[TruthTalk] "Mad Cow Disease"

[Marlin Halverson]

Dear Loved Ones: "Mad Cow Disease" is in the news again.  I believe that Mark Purdey has discovered the answers surrounding this mysterious plague.  Please visit the links to his site below and read what he has to say.  If you love truth, I believe you will appreciate the work of this hero. Love, Marlin http://www.markpurdey.com/articles.htm http://www.markpurdey.com/index.html http://www.markpurdey.com/science_the_origins_of_bse.htm "Chernobyl and BSE ? But what about the more aggressive, new strain of TSE – the massive epidemic of BSE/vCJD that suddenly blighted the UK’s young cattle and human population post November 1986? Did this big time epidemic result from an overexposure to the artificial sources of the eco-prerequisites that we are talking about here? In this respect, it seems likely that the UK’s BSE and vCJD epidemics were caused by the simultaneous exposure of cattle, cats, humans and zoo animals to a toxic combination of factors - the widely used copper-chelating organo dithiophosphate (OP) insecticides (35) and the fall out of radioactive metals from the Chernobyl eruption (20). During the 1980s British cattle herds and humans were exposed to exclusively high doses of these insecticides for warble fly and head lice control respectively (42), whereupon the prion proteins of the OP treated animals were starved of their copper co partners (35). This rendered the prion protein vulnerable to binding up with certain rogue replacement metals, such as the radioactive strontium 90, which were rained down at high concentrations onto the soils of NW Europe after the Chernobyl accident (20) – the precise region that later became the world’s most intensive hotspot of BSE." -------- "The so called “infectivity” of the prion is a misnomer and should be correctly defined as the magnetic/ “reactive” free radical generating capacity of the Mn3+ component of the prion; which remains active at all temperatures below the 550 degree “curie point”.  TSE can be likened to a solar charged battery on continuous charge; where the Mn contaminated/Cu depleted circadian pathways absorb and pile up, rather than conducting the vital life force energies of incoming ultra violet, acoustic and geomagnetic radiation. Instead of harnessing these energies for the body’s own bio-rhythmic requirements, an infrasonic shock induced metamorphosis of the Mn atom intervenes ; initiating an explosive free radical mediated pathogenesis that perverts the healthy pathways of sound and light; Cu prions are replaced by ‘hyperpolarized’ Mn prions that seed self perpetuating ‘cluster bombs’ of radical mediated neurodegeneration. TSE ensues." ----- "An impartial study of the epidemiology of BSE/vCJD suggests that the conventional consensus on the origins of BSE/vCJD is severely flawed (4)(5)(6) for the following reasons; 1. BSE has failed to surface in the cattle populations of the Middle East, India, Africa, North America, Third World countries, etc, despite these countries receiving substantial tonnages of the BSE incriminated meat and bone meal (MBM) imported from the UK from the 1960s onwards (7). UK MBM was exported either in its straight feed form or as an inclusion in cattle concentrate feeds (7). 2. 40,000 + cases of BSE have erupted in UK cows that were born after the 1988 ban on MBM entering cattle feed, and more recently, 22 cases of BSE have erupted in cows born after the 1996 ban on MBM going into animal feeding stuffs designated for all types of domestic animal (8). Furthermore, some BSE endemic countries have experienced a greater total number of BSE cases in cattle born after their respective MBM bans than in cows born before. 3. There have been no reported cases of BSE in TSE susceptible species, such as  sheep and goats (8), despite the customary inclusion of an MBM protein source in  their concentrated feeds. 4. Four of the original five Kudu antelopes which contracted BSE at the London Zoo  had no possible access to feeds containing MBM (9). 5. BSE erupted in four cattle that were raised on MAFF’s  former Liscombe  experimental farm on Exmoor – a beef suckler farm which was designed to raise  beef from an all grass/silage system without any resort to purchased in concentrate  feeds (personal communication; M. Stanbury, formerly ADAS, Quantock House, Taunton, UK.) 6. Alterations in the rendering of MBM (cessation of solvent extraction, lower temperatures) from the batch to the continuous flow system had purportedly  enabled the survival of scrapie agent in UK MBM, thus initiating the outbreak of BSE (10). However several scrapie endemic countries, such as the USA and Scandinavia, had also adopted these same BSE-causing prerequisites into their  rendering systems (11), yet these countries remain BSE-free to date (8).  7. Several US trials failed to invoke BSE in cattle after feeding or injecting massive doses of scrapie-contaminated brain homogenate (12)(13). 8. The UK’s mechanically recovered/processed beef products and baby foods –  blamed for causing vCJD in humans – were exported worldwide to countries where  the practise of “skull splitting” in small rural vCJD has never erupted. Likewise, butchers were offered as an explanation for the growing number of vCJD clusters in  rural areas. But this practise had been adopted universally by rural/urban butchers across the UK.   It is proposed that the origins of TSEs do not stem from the exposure of PrP susceptible individuals to infectious, exogenous prions, but are caused by simultaneous exposure of the PrP susceptible individual to a specific combination of environmental prerequisites; high Mn, low Cu, and high intensities of visible/non visible electromagnetic radiation (EMR)  – a package of factors that is “common” to pockets around the world where TSE clusters have traditionally emerged, whilst remaining absent in adjoining TSE-free regions (4)(5). Combined exposure of PrP susceptible genotypes to these environmental prerequisites  invokes a ‘Jekyll and Hyde’ de novo transformation of the normal native Cu PrPc into its rogue, protease resistant, pathogenic Mn3+ PrPtse isoform (5). See Diagram 3 ........."