I have found it to be AMAZING!!!! Dissolve a teaspoon of Xylitol in 8 oz.
of Colloidal silver(or distilled water) and put into an empty nasal sprayer.
The results are instant!!!! Now I can live with two cats.
TJ Garland, CMO supplier
there are no incurable illnesses-only incurable people.
Normal nasal cleaning is very much like cleaning the kitchen floor. When
the floor is dirty the first thing we do is sweep it. It's the same with the
nose. The broom for the nose are the microscopic hairs called cilia that
protrude from the cells that line our airway. We also have the benefit of
mucus in the nose, like the saw dust or other chemicals that we sometimes
put on floor to trap the dust and other material we want removed. We
normally make about a teaspoon of mucus every five minutes. The mucus is
sticky and traps almost all of the foreign particles that we breathe. The
cilia beat between 8 and 12 times a second and it takes them about 15
minutes to literally sweep the mucus to the back of the nose where we
swallow it. This sweeping works twenty-four hours a day, seven days a week
and is usually very effective. There are, however, some environmental
factors that effect this sweeping:
Dry air and not drinking enough water makes the mucus drier and harder to
clear.
*
Most colds occur in the winter when we dry our air with central
heating and don't have the same urge to drink that we have in the summer.
*
This is the season for most ear infections as well as colds.
The highest incidence of ear infections in this country, and probably the
world, is in the Native American children in Alaska. Tribal elders tell us
that before these people were "civilized" they did not have problems with
their ears. Part of the civilizing was putting them in homes instead their
traditional houses that were more open to the elements. The relative
humidity in the winter in their traditional environment is close to 100%
while that in a well insulated house is closer to 30% at best. These people
had adapted over the thousands of years they lived in this climate, and are
having trouble with the newer, nicer, but dryer environment. One of the
practices these native peoples used to have was wrapping their babies warmly
and taking them out into the cold every day. Cold air means that the nose
needs a lot more blood to warm the air, and that means more fluid in the
nose that helps to wash it out. Doctors think that these people get ear
infections because they are genetically disposed to them by the shape of the
Eustachian Canal in the back of the nose. But the same tribal elders point
out that children from the same genetic pool living in a more traditional
environment in Nome's sister city of Provideniya, Siberia do not have
trouble with their ears today. Our doctors say they are just not diagnosed,
but it does not take a doctor to tell that a screaming child that pulls on
his ear or complains of an ear ache has a problem, especially when it drains
a few days later. Tribal elders usually have more wisdom than we like to
acknowledge.
Besides the environmental factors, things that hurt the cilia will also lead
to more problems.
*
Cigarette smoke, even passive smoke from someone else, is toxic to
the cilia. Smokers and their families are only sweeping with half a broom or
less. If there is enough smoke the cilia don't sweep at all. This is why
some pediatricians consider it child abuse for parents to smoke when their
children are around.
The cilia and mucus working together clean out the great majority of
bacteria and other pollutants that enter the nose. When we look at the back
of the nose with a microscope we can see the bacteria caught in the mucus
and riding on top of the cilia. These bacteria are not going to cause
problems. In normal cleaning they are swallowed along with the mucus and are
killed by the acid in our stomachs. In order to cause infections bacteria
must find a place in the back of the nose where there is no mucus protecting
the cells. Even most of these bacteria are killed by our own antibacterial
substances in the airway surface fluid that bathes these cells.
If we cannot remove the dirt from the kitchen floor with sweeping then
we need to get out the soap and water. It's the same with the nose.
Irritants in the nose, whether they are infectious (viruses and bacteria) or
allergenic, trigger special cells called mast cells. These cells release
granules that contain histamine and an enzyme called tryptase. The tryptase
acts like soap and makes the mucus thinner. Histamine does four things in
the back of the nose:
*
it opens blood vessels in the nose so that they leak ? the water for
this washing.
*
it makes more mucus to trap more of the pollutants.
*
it irritates us so we sneeze more and get rid of it.
*
it causes broncho-constriction that protects the lungs from this
pollution.
The fluid, or plasma, comes from under the surface/epithelial cells
than line our nasal passages. This fluid percolates up around these cells
bathing them and winds up under the mucus which it virtually lifts up and
washes out making room for the new clean mucus. It is a very efficient
washing mechanism which we should try to help. Christer Svensson, a Swedish
physician who has extensively researched the role of histamine in the nose,
points out that this is a normal defensive process.
But researchers in the 1940's weren't interested in defenses. They found
that histamine was associated with a runny nose and that antihistamines
stopped the nose from draining. They sanitized the snotty nose by turning
off its defensive washing. Two things happened in the early 1970's to
promote these problems:
*
Antihistamines and decongestants were made available over the counter
and began to be heavily advertised on television.
*
Entitlement programs like Medicaid made these drugs available to our
poorer populations.
These drugs act to block our normal, but bothersome, nasal cleaning.
Antihistamines block the effects of histamine so the washing never gets
turned on and decongestants close down the blood vessels that histamine has
opened so the water gets turned off. It does not require a whole lot of
training to see that if we stop the washing we will have more dirt. This is
also suggested by the side effect studies of one of these drugs. Loratadine
is a commonly used non-sedating antihistamine now available without
prescription. The study looking for side effects in children lasted for two
weeks and was probably done in the summer, because few of the children got
upper respiratory infections or noted wheezing. But the incidence of these
two problems was doubled in the group given the drug.
While two weeks is enough time to evaluate the side effects for the
drug, such as dry nose or sedation, it is hardly long enough to see the side
effects of taking the drug for its intended purpose. We have now had an
uncontrolled thirty year trial of what happens when we block a normal
defense and we ought to pay attention.
Our ancestors dealt with a similar problem up until about three hundred
years ago. When we get cut or injured so that bacteria can get under our
skin our immune system recognizes a problem and signals that more blood is
needed to deal with it. This causes the cardinal signs of inflammation:
redness, swelling, pain, and fever. A person who went to the doctor with
these symptoms would usually be bled; their arm would be cut and allowed to
bleed into a bowl until the signs went away. It was a very effective therapy
for the symptoms because loss of blood leads to shock that is potentially
much more serious than infection ? shock trumps the immune system. The
symptoms would rapidly disappear. But more people treated this way died of
infection. And more people whose immune system is blocked by antihistamines
and decongestants die today from infections that could have been washed away
or from asthma that is triggered by pollutants that could have been
similarly removed.
We need to ask the question posed by evolutionary medicine more often:
Why did this symptom develop and is it defensive; does it help us deal with
environmental insults? If the answer is "Yes" then we ought to honor rather
than block those symptoms.
Return to HOME.
Read about more examples where we block our normal body functions of fever
and diarrhea.
Go on to read about helping clean the nose.
____________________________________________________________________________________________
References:
Am J Rhinol 1998 Jan-Feb;12(1):37-43
Nasal mucosal endorgan hyperresponsiveness.
Svensson C, Andersson M, Greiff L, Persson CG
Department of Otorhinolaryngology, Head & Neck Surgery, University
Hospital, Lund, Sweden.
Nonspecific hyperresponsiveness of the upper and lower airways is a
well-known characteristic of different inflammatory airway diseases but the
underlying mechanisms have not yet been satisfactorily explained. In
attempts to elucidate the relation of hyperresponsiveness to disease
pathophysiology we have particularly examined the possibility that different
airway endorgans may alter their function in allergic airway disease. The
nose, in contrast to the bronchi, is an accessible part of the airways where
in vivo studies of airway mucosal processes can be carried out in humans
under controlled conditions. Different endorgans can be defined in the
airway mucosa: subepithelial microvessels, epithelium, glands, and sensory
nerves. Techniques may be applied further in the nose to determine
selectively the responses/function of these endorgans. Topical challenge
with methacholine will induce a glandular secretory response, and topical
capsaicin activates sensory c-fibers and induces nasal smart. Topical
histamine induces extravasation of plasma from the subepithelial
microvessels. The plasma exudate first floods the lamina propria and then
moves up between epithelial cells into the airway lumen. This occurs without
any changes in the ultrastructure or barrier function of the epithelium. We
have therefore forwarded the view of mucosal exudation of bulk plasma as a
physiological airway tissue response with primarily a defense function.
Since the exudation is specific to inflammation, we have also suggested
mucosal exudation as a major inflammatory response among airway endorgan
functions. Using a "nasal pool" device for concomitant provocation with
histamine and lavage of the nasal mucosa we have assessed exudative
responses by analyzing the levels of plasma proteins (e.g., albumin alpha
2-macroglobulin) in the returned lavage fluids. A secretory
hyperresponsiveness occurs in both experimental and seasonal allergic
rhinitis. This type of nasal hyperreactivity may develop already 30 minutes
after allergen challenge. It is attenuated by topical steroids and oral
antihistamines. We have demonstrated that exudative hyperresponsiveness
develops in both seasonal allergic rhinitis and common cold, indicating
significant changes of this important microvascular response in these
diseases. An attractive hypothesis to explain airway hyperresponsiveness has
been increased mucosal absorption permeability due to epithelial damage,
possibly secondary to the release of eosinophil products. However, neither
nonspecific nor specific endorgan hyperresponsiveness in allergic airways
may be explained by epithelial fragility or damage since nasal absorption
permeability (measured with 51CR-EDTA and dDAVP) was decreased or unchanged
in our studies of allergic and virus-induced rhinitis, respectively. Thus,
the absorption barrier of the airway mucosa may become functionally tighter
in chronic eosinophilic inflammation.
Publication Types:
* Review
* Review, tutorial
PMID: 9513658
American Academy of Allergy, Asthma and Immunology.
The Allergy Report. Vol. 1, page 4.
_________________________________________________________________
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