One of the fundamental problems of using lactate clearance as a sole 
resuscitation target is that a normal lactate (< 2mM/L) can be present in 
25-50% of septic shock patients. The mortality of these patients is 20-50% even 
with aggressive therapy. These observations indicate that using lactate and 
ScvO2 are complimentary endpoints and not mutually exclusive.1-8

1.         Levraut J, Ciebiera JP, Chave S, et al. Mild hyperlactatemia in 
stable septic patients is due to impaired lactate clearance rather than 
overproduction. Am J Respir Crit Care Med. Apr 1998;157(4 Pt 1):1021-1026.
2.         Wacharasint P, Nakada TA, Boyd JH, Russell JA, Walley KR. 
Normal-Range Blood Lactate Concentration in Septic Shock is Prognostic and 
Predictive. Shock. May 1 2012.
3.         Dugas AF, Mackenhauer J, Salciccioli JD, Cocchi MN, Gautam S, 
Donnino MW. Prevalence and characteristics of nonlactate and lactate expressors 
in septic shock. J Crit Care. Aug 2012;27(4):344-350.
4.         Cannon CM, Holthaus CV, Zubrow MT, et al. The GENESIS Project 
(GENeralized Early Sepsis Intervention Strategies): A Multicenter Quality 
Improvement Collaborative. J Intensive Care Med. Aug 17 2012.
5.         Hernandez G, Castro R, Romero C, et al. Persistent sepsis-induced 
hypotension without hyperlactatemia: Is it really septic shock? J Crit Care. 
Dec 1 2010.
6.         Vallet B, Chopin C, Curtis SE, et al. Prognostic value of the 
dobutamine test in patients with sepsis syndrome and normal lactate values: a 
prospective, multicenter study. Crit Care Med. Dec 1993;21(12):1868-1875.
7.         Rhodes A, Lamb FJ, Malagon I, Newman PJ, Grounds RM, Bennett ED. A 
prospective study of the use of a dobutamine stress test to identify outcome in 
patients with sepsis, severe sepsis, or septic shock. Crit Care Med. Nov 
1999;27(11):2361-2366.
8.         De Backer D, Creteur J, Silva E, Vincent JL. The hepatosplanchnic 
area is not a common source of lactate in patients with severe sepsis. Crit 
Care Med. Feb 2001;29(2):256-261.

Lactate ≠ScvO2

Kathrina


From: [email protected] 
[mailto:[email protected]] On Behalf Of Erik Benjamin 
Kulstad
Sent: Friday, May 03, 2013 12:59 AM
To: Townsend, Sean, M.D.
Cc: [email protected]
Subject: Re: [Sepsis Groups] Noninvasive EGDT

I suspect new insights to this question will be answered by the ProCESS study, 
which is about to enroll the last patient...

https://crisma.upmc.com/processtrial/info2.asp


On Wed, May 1, 2013 at 1:47 PM, Townsend, Sean, M.D. 
<[email protected]<mailto:[email protected]>> wrote:
Did you happen to know that all patients in that trial received a central line 
and that CVP was optimized using it?  I think therefore we can’t call that 
trial “non-invasive.”

Also, although that trial enrolled 300 patients, the intervention did not 
differ until the level of optimizing ScvO2, i.e. all patients that benefited 
from CVP, a fluid bolus, antibiotics etc. and met targets progressively dropped 
out of the running to actually compare lactate clearance to ScvO2.  Thus, in 
the end, 29 patients got a head to head comparison of lactate clearance to 
ScvO2 optimization.

This would suggest that enrollment in the trial should have been 3000 patients 
in order to test the actual difference in intervention at the power requirement 
of 300.

One can make the argument reasonably that the assertion of non-inferiority is 
underpowered by a factor of 10.

One must also wonder about extraordinarily high lactates.  None were enrolled 
in the trial.  So, if I have a lactate of 9 and clear it by 10% to 8.1 should I 
be comfortable that I have hit my resuscitation targets?  Doesn’t make me so 
comfortable.

Sean R. Townsend, M.D.
Vice President of Quality & Safety
California Pacific Medical Center
2330 Clay Street, #301
San Francisco, CA  94115
email [email protected]<mailto:[email protected]>
office (415) 600-5770<tel:%28415%29%20600-5770>
fax (415) 600-1541<tel:%28415%29%20600-1541>

From: 
[email protected]<mailto:[email protected]>
 
[mailto:[email protected]<mailto:[email protected]>]
 On Behalf Of Ram Parekh
Sent: Tuesday, April 30, 2013 1:50 PM
To: Vipul Kella
Cc: 
[email protected]<mailto:[email protected]>
Subject: Re: [Sepsis Groups] Noninvasive EGDT

We have at our hospital and at most of the GNYHA hospitals in the New York area.

This protocol is based on the non-inferiority study of lactate clearance by 
Jones/Shapiro and was implemented with our current Stop Sepsis collaborative 
which has given ED providers the option of utilizing the 'invasive' or 
'non-invasive protocol' as EGDT options. Thus, the protocol was simultaneously 
implemented in over 50 hospitals at the same time.
On Mon, Apr 29, 2013 at 9:57 AM, Vipul Kella 
<[email protected]<mailto:[email protected]>> wrote:
Has anyone implemented the noninvasive EGDT protocol at their hospital?  What 
was your experience?

--
Vipul Kella, MD FACEP
Medical Emergency Professionals (MEP)

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--
Erik Kulstad, M.D., M.S.
Research Director
Advocate Christ Medical Center
Dept. of Emergency Medicine
4440 W. 95th St.
Oak Lawn, IL 60453
Clinical Associate Professor
University of Illinois, Chicago

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