2003: 310). Nobody is quite sure why. The baby may die because of the bile acids, which are known to cross the placenta, or as a result of being suddenly deprived of oxygen, perhaps because of placental problems.

Crafter, H. 2003. Problems of Pregnancy. Ch. 18. In D.M. Fraser and M.A. Cooper M.A. eds. Myles Textbook for Midwives. 14th edition. Edinburgh: Churchill Livingstone. pp 295-320

RCOG Jan 2006. Guideline No 43.  6.Obstetricians should be aware that the stillbirth rate for O.C is comparable to the general population. The risk of stillbirth in untreated OC is unclear. 

9. obstetricians should be aware that there is insufficient data to support or refute the popular practice of early induction (37 wks) aimed at reducing late stillbirth. It is not evidenced based.

There is also no form of fetal monitoring or treatment that can forecast foetal death.   

 

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