To save you the trouble the most relevant language from the below cited
article is 

 

 

"While scientists don't yet know exactly how epigenetic regulation affects
memory, the theory is that certain triggers, such as exercise, visual
stimulation, or drugs, unwind DNA, allowing expression of genes involved in
neural plasticity. That increase in gene expression might trigger
development of new neural connections and, in turn, strengthen the neural
circuits that underlie memory formation. "Maybe our brains are using these
epigenetic mechanisms to allow us to learn and remember things, or to
provide sufficient plasticity to allow us to learn and adapt," says John
Satterlee
<http://www.nida.nih.gov/about/organization/genetics/contacts/index.html> ,
program director of epigenetics at the National Institute on Drug Abuse, in
Bethesda, MD. 

"We have solid evidence that HDAC inhibitors massively promote growth of
dendrites and increase synaptogenesis [the creation of connections between
neurons]," says Tsai. The process may boost memory or allow mice to regain
access to lost memories by rewiring or repairing damaged neural circuits.
"We believe the memory trace is still there, but the animal cannot retrieve
it due to damage to neural circuits," she adds. "

 

-----Original Message-----
From: Ed Porter [mailto:[EMAIL PROTECTED] 
Sent: Thursday, December 11, 2008 10:28 AM
To: 'agi@v2.listbox.com'
Subject: FW: [agi] Lamarck Lives!(?)

 

An article related to how changes in the epigenonme could affect learning
and memory (the subject which started this thread a week ago)

 

 

http://www.technologyreview.com/biomedicine/21801/

 

 




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agi
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