----------
Iron Silver Cells [battery]
These have a very high energy density, and a good cycle life. It is an
alkaline battery with a KOH electrolyte, and the working materials are
silver oxide and metallic iron.
"BE" produces NaOH and probably KOH as well.
Add Silver to that deal ?
It it were done using silver electrodes for iontophorosis [direct injection
of metallic ions and various medicines using electricity ]..ya "might" get
metallic Iron deposits to "discharge" into some soluble byproduct or the
other...or maybe just silver plate them. [er]
Then there's Tetrasilver Tetroxide....a form of silver oxide...and that
black stuff on one of the electrode in a DC CS generator, that which makes
a batch go yellow if it's formed in the water.
Rumor has it that the brown murky reaction when adding Hydrogen Peroxide to
a batch of CS too soon is the process by which Tetrasilver Tetroxide is made.
Concentrate that in a centrifuge?
Anyhoo, Silver and Iron reactions happen and likely bears looking into
more deeply.
Could it be that these Iron deposits ARE acting like a battery and it's
the stray power discharge that interferes with the normal electro-chemical
[neurotransmitter] function of snapping synapses? [ turning them into
napping synapses?]
VEDDY Interestink: [and WAY over my head
] http://en.wikipedia.org/wiki/Hypochlorous_acid
In <http://en.wikipedia.org/wiki/Organic_synthesis>organic synthesis, HOCl
converts <http://en.wikipedia.org/wiki/Alkene>alkenes to
<http://en.wikipedia.org/wiki/Chlorohydrin>chlorohydrins.<http://en.wikipedia.org/wiki/Hypochlorous_acid#cite_note-1>[2]
In <http://en.wikipedia.org/wiki/Biology>biology, hypochlorous acid is
generated in activated <http://en.wikipedia.org/wiki/Neutrophil>neutrophils
by myeloperoxidase-mediated peroxidation of chloride ions, and contributes
to the destruction of <http://en.wikipedia.org/wiki/Bacteria>bacteria
<http://en.wikipedia.org/wiki/Hypochlorous_acid#cite_note-2>[3]<http://en.wikipedia.org/wiki/Hypochlorous_acid#cite_note-ref93-3>[4]<http://en.wikipedia.org/wiki/Hypochlorous_acid#cite_note-ref3-4>[5]
and this is used in water treatment such as the acid being the active
sanitizer[<http://en.wikipedia.org/wiki/Wikipedia:Citation_needed>citation
needed] in hypochlorite-based swimming pool products.
In food service and water distribution, specialized equipment to generate
weak solutions of HOCl from water and salt is sometimes used to generate
adequate quantities of safe (unstable) disinfectant to treat food
preparation surfaces and water
supplies.<http://en.wikipedia.org/wiki/Hypochlorous_acid#cite_note-5>[6]<http://en.wikipedia.org/wiki/Hypochlorous_acid#cite_note-6>[7
Inhibition of glucose oxidation
[mention of iron and sulphur..silver and sulphur? think tarnish]
In 1948, Knox et
al.<http://en.wikipedia.org/wiki/Hypochlorous_acid#cite_note-ref48-22>[23]
proposed the idea that inhibition of
<http://en.wikipedia.org/wiki/Glucose>glucose oxidation is a major factor
in the bacteriocidal nature of chlorine solutions. He proposed that the
active agent or agents diffuse across the cytoplasmic membrane to
inactivate key <http://en.wikipedia.org/wiki/Thiol>sulfhydryl-containing
<http://en.wikipedia.org/wiki/Enzyme>enzymes in the
<http://en.wikipedia.org/wiki/Glycolysis>glycolytic pathway. This group was
also the first to note that chlorine solutions (HOCl) inhibit
<http://en.wikipedia.org/wiki/Thiol>sulfhydryl
<http://en.wikipedia.org/wiki/Enzyme>enzymes. Later studies have shown
that, at bacteriocidal levels, the
<http://en.wikipedia.org/wiki/Cytosol>cytosol components do not react with
HOCl.<http://en.wikipedia.org/wiki/Hypochlorous_acid#cite_note-ref1-0>[1]
In agreement with this, McFeters and
Camper<http://en.wikipedia.org/wiki/Hypochlorous_acid#cite_note-ref59-35>[36]
found that <http://en.wikipedia.org/wiki/Aldolase>aldolase, an
<http://en.wikipedia.org/wiki/Enzyme>enzyme that Knox et
al.<http://en.wikipedia.org/wiki/Hypochlorous_acid#cite_note-ref48-22>[23]
proposes would be inactivated, was unaffected by HOCl
<http://en.wikipedia.org/wiki/In_vivo>in vivo. It has been further shown
that loss of <http://en.wikipedia.org/wiki/Thiol>sulfhydryls does not
correlate with
inactivation.<http://en.wikipedia.org/wiki/Hypochlorous_acid#cite_note-ref46-21>[22]
That leaves the question concerning what causes inhibition of
<http://en.wikipedia.org/wiki/Glucose>glucose oxidation. The discovery that
HOCl blocks induction of
<http://en.wikipedia.org/wiki/%CE%92-galactosidase>²-galactosidase by added
<http://en.wikipedia.org/wiki/Lactose>lactose<http://en.wikipedia.org/wiki/Hypochlorous_acid#cite_note-ref8-36>[37]
led to a possible answer to this question. The uptake of radiolabeled
substrates by both ATP hydrolysis and proton
<http://en.wikipedia.org/wiki/Co-transport>co-transport may be blocked by
exposure to HOCl preceding loss of
viability.<http://en.wikipedia.org/wiki/Hypochlorous_acid#cite_note-ref1-0>[1]
From this observation, it proposed that HOCl blocks uptake of nutrients by
inactivating transport
proteins.<http://en.wikipedia.org/wiki/Hypochlorous_acid#cite_note-ref1-0>[1]<http://en.wikipedia.org/wiki/Hypochlorous_acid#cite_note-ref9-20>[21]<http://en.wikipedia.org/wiki/Hypochlorous_acid#cite_note-ref59-35>[36]<http://en.wikipedia.org/wiki/Hypochlorous_acid#cite_note-ref13-37>[38]
The question of loss of glucose oxidation has been further explored in
terms of loss of respiration. Venkobachar et
al.<http://en.wikipedia.org/wiki/Hypochlorous_acid#cite_note-ref96-38>[39]
found that succinic dehydrogenase was inhibited in vitro by HOCl, which led
to the investigation of the possibility that disruption of
<http://en.wikipedia.org/wiki/Electron_transport>electron transport could
be the cause of bacterial inactivation. Albrich et
al.<http://en.wikipedia.org/wiki/Hypochlorous_acid#cite_note-ref3-4>[5]
subsequently found that HOCl destroys
<http://en.wikipedia.org/wiki/Cytochrome>cytochromes and
******<http://en.wikipedia.org/wiki/Iron-sulfur_cluster>iron-sulfur
clusters****** and observed that oxygen uptake is abolished by HOCl and
adenine nucleotides are lost. Also observed was, that irreversible
oxidation of <http://en.wikipedia.org/wiki/Cytochrome>cytochromes
paralleled the loss of respiratory activity. One way of addressing the loss
of oxygen uptake was by studying the effects of HOCl on succinate dependent
<http://en.wikipedia.org/wiki/Electron_transport>electron
transport.<http://en.wikipedia.org/wiki/Hypochlorous_acid#cite_note-ref43-39>[40]
Rosen et
al.<http://en.wikipedia.org/wiki/Hypochlorous_acid#cite_note-ref79-33>[34]
found that levels of reductable
<http://en.wikipedia.org/wiki/Cytochrome>cytochromes in HOCl-treated cells
were normal, and these cells were unable to reduce them. Succinate
dehydrogenase was also inhibited by HOCl, stopping the flow of electrons to
oxygen. Later
studies<http://en.wikipedia.org/wiki/Hypochlorous_acid#cite_note-ref71-31>[32]
revealed that Ubiquinol oxidase activity ceases first, and the still-active
<http://en.wikipedia.org/wiki/Cytochrome>cytochromes reduce the remaining
quinone. The <http://en.wikipedia.org/wiki/Cytochrome>cytochromes then pass
the <http://en.wikipedia.org/wiki/Electron>electrons to
<http://en.wikipedia.org/wiki/Oxygen>oxygen, which explains why the
<http://en.wikipedia.org/wiki/Cytochrome>cytochromes cannot be reoxidized,
as observed by Rosen et
al.<http://en.wikipedia.org/wiki/Hypochlorous_acid#cite_note-ref79-33>[34]
However, this line of inquiry was ended when Albrich et
al.<http://en.wikipedia.org/wiki/Hypochlorous_acid#cite_note-ref2-7>[8]
found that cellular inactivation precedes loss of respiration by using a
flow mixing system that allowed evaluation of viability on much smaller
time scales. This group found that cells capable of respiring could not
divide after exposure to HOCl.
..and a WHOLE BUNCH MORE..............................
Humm "e.g. You can work out that iron II hydroxide is insoluble and that
iron II sulfate and sodium hydroxide [the other "BE" electrochemical] are
soluble "
Exactly what sort of Iron ARE those Iron deposits?
[Hook yerself up and have a boiled egg with onions?]
For better or worse, "BE" just might do "something" to MS and Silver play
some sort of role....Helliphino Rhinopreposterous [a very confused
aminal] what.
..now for a big bowl of Possibility Soup and see what comes out the other end.
Ode
At 04:58 PM 7/19/2010 -0400, you wrote:
Classification: UNCLASSIFIED
Caveats: NONE
Some Doctor overseas I think developed a MS cure on idea that
MS was caused by constriction on blood vessels in brain and Iron deposits.
Developed cure for his Wife if I remember right.
I some of this a time back misplaced the original one but others are test it
also.
Found the article Here it is with several others on subject.
I was very interested because I lost a Sister to MS.
If short cut does not come through look up, ends up it was an Italian
Doctor.
Doctor cures wife of MS
http://www.gizmag.com/ccsvi-multiple-sclerosis-ms-cure-zamboni/13447/
http://www.everydayhealth.com/blog/trevis-life-with-multiple-sclerosis-ms/a-
controversial-new-cure-for-multiple-sclerosis/
-http://singularityhub.com/2010/03/22/cure-for-multiple-sclerosis-verified-i
n-latest-research-trials-coming-soon/
Bob
http://www.academicjournals.org/sre/abstracts/abstracts/abstracts2007/Au
g/Iroha%20et%20al.htm
Antibacterial efficacy of colloidal silver alone and in combination with
other antibiotics on isolates from wound Infections
"A total of ten clinical bacterial isolates comprising five isolates of
Escherichia coli isolated from surgical wound patients at the Federal
Medical Centre, Abakaliki and five isolates of Staphylococcus aureus
isolated from the wound sites of burnt patients at the University of
Nigeria Teaching Hospital, Enugu were evaluated for their susceptibility
to colloidal silver at 5 and 20 ppm using agar dilution method and the
killing rate technique. Also, interaction studies between colloidal
silver and some conventional antibiotics were carried out against the
clinical isolates of Escherichia coli and S. aureus using disc diffusion
technique. The result of the study shows that the test organisms were
sensitive to the colloidal silver at both concentrations and also to
some of the antibiotics in the paper disc. The killing rate studies
revealed that the colloidal silver is highly bactericidal against the
test isolates at both concentrations. The drug interaction study showed
no antagonism, indicating that concomitant use of colloidal silver with
these antibiotics may not affect the therapeutic efficacy of either of
these agents."
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