[RE]CS>BB Archives: Food poisoning, botulinum, CS spectacular

[Brooks Bradley]

Dear Malcolm,
The actual damage inflicted by botulinum is from the nerve toxins released as a result of the metabolism of the pathogen itself....not from some active component designed to enervate a target victim. These toxins do not give evidence of being a life form. Colloidal Silver has demonstrated (at least in our tests) no
observable control over the toxin proper.....only upon the presenting bacterial colony.
This being the case, it is reasonable to assume that "late-stage" cases would be, immediately, life-threatening and as such would demand antitoxin---among other modalities, in order to control/prevent pulmonary paralysis----and death. Because of the very painful presentations in the earlier stages the victim is unlikely to accept delays in attention. Fortunately, several options are, usually, open for successful treatment.
Unless one ingests a rather large quantity of toxin-laden foodstuff, the immediate
control of the reproductive capacity of the pathogen should be quite adequate for a favorable resolution. Stopping the bacterial reproduction, stops the increase in the damaging nerve toxin. While the Herxheimer's Reaction may be very uncomfortable/painful, it would be very unlikely to present life-threatening conditions. The cardinal consideration for Botulism is
to STOP the pathogen from reproducing and expanding volumetrically......AS RAPIDLY AS YOU CAN.
In answer to you query relating to pleomorphism....we have never encountered evidence
for mutations----either way (virus to bacteria or bacteria to virus) among any of the pathogens presenting in the alimentary tract of humans.
Neither have we detected pathogenic, companion spore forms in botulinum insults (this does not mean they could not be present just that we have not detected them).
However, if pathogenic spore forms were acceptable companions to a majority of botulinum presentations, it is reasonable to assume they would be self-evident to some of the researchers-----during the immediately past decades.
My apologies for being unable to furnish you a more detailed reply, but I must go now.
Sincerely, Brooks.



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---------[ Received Mail Content ]----------

Subject : CS>BB Archives: Food poisoning, botulinum, CS spectacular

Date : Sun, 22 Jun 2008 02:24:34 +0800

From : "Rowena" <new...@aapt.net.au>

To : <silver-list@eskimo.com>





Brooks,



Thanks for your post and underlying experiments, however I remain confused

about the roles of: first, botulinum the active bacterium itself, second,

the debris from dead bacterial residue and thus the notorious "Herx" effect,

and third the toxin excreted by the live bacterium under a 'favorable' i.e.

anaerobic non-acidic environment - for instance most conspicuously home

canning.



I had accepted the third scenario that only the toxin was a threat, and this

opinion was bolstered by the idea that stomach acid would summarily destroy

any live botulinae, but that the toxin, if already present in the food,

would survive the torments of cooking or canning. Apparently my beliefs,

fostered by the dept of agriculture pamphlets on home food preservation, are

overly simplistic.



Can CS, either particulate or ionic, deactivate the toxin itself? Or is the

toxin relatively short lived? Or does CS block its normal pathways, or does

Gatorade facilitate them? Does the opportunity afforded the bacteria to

'bloom' account in part or in whole for variations in response to CS

therapy? If so, how does it bloom in the presence of stomach acid? Or does

it instead hold out until it enters the gut? Pleomorphism, spore

forms??



Confusion!!



Take care, Malcolm



At 08:36 PM 6/7/06 -0600, you wrote:



I agree with Marshall's general proposition. About nine years ago we

conducted rather detailed evaluations of the effects of EIS type colloidal

silver, as an address against bacterial/Viral alimentary presentations. We

chose Botulinum as the primary; subject for our principal investigations

(botulinum is the most dangerous among all of the types we investigated).

Active research revealed that concentrations of CS as low as 3 ppm were

effective, but rather large volumes were required for rapid-onset positive

results (about 10 ounces initial dosage, followed by another 10 ounces in

approximately 20 minutes). Favorable response WAS NOT a direct effect of

linear increase of PPM strength; e.g. 10 ppm CS yielded favorable results

4X as rapidly, as did 2 ppm concentrations ..for equivalent volumetric

measure. We did not encounter a single case of Jarisch-Herxheimer's

reaction from the 60 cases involved among our volunteer, experimental,

population. I notice various speculations, from among the immediate

previous postings, which speculate on the potential dangers of Herx

reactions from possible toxic components among the bacterial debris residue.



While such does appear a possibility, actual practice did not produce such

an occurrence .in our evaluations. Our staff postulated a number of

speculations for the actual circumstance! We experienced ..the most

acceptable being that the debris was, actually, less toxic to the victim's

alimentary system - than one might initially presume. Additionally, it is

speculation, only, as to the magnitude of possible "toxic" debris moving

through the epithelial tissue into the circulation system - where serious

consequences could occur. Botulism appears to be a very opportunistic

reproducer and time is crucial for it effecting an explosive bloom capable

of a terminal insult. Our research tends to add credibility to this

assumption, as any CS-based measures instigated, halted continued

reproduction in brief time windows (usually in SPECTACULAR FASHION).

Sometimes effecting total cramping relief within eight to ten minutes.

Interestingly, in those cases where CS was combined with Gatorade, the

favorable response was measurably reduced - indicating a consequential

degree of chloride combination with some of the ionic component - possibly.

In any case, the solutions containing the higher content of particulate

silver were measurably superior in achieving favorable results. EIS colloid

has demonstrated to be without EQUAL AS AN EXPERIMENTAL PROTOCOL in

addressing all forms of food-related bacterial or viral poisons presenting

ANYWHERE in the alimentary tract ..at least in our experimental researches.

Others way have encountered different results. My family members never go

out to eat ANYWHERE without carrying a small bottle of 20 ppm Colloidal

Silver such has saved many of us much misery and eliminated the prospects of

serious consequences - over the immediately-past eight or nine years. My

apologies for such a lengthy post but I hope this information will be

favorably considered by interested members in their personal experimental

researches.



Sincerely, Brooks Bradley.

Eric Harborne Research Foundation.



p.s. In the one circumstance where we were able to include a control

component (multiple numbers of people presenting simultaneously), among the

7 cases admitted to the emergency room of the local hospital, all but one

required hospitalization. Four required retention for more than 5 days.

All required multiple "balanced fluid electrolyte" IVs. One required many

IVs and 11 days hospital confinement. The 5 cases involving the volunteers

who chose to be involved in our experimental protocol yielded: 2 who were

completely stabilized within 20 minutes of oral ingestion of 4 ounces of 10

ppm Colloidal Silver; 1 was stabilized and pain-free within 45 minutes of

receiving the same protocol; 2 were pain-free, with no nausea symptoms,

presenting only traces of bowel looseness two hours after receiving the

same protocol as the other three.



To: silver-list@eskimo.com

Subject: Re: CS>Food poisoning

Date: Tue, 06 Jun 2006 10:17:29 -0400



Kandee Edge wrote:



My mother in law just got food poisoning. She is having a terrible time

with it and cannot take any electrolytes, a.k.a. Gatorade. My question is,

if I have 3-5ppm CS, how much should she take. I was going to give her some

in hopes it would help, but wasn't sure of the dosage. Does anyone have any

experience or testimonies with CS and food poisoning?









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