At 11:07 AM 4/29/2005 -0400, you wrote:
>
>Ode Coyote wrote:
>
>>   Even then..from following links on Rosemarys 'old' site, links now
>> missing...the rate of argyria in silver workers is 1 in 2000. ie: they
>> tried to find some cases to study, looked in the most likely place to find
>> some and had a really hard time.
>
>Could be genetic, or only a small number of the workers got exposed to uv
light
>shortly after inhaling (tanning beds?).
##  Inhaled silver dust takes something like 30 days to be excreted, if the
particles are small.
 If they are big, I suppose many would be coughed up.
 I would expect the dust to be a mix.
 I would expect a miner to see sunlight at least once a month.
>> There was one researcher that expressed extreme frustration in trying to
>> induce arygria in animals so he could study it.
>
>Wonder what he did?  Maybe he did not realize that he needed to expose
them to
>uv light to trigger the initial photoreduction, or they had fur that blocked
>light.
##  He injected the animals with mega doses of silver citrate and other
compounds at near lethal levels. Part of the study was about seeing if any
tumors developed.  Small benign tumors developed at some of the injection
sites.
 To NOT know the role of UV light would be completely idiotic. At least
this guy had enough intelligence to know and admit he was having trouble
which generally leads to finding out why.  I don't remember what he found
out.  That 'might' be where the selenium connection came from.
 I vaguely remember that link going to an EPA site and a collection of
studies with "Colloidal Silver" on the cover page...with absolutely no
mention of "colloidal silver" in any of the studies.

Looking for link.... 
 Humm, it's still an active link but the info has been watered down into a
Readers Digest form over the past 4 or 5 years.
 It used to be a whole bunch of pages with every detail included.

http://www.epa.gov/iris/subst/0099.htm#I.A.

Ode
>
>Marshall
>
>>
>> [the same report?]
>> Ode
>>
>> At 02:11 PM 4/28/2005 -0400, you wrote:
>> >
>> >I think I may have finally figured out how one can get argyria when
>> >breathing in silver dust, and how silver dust ends up being eliminated
>> >by the body via the kidneys despite the large particle size.
>> >
>> >I have puzzled how silver particles, too large to be absorbed, end up
>> >causing argyria or being eliminated by the kidneys. for people like
>> >silver mine workers.
>> >
>> >There are two possibilities:
>> >
>> >1. If the cells of the lungs are deprived of oxygen causing hypoxia
>> >(Hypoxia mean low in oxygen), then the Mitochondrial release of H2O2 is
>> >extracellular.
>> >See
>> >http://www3.interscience.wiley.com/cgi-bin/abstract/109905841/ABSTRACT
>> >
>> >2. The silver acts as an oxidizing catalyst causing hyperoxia of the
>> >cells (hyperoxia means too much oxygen), which interestingly enough also
>> >causes cells to produce H2O2.
>>
>http://www.clinicalobgyn.com/pt/re/clnobgyn/abstract.00003081-200309000-000
>>
17.htm;jsessionid=CxnsZEjxsFPeZcdMg2S2T1QlRcqHtJp11yVi25MHcYrMRxHePXHQ!21124
>> 22791!-949856031!9001!-1
>> >
>> >Hyperoxia increases H2O2 release by lung mitochondria and microsomes
>> >
>> >My hypothesis is that a silver particle covers one or more cells. These
>> >cells are cut off from oxygen (or over oxygenated) and begin expelling
>> >H2O2.  The hydrogen peroxide then reacts with the silver producing
>> >silver oxide which is immediately absorbed into the blood and becomes
>> >silver chloride.  Since the particles of silver breathed in are too
>> >large to get into the blood stream directly, there is nothing for the
>> >silver chloride to plate out on, until it is either expelled by the
>> >kidneys, or exposed to light in the skin and photoreduced. Once that
>> >happens more can plate out on those particles causing argyria.
>> >
>> >Marshall
>> >
>> >
>> >
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