Here below is a fascinating long extract by CRAIG-OXLEY culled from
<http://z13.invisionfree.com/THE_UNHIVED_MIND/index.php?showtopic=4646>
discussing virus contradictions, environmental toxins and viral causation
with special regard to polio in a most reasoned manner. With references.
JOhn
Pesticide Composite: Summary
Just over three billion pounds of persistent pesticides are represented in
the graph below. (SEE:
<http://z13.invisionfree.com/THE_UNHIVED_MIND/index.php?showtopic=4646>)
Virtually all peaks and valleys correlate with a direct one-to-one
relationship with each pesticide as it enters and leaves the US market.
Generally, pesticide production precedes polio incidence by 1 to 2 years. I
assume that this variation is due to variations in reporting methods and
the time it takes to move pesticides from factory to warehouse, through
distribution channels, onto the food crops and to the dinner table.
A composite of the three previous graphs, of the persistent pesticides —
lead, arsenic, and the dominant organochlorines (DDT and BHC) — is
represented in the following:
These four chemicals were not selected arbitrarily. These are
representative of the major pesticides in use during the last major polio
epidemic. They persist in the environment as neurotoxins that cause
polio-like symptoms, polio-like physiology, and were dumped onto and into
human food at dosage levels far above that approved by the FDA. They
directly correlate with the incidence of various neurological diseases
called “polio” before 1965. They were utilized, according to Biskind,
in the “most intensive campaign of mass poisoning in known human
history.”
Virus Causation
A clear, direct, one-to-one relation between pesticides and polio over a
period of 30 years with pesticides preceding polio incidence in the context
of the CNS related physiology just described, leaves little room for
complicated virus arguments, even as a co-factor, unless there exists a
rigorous proof for virus causation. Polio shows no movement independent
from pesticide movement as one would expect for the virus model.
Medical propagandists promote images of a predatory, infectious virus,
invading the body and quickly replicating to a level that causes disease,
however, in the laboratory, poliovirus does not easily behave in such a
predatory manner. Attempts to demonstrate virus causation are performed
under extremely artificial and aberrant conditions.
Poliovirus causation was first established in the mainstream mind by
publications of an experiment by Landsteiner and Popper in Germany,
1908-1909.13 Their method was to inject a pulverized purée of diseased
brain tissue into the brains [correction, intraperitoneally] of two
monkeys. One monkey died and the other was sickened.
Proof of poliovirus causation was headlined by orthodoxy. This, however,
was an assumption — not a proof — of virus causation. The weakness of
this method is obvious to everyone except certain viropathologists and has
recently been criticized by the molecular biologist Peter Duesberg
regarding a modern-day attempt to establish virus causation for kuru,
another CNS disease.14 Since 1908, the basic test has been repeated
successfully many times using monkeys, dogs and genetically altered mice.
The injected material has even been improved — scientists now use a
saline solution containing purified poliovirus. However, a crucial weakness
exists — polio epidemics do not occur via injections of poliovirus
isolate into the brains of the victims through a hole drilled in their
skull — except, of course, in laboratories and hospitals.
If injection into the brain is really a valid test for causation then it
should serve especially well as a proof for pesticide causation. I propose
that pesticides be injected directly into the brains of test animals. If
paralysis and nerve degeneration subsequently occur, we then would have
proved that pesticides cause polio.
Going further, towards much higher standards of proof than those used to
prove virus causation, pesticides could be fed to animals and found to
cause CNS disease. This has already been done with DDT and the histology of
the spine and brain was poliomyelitis. Virus proofs require injection,
often intracranial, to get any reaction from the experimental animal. It is
axiomatic that a theory is only as good as its ability to predict future
events. I predict that such a test would prove pesticides to be the most
reliable causative factor.
The injection of purée of diseased brain tissue into the brains of dogs
was the method preferred by Louis Pasteur to establish virus causation with
rabies, another CNS disease. A recent, definitive biography of Pasteur
finds him to be a most important publicist for germ theory, a crucial
promoter for the notion that rabies is caused by a virus. Unfortunately,
his rabies experiments were biased and unsupported by independent studies.15
Therefore, in my opinion, even a cofactor theory, where pesticides catalyze
predatory poliovirus activity, or where pesticides weaken the immune system
to allow opportunistic predatory poliovirus activity, cannot stand up to
simple, common sense explanations that include the concept of a symbiotic
virus. Neurotoxins are enough of a cause for neurological disease.
The most obvious theory — pesticide causation — should be the dominant
theory. But the opposite exists, a pervasive silence regarding pesticide
causation juxtaposed against a steady stream of drama regarding virus
causation. In light of the evidence presented herein, the silence could
ultimately discredit mainstream medical science, institutions of the
environmental movement, and the World Health Organization (which directs
both DDT application for mosquito campaigns and polio vaccination,
world-wide).
Virus Presence
When the symptoms of polio are recognized, there is often a claim of virus
presence in the body of the polio victim. Sometimes a virus is found.
Sometimes that virus is an enterovirus (a virus of the digestive tract).
Sometimes that enterovirus is a poliovirus. During polio epidemics,
orthodoxy blames the poliovirus, and therefore, my argument for the
innocence of the poliovirus requires an explanation of these claims of
virus presence and the presence of an agent called the poliovirus. Here are
three points:
a) Economic Impetus: During the great epidemic of 1942-1962 polio victims
were diagnosed with poliovirus-caused polio, regardless of whether or not
the poliovirus was found, because the NFIP (March of Dimes) funds paid only
for this kind of polio. Therefore, if patients were going to spend time
hospitalized, in iron lungs and undergoing therapy, it would have been
economically imperative for the hospital to diagnose them in this way.16
Thus, presence of poliovirus in poliomyelitis was rarely determined in
order to arrive at a diagnosis of polio.
Other Pathogens: Even if one believes in virus culpability, other viruses
are also claimed by orthodoxy to be the cause of polio-like CNS diseases
which are "clinically indistinguishable" from polio. In the 1940-50s,
relatively few polio victims were confirmed technically for presence of the
poliovirus. In 1958, a laboratory analyses of 222 diagnosed polio victims
(Detroit epidemic) found poliovirus in only 51% of the cases. When multiple
pathogens are hunted, a mix of pathogens, multiple viruses, fungi, and
bacteria, can be associated with a single diagnosed case of polio.
QUOTE
Coxsackievirus and echoviruses can cause paralytic syndromes that are
clinically indistinguishable from paralytic poliomyelitis. (John H. Menkes,
Textbook Of Child Neurology, 5th ed., (1995) p420)
During a polio epidemic, such cases, would have likely been diagnosed as
"polio". After the 1970s, with the supposed approaching extinction of the
poliovirus, such cases would have been diagnosed as encephalitis or
meningitis.
c) Benign Virus: The poliovirus is considered to have been endemic
throughout the world going back to ancient times, yet this is not the case
with paralytic polio. According to Arno Karlen, author of Man and Microbes,
the “polio virus lives only in people; it probably adapted to the human
small intestine countless millennia ago.” He continues, “. . . some
historians have claimed that [paralytic] polio goes back to ancient Egypt;
it may, but the evidence is thin.”20
Karlen makes a lot of sense here in view of the pesticide graphs,
Biskind’s arguments and ancient historians describing paralysis from the
inhalation of vaporized chemicals during blacksmithing operations. However,
Karlen goes on to write that “the first undisputed case dates from the
late eighteenth century.” This statement, however, must be invalid (in
its attempt to establish polio images that have a basis in early history)
because of Menkes’ statement (above) that other viruses can also be
causative for polio symptoms and because common industrial poisons such as
arsenic and lead compounds can cause polio-like symptoms. Poisoning, as a
method of assassination has also been frequently employed. It is not
unreasonable to assume that unsuccessful poisonings may have left their
victims paralyzed. Thus, Karlen’s offer of an undisputed case as early as
the late 18th century can be no more than a guess.
Orthodox medical literature can offer no evidence that the poliovirus was
anything else than benign until the first polio epidemic, which occurred in
Sweden in 1887. This small epidemic occurred 13 years after the invention
of DDT in Germany, in 1874, and 14 years after the invention of the first
mechanical pesticide crop sprayer, which was used to spray formulations of
water, kerosene, soap and arsenic. The epidemic also occurred immediately
following an unprecedented flurry of pesticide innovations. This is not to
say that DDT was the actual cause of the first polio epidemic, as arsenic
was then in widespread use and DDT is said to have been merely an academic
exercise. However, DDT or any of several neurotoxic organochlorines already
discovered could have caused the first polio epidemic if they had been used
experimentally as a pesticide. DDT’s absence from early literature is
little assurance that it was not used.
We need to remember that the poliovirus is an enterovirus. There are at
least 72 known enteroviruses discovered to date. According to Duesberg,
many enteroviruses are harmless “passenger viruses.”21 In view of the
material presented here, probably unknown to Duesberg, it is reasonable
that we also view poliovirus as harmless outside of extreme laboratory
conditions.
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