Matthew said, If the 0.1 M solution is transparent and colorless, then a 0.1 millimolar solution (about 10 ppm Ag+) will surely be transparent and colorless too, don't you agree?
Yes, I do. My point was that, because of silver nitrates distinct staining propensity, it seems to lend itself to argyria much more readily. Would it do that if it did not have this pronounced staining characteristic? To attempt to determine that, we would need to see if other, colorless silver preparations that do not have staining characteristics also cause argyria. Have you read anywhere what the ppm of the silver nitrate was that caused argyria? What would have been the ppm of the silver nitrate that we believe Rosemary Jacobs used (based on the practice of medical doctors of the day at that time)? However, in my mind, the usual warnings from various agencies of silver nitrates tendency to stain the skin make me wonder how big a part that staining mechanism plays in its causation of argyria. Also, I did get a hold of Hill and Pillsbury's summary (chapter IX, pp 128-131.) They report: "Generalized argyria has been reported to have been produced by EVERY compound of silver in common clinical use at the present (1939) time, with the exception of silver oxide." (capitalization mine.) The various reports I have read from the turn of the century about argyria (excluding the silver-smith type) were mostly deliberately induced by the administration of very large amounts of very high ppm silver compounds to rats, dogs, etc. The above statement from Hill & Pillsbury is not concerning argyria contracted by humans. Yes, every silver compound administered in high dosages with high ppm levels (1,000+ ppm) produced argyria in lab animals, and would have no doubt done so in humans. In their summary, Hill and Pillsbury also state about silver: "Transportation in the body occurs principally via the blood stream and to some extent via the lymphatics. A majority of investigators believe that it is transported in the form of chloride or albuminate." Silver albuminate could be formed by a reaction of blood serum (not blood cells) with silver ions in solution. This last statement would seem to make any type of silver ingestion dangerous, since we are all ingesting silver ions in solution. Im sorry, but it is a meaningless statement. To state that something could be formed merely means that it is hypothetically possible. In fact, the experience of many users disputes it. We must be sure we are not talking apples and oranges here. My interest is not in whether it is possible to cause argyria in specific, artificial conditions, using specialized pharmaceutical preparations, but whether it is likely, or even possible to cause it within the parameters of the folks who are making home-made CS. It is my firm stance that if a person makes CS with pure, Fine silver, using any type of water, and does not brew it to a coffee color, argyria is a non-factor. Keep it clear to yellow and it doesnt matter how much you drink. I am concerned that our continuing discussion will become a contention, which I want to avoid. Is it possible to contract argyria from a silver preparation? Yes. Is it possible to get it from a clear one? Yes (if its silver nitrate). Is it possible to accidentally do it at home making it with low voltage (below 120 volts) and Fine silver wire/rods/bars? Not if you keep it clear or yellow. (IMHO) Terry Chamberlin ______________________________________________________________________ Post your free ad now! http://personals.yahoo.ca -- The Silver List is a moderated forum for discussing Colloidal Silver. Instructions for unsubscribing are posted at: http://silverlist.org To post, address your message to: silver-list@eskimo.com Silver List archive: http://escribe.com/health/thesilverlist/index.html Address Off-Topic messages to: silver-off-topic-l...@eskimo.com OT Archive: http://escribe.com/health/silverofftopiclist/index.html List maintainer: Mike Devour <mdev...@eskimo.com>