CSRe: concentrated CS and 'aglomeration'

2001-12-20 Thread Kevin Nolan
Hello, Ivan.
 Thanks for your response. It seems that symantics has been the 
main reason for disagreement. We agree then that available silver ion 
concentration is the prime determinant for 'CS' effectiveness, and that water 
contaminated with chloride or sulphide ion acts to reduce silver ion via 
precipitation. I would welcome any feedback on the mattter of chelation, 
though. For many mineral supplements, the claim is made that, eg magnesium or 
potassium is delivered far more effectively within the body as say orotate or 
aspartate than as say, a simple salt like chloride. Could this also apply to CS 
for instance?

On the matter of ions being neutralized by polar water molecules I must 
disagree. Certainly the potential energy of 'naked' ions is reduced via 
solvation, ie the process is spontaneous. The oriented dipole moments of the 
cluster of water molecules surrounding each ion 'blunts' the ion's electric 
intensity, but that in no way effects the overall charge balance. The only way 
to neutralize charge of one sign is the presence of an equal quantity of charge 
of the opposite sign. Hence ions in solution must always exist as components of 
a salt, acid, or base. (By application of a high electrostatic potential, a 
tiny amount of excess ions is possible, but hardly practical or useful!) An 
analogy between conservation of charge and Newton's law of action and reaction 
might help. Imagine poking someone with a sharp stick (read 'bare' ion) versus 
with a cushion (read solvated ion), equal force (read charge) being applied in 
each case. Owing to the greater pressure, the stick will feel like a lot more 
force is being applied, but in fact the person must react back with equal force 
in both instances, in order not to fall over. It's the old pull yourself up by 
your bootstraps thing.

regards, Kevin Nolan ken...@optusnet.com.au

Original:

Kevin, I was trying to show that it is not the salt but the silver load 
thatmay cause argyria or other problems. Most salts are absorbed as dissolved 
ions, the cations taking different pathways to the anions. If a salt was to be 
absorbed as a compound, then the fact that this is so would suggest that the 
salt does not disassociate readily, and therefore would be less reactive than 
the component ions. The difference between compounds and CS is the 
concentration at with they are supplied. It matters only that free silver ions 
are available to react with body tissue, and the number of ions that do so.

If several different salt solutions, containing the same concentrations of free 
silver ions are tested against microbes, I
would expect the results to be similar, disregarding the effect the anion may 
have. In aquatic environments, waters which have high sulphide or chloride 
conditions, high silver concentrations have little effect on susceptible marine 
creatures as there is little in the way of free silver ions, however in places 
that have a high concentration of dissolved silver pollution, aquatic creatures 
can be severely effected. 

I am always wary when someone says that this physical event cannot
occur ;-)
Surely the electrostatic effect of ions in solution is rendered neutral by the 
solvation by the polar water molecules, creating large crystal like structures 
which are essentially neutral at their boundary with the bulk solution. I do 
not think this has been well investigated, mainly due to the difficultly of 
forming and capturing ions of a single species.
Your thoughts good sir?Ivan.


RE: CSRe: concentrated CS and 'aglomeration'

2001-12-19 Thread I Anderson
Kevin,

I was trying to show that it is not the salt but the silver load that
may cause argyria or other problems. Most salts are absorbed as
dissolved ions, the cations taking different pathways to the anions.

If a salt was to be absorbed as a compound, then the fact that this is
so would suggest that the salt does not disassociate readily, and
therefore would be less reactive than the component ions. The
difference between compounds and CS is the concentration at with they
are supplied. It matters only that free silver ions are available to
react with body tissue, and the number of ions that do so.

If several different salt solutions, containing the same
concentrations of free silver ions are tested against microbes, I
would expect the results to be similar, disregarding the effect the
anion may have.

In aquatic environments, waters which have high sulphide or chloride
conditions, high silver concentrations have little effect on
susceptible marine creatures as there is little in the way of free
silver ions, however in places that have a high concentration of
dissolved silver pollution, aquatic creatures can be severely
effected.

I am always wary when someone says that this physical event cannot
occur ;-)
Surely the electrostatic effect of ions in solution is rendered
neutral by the solvation by the polar water molecules, creating large
crystal like structures which are essentially neutral at their
boundary with the bulk solution. I do not think this has been well
investigated, mainly due to the difficultly of forming and capturing
ions of a single species.

Your thoughts good sir?

Ivan.

-Original Message-
From: Kevin Nolan [mailto:ken...@optusnet.com.au]
Sent: Tuesday, 18 December 2001 2:15 p.m.
To: silver-list@eskimo.com
Subject: CSRe: concentrated CS and 'aglomeration'


In digest #1195, Ivan anderson wrote, in two separate postings:

If a silver salt or protein were to enter the body as a compound,
then it is much less likely that it will be deposited in the tissue,
simply because the compound must have a high affinity for the silver
and would be far less reactive. This is why silver salts and proteins
have less germicidal effect in the test tube than silver ions.
However, because these compounds are often supplied at very high
concentrations, the available silver ions (while small as a percentage
of total dose) may in fact be large enough to cause some deposition in
tissue.

The term 'salt' should really only be applied to the solid
compound. When a salt is dissolved it ceases to be a compound and is
then a aqueous mixture of the two separate and distinct ions from
which it was formed. Silver hydroxide, when dissolved, is simply
silver ions in slightly more alkaline water. The same applies to
silver nitrate, which completely dissolves at very high
concentrations. The silver is available as silver ions, as is the
nitrate. Unfortunately, the nitrate is corrosive to tissue It is
most likely not the species that the silver ions are derived from that
can cause problems, but rather the concentration in which they are
present.

Leaving aside organic silver compounds and mild silver proteins, there
seems to be a contradiction between the first and second quotes above,
in relation to salts vs ions. We agree that whether as a silver salt
such as silver nitrate, or (as Marshall Dudley would point out) a base
such as silver hydroxide, when dissolved in water such ionic compounds
dissociate fairly well completely to give just a mixture of ions. How
then can it be said that dissolved silver salts are much less
effective than silver ions? Are they not in effect one and the same? A
while back I pointed out it is impossible to have just one ionic
species in solution - charge neutrality requires that ionic silver (or
any other cation) must always be accompanied by anions of some kind,
whether hydroxyl, nitrate, acetate or whatever.

This brings the matter back full circle. Despite all the words so far,
the following basic issue has not been answered:
Suppose we have different ionic silver compounds each made up to the
same PPM dissolved silver ion in solution. Examples might be 10PPM
silver each as silver hydroxide, silver acetate, silver citrate,
silver bicarbonate. For none of these is the anion either poisonous or
germicidal. Should we expect the germicidal power to be essentially
equivalent, whether in the test tube, or in the body? If not, on what
basis?

regards, Kevin Nolan ken...@optusnet.com.au


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CSRe: concentrated CS and 'aglomeration'

2001-12-17 Thread Kevin Nolan
In digest #1195, Ivan anderson wrote, in two separate postings:

If a silver salt or protein were to enter the body as a compound, then it 
is much less likely that it will be deposited in the tissue, simply because the 
compound must have a high affinity for the silver and would be far less 
reactive. This is why silver salts and proteins have less germicidal effect in 
the test tube than silver ions. However, because these compounds are often 
supplied at very high concentrations, the available silver ions (while small as 
a percentage of total dose) may in fact be large enough to cause some 
deposition in tissue.

The term 'salt' should really only be applied to the solid compound. When 
a salt is dissolved it ceases to be a compound and is then a aqueous mixture of 
the two separate and distinct ions from which it was formed. Silver hydroxide, 
when dissolved, is simply silver ions in slightly more alkaline water. The same 
applies to silver nitrate, which completely dissolves at very high 
concentrations. The silver is available as silver ions, as is the nitrate. 
Unfortunately, the nitrate is corrosive to tissue It is most likely not the 
species that the silver ions are derived from that can cause problems, but 
rather the concentration in which they are present.

Leaving aside organic silver compounds and mild silver proteins, there seems to 
be a contradiction between the first and second quotes above, in relation to 
salts vs ions. We agree that whether as a silver salt such as silver nitrate, 
or (as Marshall Dudley would point out) a base such as silver hydroxide, when 
dissolved in water such ionic compounds dissociate fairly well completely to 
give just a mixture of ions. How then can it be said that dissolved silver 
salts are much less effective than silver ions? Are they not in effect one and 
the same? A while back I pointed out it is impossible to have just one ionic 
species in solution - charge neutrality requires that ionic silver (or any 
other cation) must always be accompanied by anions of some kind, whether 
hydroxyl, nitrate, acetate or whatever.

This brings the matter back full circle. Despite all the words so far, the 
following basic issue has not been answered:
Suppose we have different ionic silver compounds each made up to the same PPM 
dissolved silver ion in solution. Examples might be 10PPM silver each as silver 
hydroxide, silver acetate, silver citrate, silver bicarbonate. For none of 
these is the anion either poisonous or germicidal. Should we expect the 
germicidal power to be essentially equivalent, whether in the test tube, or in 
the body? If not, on what basis?

regards, Kevin Nolan ken...@optusnet.com.au



Re: CSRe: concentrated CS and 'aglomeration'

2001-12-16 Thread Judith Thamm
Sufficient to burn the skin off!  It was supposed to burn the proud flesh
off - but only sting for about 15 minutes...  So it had to be a good
strength.  The son I had then is the one that has just produced my latest
grand daughter... so it happened a few years ago - 33 I think..
Judith.


 One has to look at the strengths.  Just like H2O2, 3% is harmless, but 35%
will
 burn your skin off.  What concentration did the doctor use.  I wonder what
 concentration is used in the eyes.

 Marshall

 Judith Thamm wrote:

  You know, in 1968 when my older son was born, the dr couldn't wait so he
  sliced me through so the birth canal was wider and the baby came more
  quickly.  Where I was stitched up, I developed proudflesh.  When I went
back
  to the dr he put silver nitrate on it.  For 24 hours that stuff burned -
I
  didn't sleep at all until the pain stopped - I couldn't sit down and
even
  lying down was little help.  It was a nightmare - and they are going to
put
  that in a baby's eyes!!!  How horrifying!
 
  Judith.
  - Original Message -
  From: Marshall Dudley mdud...@execonn.com
  To: silver-list@eskimo.com
  Sent: Friday, December 14, 2001 3:34 AM
  Subject: Re: CSRe: concentrated CS and 'aglomeration'
 
   It appears that silver nitrate is being used again on babies:
  
   http://www.parenthoodweb.com/articles/phw1380.htm
   http://www.sangfroid.com/babypix/plan.html
  
http://www.lamaze.com/baby/firsthour/articles/0,9474,167862_80951,00.html
  
   From what I recall silver nitrate was discontinued in the 70's due to
   pharmacutical companies pressing for the use of patented antibiotics
which
   would earn them more money.
  
   Sometime (I believe in the late 80's in Knoxville), a hospital was
sued
  because
   a baby went blind because the antibiotic was not broad enough spectrum
to
  kill
   something it was exposed to in the birth canal.  At the trial they
  presented
   evidence that silver notrate would have killed it and that the only
reason
  they
   stopped using silver nitrate was to increase profits by using a
patented
   antibiotic. I remember thinking at the time it serves them right, even
  though I
   knew nothing of CS at the time.
  
   I did not realize they had started using it again until a little while
ago
  when
   I was researching the history.  Since they now offer both, my
suspicion is
  that
   they are doing this to cover themselves against another law suit, but
tell
  the
   parents that the silver nitrate will burn the eyes to get them to
use
  the
   other as much as possible.
  
   Marshall
  
   Ode Coyote wrote:
  
  Reid
 My chemistry is also pretty limited. I think you have me mixed up
with
someone elses postings.
 From what I gather, silver nitrate is dangerous because very small
  amounts
contain a lot of silver ...and... silver nitrates are corrosive to
body
tissues and leave black stains.
 Even still, until fairly recently, it was used in virtually all new
  born
babies eyes to prevent infections.
 Ken
   
Reid Harvey wrote:

 Ken,

 Also, back to my limited chemistry, I would like to know why it
is
  that
 one would have to isolate one element in a simple compound as the
 culprit in a health problem.  For example with silver nitrate why
  does
 the problem have to be either the silver or the nitrate.  I think
the
 problem is probably the compound.  Another example:  it's been
stated
 here that gold chloride is highly toxic, yet gold metal is
beneficial
 and chlorine is a lot less toxic.

You have a good point here, although gold chloride is not a good
  example
since chlorine is deadly toxic.A good example would be hydrogen
  cyanide,
HCN, which is deadly toxic, yet hydrogen, carbon and nitrogen alone
are
totally harmless.

Marshall



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RE: CSRe: concentrated CS and 'aglomeration'

2001-12-15 Thread I Anderson
Reid,

The term 'salt' should really only be applied to the solid compound.
When a salt is dissolved it ceases to be a compound and is then a
aqueous mixture of the two separate and distinct ions from which it
was formed. Silver hydroxide, when dissolved, is simply silver ions in
slightly more alkaline water.

The same applies to silver nitrate, which completely dissolves at very
high concentrations. The silver is available as silver ions, as is the
nitrate. Unfortunately, the nitrate is corrosive to tissue.

Gold chloride dissolves as gold 3+ ions and chloride ions. The
chloride is non toxic but the gold ions will oxidise body tissue and
enzymes and can cause problems. Metallic gold on the other hand, has
all its electrons and is quite non-reactive.

It is most likely not the species that the silver ions are derived
from that can cause problems, but rather the concentration in which
they are present.

Ivan.

 -Original Message-
 From: Reid Harvey [mailto:ceram...@bol-online.com]
 Sent: Thursday, 13 December 2001 3:00 p.m.
 To: silver list
 Subject: CSRe: concentrated CS and 'aglomeration'


 Ken,
 I never stopped to think that the silver may be the
 hydroxide, evedince
 that my understanding of chemistry is limited. (Sometimes I stop and
 think and sometimes I just stop.)  Can you fill me in on how
 it is that
 the hydroxide is a salt? Does anybody else have an answer? I feel
sure
 that you're correct that the reason this is not harmful to us
 CS lovers
 in the long run is that 10ppm is extremely dilute.  Of course
 this begs
 a question about assertions here that it's not a problem to
 ingest ionic
 silver at the really high concentrations.  If this sort of CS is
 practically unattainable without the salts then responsible people
 should talk about some kind of an upper ppm limit for
 ingesting.  On the
 other hand finding that number may be difficult.

 Also, back to my limited chemistry, I would like to know why
 it is that
 one would have to isolate one element in a simple compound as the
 culprit in a health problem.  For example with silver nitrate why
does
 the problem have to be either the silver or the nitrate.  I think
the
 problem is probably the compound.  Another example:  it's been
stated
 here that gold chloride is highly toxic, yet gold metal is
beneficial
 and chlorine is a lot less toxic.
 Salaam,
 Reid


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RE: CSRe: concentrated CS and 'aglomeration'

2001-12-15 Thread I Anderson
Jason,

The body reacts to metals electro-chemically. The fate of these metals
depends upon the physical properties they exhibit. Silver will bind to
albumin in the blood plasma, selenides, sulphides, ascorbates,
metaloproteins and so on. Some will inevitably bind to these groups
which may be found in tissue and organs. If the concentration of
silver is high, and the concentration of selenium (for example) is
low, then more silver is likely to bind with tissue. When the bodies
supply of metaloproteins become low, because of their use in binding
with silver (or some other metal) then more are manufactured, if large
amounts are required this may trigger an immune response, a rise in
the white blood cell count, and an increase in body temp.

If a silver salt or protein were to enter the body as a compound, then
it is much less likely that it will be deposited in the tissue, simply
because the compound must have a high affinity for the silver and
would be far less reactive. This is why silver salts and proteins have
less germicidal effect in the test tube than silver ions. However,
because these compounds are often supplied at very high
concentrations, the available silver ions (while small as a percentage
of total dose) may in fact be large enough to cause some deposition in
tissue.

Don't be fooled into thinking that solid food is treated any
differently to liquid food. Food enters the body as ions and
molecules, and water enters as water.

Even when silver was normally supplied as highly concentrated salts or
proteins, only a very small percentage of its users developed argyria.

Ivan.

 -Original Message-
 From: Jason / AVRA [mailto:silverd...@hotmail.com]
 Sent: Thursday, 13 December 2001 3:44 p.m.
 To: silver-list@eskimo.com
 Subject: Re: CSRe: concentrated CS and 'aglomeration'




 From: Kevin Nolan ken...@optusnet.com.au
 But is argyria solely due to the high silver dosage possible
 with silver
 nitrate (very high solubility), or is there some kind of
 synergy effect as
 well?
 regards, Kevin Nolan

 Kevin:

 The slate blue colour of argyria is not entirely due as one
 might suspect,
 to the deposition of metallic silver (Petering, 1976), but
 largely to an
 increased deposition of melanin.

 Apparently, the issue of Argyria ( the two types ), is not as
 simple as it
 may first appear.

 Silver, in any form, when it reaches a certain theoretical
 concentration
 in the body is a toxic substance.  Like most toxic
 substances, the effects
 will only last while the substance is active in the body.
 The question is:
 When and why does silver accumulate in the body, and when and
 why does it
 not?

 The body knows, and responds, to a toxic substance when
 detected in the
 body.  This is a toxic response...  It happens every time one
 eats garlic,
 for instance.  The body moves to remove the substance as quickly as
 possible.  This is a system-wide, metabolic response of the
 body.  This
 response is markedly different than the normal elimination
 responses in the
 body.

 First, I believe that with the colloidal silver that most
 use, the body does
 NOT detect the silver as toxic.  I don't believe the
 concentration is great
 enough, even at say a liter per day.  I believe that isolated
silver,
 whether in ion form or in colloidal particle form, is not
 deposited nor
 accumulated in organs or skin tissue, whether a toxic response is
 experienced or not.

 I believe that some, if not many, silver salts and proteins
 DO deposit
 silver in the tissue, because the body does not handle these
 substances in
 the same manner as it does water.  This is the key concept.
  What is the
 body trying to do with these salts and proteins?

 I can induce a toxic reaction to isolated silver through
 heavy lung use.  If
 the silver accumulated in the organs, then the symptoms would
 continue as
 documented in many materia medicas.  However, the symptoms
 are temporary,
 corresponding very closely with the conclusions drawn in
 Roger Altman's
 informal study on silver elimination.

 However, I believe that even a teaspoon of silver
 protein/salts can easily
 cause a toxic reaction in the body.  I believe this based upon the
 documented cases of Argyria from the old school medical
 documents and the
 EPA listed studies.  When a toxic reaction occurs to these
 substances,
 silver begins to accumulate in tissues.  Obviously, this
 doesn't ( or should
 say 'didn't ) occur in every case, or there would be
 thousands of more
 documented cases of Argyria back then.  Evidently, Argyria (
 to the point
 where it was noticed and recorded ) only occured in a small
 percentage of
 the people using silver-based products.  These people may
 have accumulated
 silver more quickly through vitamin E and Selenium
 deficiencies.  There may
 be other factors involved as well.

 The whole idea here:  One wants the silver product to be
 treated exactly
 like water, not like a food substance or a solid.  That is
 exactly what you
 get

RE: CSRe: concentrated CS and 'aglomeration'

2001-12-15 Thread Jason / AVRA

Ivan:

Granted, I am certainly not yet an expert on how substances are handled by 
the body, biologically speaking.  However, the belief that water is handled 
in the same manner as foods in the body is not a universally accepted 
concept.  In Your Body's Many Cries for Water, Fereydoon Batmanghelidj 
M.D. expounds upon his general premise that free water ( as he calls it ) 
is not handled by the body in the same manner as water included in food or 
other liquids.


Here is a very interesting quote from an interview:

Biser: That brings up a good point. When we talked to Mr. Clevenger, he said 
that he got a lot more energy from drinking water. Was he just imagining 
this or does this really happen?


Batmanghelidj: It has to happen, because water is one of the main sources of 
energy for the brain and the entire body.


Biser: What? A source of energy? How could that be? I thought water was just 
an inert substance.


Batmanghelidj: This is the tragic mistake made by modern medicine. In their 
minds, what is important to the solids in the body - the proteins, the 
minerals, the vitamins. To them, water is incidental. It is merely a solvent 
- a packing material and a means of transport. But this view is incorrect.


Biser: So what role does water play in body chemistry?

Batmanghelidj: Water is a source of energy. One of the ways water produces 
energy is through hydrolysis. Hydrolysis means the splitting of water into 
its two components: hydrogen and oxygen. Whenever this occurs, energy is 
released. The energy generated by water helps produce ATP, a compound that 
stores body energy. Just as there is solar energy, there is hydro-electric 
energy.


I think certain parts of the brain draw most of their energy from water.

Biser: How important a source of energy is hydrolysis?

Batmanghelidj: Certain parts of the brain draw most of their energy from 
water. Neurotransmission is heavily dependent on energy from water.


Water affects nerve transmission in one other way to. There seem to exist 
small waterways or micro-streams along the length of nerves that float the 
packaged materials along biological structures called microtubules. These 
waterways transport the products manufactured in the brain to their 
destinations in the nerve endings.


Biser: It's pretty amazing. I had no idea.

Batmanghelidj: Most people don't. Especially doctors.

What occurs along the the nerves is an exchange of charged minerals, called 
cations. Cation exchange gets its energy from water.


Batmanghelidj: Dehydration is a primary factor in the producing of disease 
all over the body.


Dehydration is one of the greatest producers of free radicals in the body. 
Water is the greatest free radical remover. Free radicals cut amino acids 
from the proteins. They do damage to the cell architecture. The mechanism of 
water's role in preventing free radicals is extremely complicated to 
explain. It involves white cell metabolism in the bloodstream. The body 
mechanisms that are supposed to prevent the generation of free radicals get 
destroyed by dehydration.


Biser: So people who eat good foods and vitamins and who avoid water are in 
for a big surprise if they think they have avoided pre mature aging?


Batmanghelidj: That's right! People are overemphasizing the solids and 
ignoring the role of water in energy generation and body metabolism.


Biser: Another problem is that the healthful people drink fruit juices 
instead of water.


Batmanghelidj: They don't realize that everything gets turned into glucose. 
Excess sugar has an effect on the brain. And too much insulin will dry out 
the system. It will take water out of the extra cellular environment, and as 
a result, more food will be eaten - because insulin produces artificial 
hunger.


When we eat excessively as a result of insulin, that which cannot be used 
will be converted to fat.


Readers should not concentrate on fruit juices instead of water.


Biser: One man wrote in after our initial interview claiming that - no pun 
intended - you were all wet about hydrolysis. He said the process does'n't 
release energy, it requires it. What is your response?


Batmanghelidj: The man is not acquainted with new scientific findings. I am 
not talking about hydrolysis in a test tube. I am talking about hydrolysis 
(the splitting of water) as it occurs in living systems.


*

I think chemistry, especially organic chemistry, is wrought with 
misunderstandings ( such as the recent post clarifying that when substances 
are in H2O, and the solution is further diluted, the molecules become closer 
- this is only common sense based on the super-principle that like attracts 
like and opposites polarize ) because the science has lost the perspective 
of viewing systems as a whole.


When you drink one glass of quality drinking water, there is an instant 
resonance with the whole body, as a living, energetic system.


I can't fault either your logic or 

Re: CSRe: concentrated CS and 'aglomeration'

2001-12-15 Thread Marshall Dudley
One has to look at the strengths.  Just like H2O2, 3% is harmless, but 35% will
burn your skin off.  What concentration did the doctor use.  I wonder what
concentration is used in the eyes.

Marshall

Judith Thamm wrote:

 You know, in 1968 when my older son was born, the dr couldn't wait so he
 sliced me through so the birth canal was wider and the baby came more
 quickly.  Where I was stitched up, I developed proudflesh.  When I went back
 to the dr he put silver nitrate on it.  For 24 hours that stuff burned - I
 didn't sleep at all until the pain stopped - I couldn't sit down and even
 lying down was little help.  It was a nightmare - and they are going to put
 that in a baby's eyes!!!  How horrifying!

 Judith.
 - Original Message -
 From: Marshall Dudley mdud...@execonn.com
 To: silver-list@eskimo.com
 Sent: Friday, December 14, 2001 3:34 AM
 Subject: Re: CSRe: concentrated CS and 'aglomeration'

  It appears that silver nitrate is being used again on babies:
 
  http://www.parenthoodweb.com/articles/phw1380.htm
  http://www.sangfroid.com/babypix/plan.html
  http://www.lamaze.com/baby/firsthour/articles/0,9474,167862_80951,00.html
 
  From what I recall silver nitrate was discontinued in the 70's due to
  pharmacutical companies pressing for the use of patented antibiotics which
  would earn them more money.
 
  Sometime (I believe in the late 80's in Knoxville), a hospital was sued
 because
  a baby went blind because the antibiotic was not broad enough spectrum to
 kill
  something it was exposed to in the birth canal.  At the trial they
 presented
  evidence that silver notrate would have killed it and that the only reason
 they
  stopped using silver nitrate was to increase profits by using a patented
  antibiotic. I remember thinking at the time it serves them right, even
 though I
  knew nothing of CS at the time.
 
  I did not realize they had started using it again until a little while ago
 when
  I was researching the history.  Since they now offer both, my suspicion is
 that
  they are doing this to cover themselves against another law suit, but tell
 the
  parents that the silver nitrate will burn the eyes to get them to use
 the
  other as much as possible.
 
  Marshall
 
  Ode Coyote wrote:
 
 Reid
My chemistry is also pretty limited. I think you have me mixed up with
   someone elses postings.
From what I gather, silver nitrate is dangerous because very small
 amounts
   contain a lot of silver ...and... silver nitrates are corrosive to body
   tissues and leave black stains.
Even still, until fairly recently, it was used in virtually all new
 born
   babies eyes to prevent infections.
Ken
  
   Reid Harvey wrote:
   
Ken,
   
Also, back to my limited chemistry, I would like to know why it is
 that
one would have to isolate one element in a simple compound as the
culprit in a health problem.  For example with silver nitrate why
 does
the problem have to be either the silver or the nitrate.  I think the
problem is probably the compound.  Another example:  it's been stated
here that gold chloride is highly toxic, yet gold metal is beneficial
and chlorine is a lot less toxic.
   
   You have a good point here, although gold chloride is not a good
 example
   since chlorine is deadly toxic.A good example would be hydrogen
 cyanide,
   HCN, which is deadly toxic, yet hydrogen, carbon and nitrogen alone are
   totally harmless.
   
   Marshall
   
   
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 silver.
   
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Re: CSRe: concentrated CS and 'aglomeration'

2001-12-15 Thread Judith Thamm
He was supposed to be somewhere else at 7.30 am and it was already 7.15 am.
proudflesh is where an infection occurs in the scar tissue.  So I would not
have had the problems unless he had not been in a hurry.  He was later
kicked out of the practice he was in.  He told our next door neighbour to
'take two aspirins and lie down' - and she was dead of a heart attack when
her husband got home from work that day.
Judith.

- Original Message -
From: Josephine joseph...@poidogs.com
To: silver-list@eskimo.com
Sent: Sunday, December 16, 2001 2:40 AM
Subject: Re: CSRe: concentrated CS and 'aglomeration'


 I think the horrifying part was also the impatient doctor.
 What was his rush?
 Did he have a hot date that night?
 Excuse me, but I am already assuming it is a HE because it sounds like a
HE.
 I wonder if you would have had to endure the burning pain had the doctor
 allowed you
 to deliver without the slicing.
 Josephine
 Hawaii



  You know, in 1968 when my older son was born, the dr couldn't wait so he
  sliced me through so the birth canal was wider and the baby came more
  quickly.  Where I was stitched up, I developed proudflesh. 



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Re: CSRe: concentrated CS and 'aglomeration'

2001-12-14 Thread Judith Thamm
You know, in 1968 when my older son was born, the dr couldn't wait so he
sliced me through so the birth canal was wider and the baby came more
quickly.  Where I was stitched up, I developed proudflesh.  When I went back
to the dr he put silver nitrate on it.  For 24 hours that stuff burned - I
didn't sleep at all until the pain stopped - I couldn't sit down and even
lying down was little help.  It was a nightmare - and they are going to put
that in a baby's eyes!!!  How horrifying!

Judith.
- Original Message -
From: Marshall Dudley mdud...@execonn.com
To: silver-list@eskimo.com
Sent: Friday, December 14, 2001 3:34 AM
Subject: Re: CSRe: concentrated CS and 'aglomeration'


 It appears that silver nitrate is being used again on babies:

 http://www.parenthoodweb.com/articles/phw1380.htm
 http://www.sangfroid.com/babypix/plan.html
 http://www.lamaze.com/baby/firsthour/articles/0,9474,167862_80951,00.html

 From what I recall silver nitrate was discontinued in the 70's due to
 pharmacutical companies pressing for the use of patented antibiotics which
 would earn them more money.

 Sometime (I believe in the late 80's in Knoxville), a hospital was sued
because
 a baby went blind because the antibiotic was not broad enough spectrum to
kill
 something it was exposed to in the birth canal.  At the trial they
presented
 evidence that silver notrate would have killed it and that the only reason
they
 stopped using silver nitrate was to increase profits by using a patented
 antibiotic. I remember thinking at the time it serves them right, even
though I
 knew nothing of CS at the time.

 I did not realize they had started using it again until a little while ago
when
 I was researching the history.  Since they now offer both, my suspicion is
that
 they are doing this to cover themselves against another law suit, but tell
the
 parents that the silver nitrate will burn the eyes to get them to use
the
 other as much as possible.

 Marshall

 Ode Coyote wrote:

Reid
   My chemistry is also pretty limited. I think you have me mixed up with
  someone elses postings.
   From what I gather, silver nitrate is dangerous because very small
amounts
  contain a lot of silver ...and... silver nitrates are corrosive to body
  tissues and leave black stains.
   Even still, until fairly recently, it was used in virtually all new
born
  babies eyes to prevent infections.
   Ken
 
  Reid Harvey wrote:
  
   Ken,
  
   Also, back to my limited chemistry, I would like to know why it is
that
   one would have to isolate one element in a simple compound as the
   culprit in a health problem.  For example with silver nitrate why
does
   the problem have to be either the silver or the nitrate.  I think the
   problem is probably the compound.  Another example:  it's been stated
   here that gold chloride is highly toxic, yet gold metal is beneficial
   and chlorine is a lot less toxic.
  
  You have a good point here, although gold chloride is not a good
example
  since chlorine is deadly toxic.A good example would be hydrogen
cyanide,
  HCN, which is deadly toxic, yet hydrogen, carbon and nitrogen alone are
  totally harmless.
  
  Marshall
  
  
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silver.
  
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Re: CSRe: concentrated CS and 'aglomeration'

2001-12-14 Thread Josephine
I think the horrifying part was also the impatient doctor.
What was his rush?
Did he have a hot date that night?
Excuse me, but I am already assuming it is a HE because it sounds like a HE.
I wonder if you would have had to endure the burning pain had the doctor
allowed you
to deliver without the slicing.
Josephine
Hawaii



 You know, in 1968 when my older son was born, the dr couldn't wait so he
 sliced me through so the birth canal was wider and the baby came more
 quickly.  Where I was stitched up, I developed proudflesh. 



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Re: CSRe: concentrated CS and 'aglomeration'

2001-12-13 Thread Kevin Nolan
Jason - between myself and Reid we have been working you pretty hard. Thanks
for your thoughtful feedback. There are specifics not covered (like which
other silver salts are problematic) but I understand your point - stick to
pure CS and you can't go far wrong. Go take a break!

Reid - on your question about where does the hydroxide come from. It's
part-and-parcel of the electrolysis process in producing ionic silver. To a
lesser extent I believe bicarbonate ions, HCO3- are also produced, owing to
dissolved CO2 present in the water. Roger Altman's course covered it all
better than I can - search the archives for more.

regards, Kevin Nolan

- Original Message -
From: Jason / AVRA silverd...@hotmail.com
To: silver-list@eskimo.com
Sent: Thursday, December 13, 2001 1:44 PM
Subject: Re: CSRe: concentrated CS and 'aglomeration'


 From: Kevin Nolan ken...@optusnet.com.au
 But is argyria solely due to the high silver dosage possible with silver
 nitrate (very high solubility), or is there some kind of synergy effect as
 well?
 regards, Kevin Nolan

 Kevin:

 The slate blue colour of argyria is not entirely due as one might
suspect,
 to the deposition of metallic silver (Petering, 1976), but largely to an
 increased deposition of melanin.

 Apparently, the issue of Argyria ( the two types ), is not as simple as it
 may first appear.

 Silver, in any form, when it reaches a certain theoretical concentration
 in the body is a toxic substance.  Like most toxic substances, the effects
 will only last while the substance is active in the body.  The question
is:
 When and why does silver accumulate in the body, and when and why does it
 not?

 The body knows, and responds, to a toxic substance when detected in the
 body.  This is a toxic response...  It happens every time one eats garlic,
 for instance.  The body moves to remove the substance as quickly as
 possible.  This is a system-wide, metabolic response of the body.  This
 response is markedly different than the normal elimination responses in
the
 body.

 First, I believe that with the colloidal silver that most use, the body
does
 NOT detect the silver as toxic.  I don't believe the concentration is
great
 enough, even at say a liter per day.  I believe that isolated silver,
 whether in ion form or in colloidal particle form, is not deposited nor
 accumulated in organs or skin tissue, whether a toxic response is
 experienced or not.

 I believe that some, if not many, silver salts and proteins DO deposit
 silver in the tissue, because the body does not handle these substances in
 the same manner as it does water.  This is the key concept.  What is the
 body trying to do with these salts and proteins?

 I can induce a toxic reaction to isolated silver through heavy lung use.
If
 the silver accumulated in the organs, then the symptoms would continue as
 documented in many materia medicas.  However, the symptoms are temporary,
 corresponding very closely with the conclusions drawn in Roger Altman's
 informal study on silver elimination.

 However, I believe that even a teaspoon of silver protein/salts can easily
 cause a toxic reaction in the body.  I believe this based upon the
 documented cases of Argyria from the old school medical documents and
the
 EPA listed studies.  When a toxic reaction occurs to these substances,
 silver begins to accumulate in tissues.  Obviously, this doesn't ( or
should
 say 'didn't ) occur in every case, or there would be thousands of more
 documented cases of Argyria back then.  Evidently, Argyria ( to the point
 where it was noticed and recorded ) only occured in a small percentage of
 the people using silver-based products.  These people may have accumulated
 silver more quickly through vitamin E and Selenium deficiencies.  There
may
 be other factors involved as well.

 The whole idea here:  One wants the silver product to be treated exactly
 like water, not like a food substance or a solid.  That is exactly what
you
 get with a low PPM, fine particle/ionic isolated silver.  Luckily, there
is
 every indication that silver oxide and silver chloride do not deposit
silver
 into tissues.  Keep in mind:  just because silver comes in CONTACT with
 tissues, does not mean it will be DEPOSITED in tissues.

 It certainly APPEARS as if the protein/salt combination CAUSES silver to
be
 deposited in tissues.

 Undoubtedly, there is a lot we don't know.  We don't know it, probably
 because nobody does!

 Based on the information provided by Ken ( the lung study of metallic
silver
 ), it is becoming clearer that the issue of permanent silver toxicity is
one
 of compounds and not silver itself.  However, it is good to keep in mind
 that it IS the silver that ultimately is responsible for the effects.





 _
 MSN Photos is the easiest way to share and print your photos:
 http://photos.msn.com/support/worldwide.aspx


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Re: CSRe: concentrated CS and 'aglomeration'

2001-12-13 Thread Ode Coyote


  Reid
 My chemistry is also pretty limited. I think you have me mixed up with
someone elses postings.
 From what I gather, silver nitrate is dangerous because very small amounts
contain a lot of silver ...and... silver nitrates are corrosive to body
tissues and leave black stains.
 Even still, until fairly recently, it was used in virtually all new born
babies eyes to prevent infections.
 Ken


Reid Harvey wrote:

 Ken,

 Also, back to my limited chemistry, I would like to know why it is that
 one would have to isolate one element in a simple compound as the
 culprit in a health problem.  For example with silver nitrate why does
 the problem have to be either the silver or the nitrate.  I think the
 problem is probably the compound.  Another example:  it's been stated
 here that gold chloride is highly toxic, yet gold metal is beneficial
 and chlorine is a lot less toxic.

You have a good point here, although gold chloride is not a good example
since chlorine is deadly toxic.A good example would be hydrogen cyanide,
HCN, which is deadly toxic, yet hydrogen, carbon and nitrogen alone are
totally harmless.

Marshall


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Re: CSRe: concentrated CS and 'aglomeration'

2001-12-13 Thread Ode Coyote
  Sure.  And it might explain why all CS is effective no matter how badly
it's made.
 No matter how many huge particles there are, there are also small ones
present.
 The body just sorts them out according to what it can use, sending them to
where they can go.
 What can't get in goes through and what can get in..does. [Working like a
simple membrane filter]

 BTW  I filled a jug with my 300+ PPM tap water.  It also appears darker
than distilled but not as dark as the jug full of clear CS [in indirect
lighting]

 The well water has a weaker TE than the CS...but many times the meter
reading.
It follows that the conductivity of the tap water is due to ionic or
dissolved minerals and ions don't reflect red laser light [too small for
the wavelength?] and the tap water has a lower concentration of suspended
particles than the CS.
 They both have a metallic taste but the CS has a much stronger metallic
flavor.
 Could it be that we don't taste ions very well?
 Could the metallic flavor be from a battery effect on the tongue similar
to tasteing metal when touching a nine volt battery onto the tongue?

 If we had a variable color laser, or even a bright flashlight, could the
dominant particle size be determined by which wavelength gets reflected the
most?
Ken

At 08:33 AM 12/14/01 +0600, you wrote:
Ken,
This is all very interesting, a new twist I haven't seen here before.
So an indication of a majority of particles of small size is to be found
where there is pale yellow with good TE.  If this is a large majority I
would imagine there would be an argument to suggest that this CS is no
less beneficial than the 10ppm CS.  In this scenario the fine particles
would be penetrating where the large particles could not go, but the
large particles would be beneficial at the outer surfaces, which also
need CS.
Reid

Ode Coyote wrote:
  Can you get any color CS without a TE?  I haven't found it yet.
I believe that having a color requires a particle and a particles are
what
makes a TE visible.
 If the TE is weak and the color is ,say, pale yellow... That would
indicate to me that the majority of the particles are fairly largish.
But
getting a pale yellow with a strong TE would indicate that the minority
of
particles are that of the yellow color producing size and the majority
are
of the smaller colorless variety.

 Something i noticed the other day..
A gallon of 25 +/- PPM crystal clear CS that's returned to the milk jug
the
distilled water came out of is significantly darker than an identical
jug
of distilled water.
...some sort of refraction effect i suppose.
Humm, I wonder if my 300PPM tap water does the same?
Ken






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Re: CSRe: concentrated CS and 'aglomeration'

2001-12-13 Thread Marshall Dudley
It appears that silver nitrate is being used again on babies:

http://www.parenthoodweb.com/articles/phw1380.htm
http://www.sangfroid.com/babypix/plan.html
http://www.lamaze.com/baby/firsthour/articles/0,9474,167862_80951,00.html

From what I recall silver nitrate was discontinued in the 70's due to
pharmacutical companies pressing for the use of patented antibiotics which
would earn them more money.

Sometime (I believe in the late 80's in Knoxville), a hospital was sued because
a baby went blind because the antibiotic was not broad enough spectrum to kill
something it was exposed to in the birth canal.  At the trial they presented
evidence that silver notrate would have killed it and that the only reason they
stopped using silver nitrate was to increase profits by using a patented
antibiotic. I remember thinking at the time it serves them right, even though I
knew nothing of CS at the time.

I did not realize they had started using it again until a little while ago when
I was researching the history.  Since they now offer both, my suspicion is that
they are doing this to cover themselves against another law suit, but tell the
parents that the silver nitrate will burn the eyes to get them to use the
other as much as possible.

Marshall

Ode Coyote wrote:

   Reid
  My chemistry is also pretty limited. I think you have me mixed up with
 someone elses postings.
  From what I gather, silver nitrate is dangerous because very small amounts
 contain a lot of silver ...and... silver nitrates are corrosive to body
 tissues and leave black stains.
  Even still, until fairly recently, it was used in virtually all new born
 babies eyes to prevent infections.
  Ken

 Reid Harvey wrote:
 
  Ken,
 
  Also, back to my limited chemistry, I would like to know why it is that
  one would have to isolate one element in a simple compound as the
  culprit in a health problem.  For example with silver nitrate why does
  the problem have to be either the silver or the nitrate.  I think the
  problem is probably the compound.  Another example:  it's been stated
  here that gold chloride is highly toxic, yet gold metal is beneficial
  and chlorine is a lot less toxic.
 
 You have a good point here, although gold chloride is not a good example
 since chlorine is deadly toxic.A good example would be hydrogen cyanide,
 HCN, which is deadly toxic, yet hydrogen, carbon and nitrogen alone are
 totally harmless.
 
 Marshall
 
 
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Re: CSRe: concentrated CS and 'aglomeration'

2001-12-13 Thread Marshall Dudley
The hydroxide radicals are already there in the dyhydrogen monoxide which
composes 99.% of the CS. This is nothing more than H(OH) a hydrogen ion
and an hydroxide radical. If any hydrogen bubbles off, then the hydroxide
radical is freed.

BTW here is an msds on the substance:

http://www.radix.net/~fornax/me/msdsdhmo.html

and a complete web site devoted to it:

http://www.dhmo.org/ which states:

Its basis is the unstable radical Hydroxide, the components of which are
found in a number of caustic, explosive and poisonous compounds such as
Sulfuric Acid, Nitroglycerine and Ethyl Alcohol. 

Of course to most of us, we call it water.

Marshall

Reid Harvey wrote:

 Kevin,
 First off I should say that we produce concentrated CS using de-ionized
 water, of TDS less than 10.  I am unclear as to just where the
 dissociated hydroxide ions are coming from.
 Reid

 Kevin Nolan wrote:
 Silver hydroxide would not be a salt, but rather a base.  IE sodium
 hydroxide is lye. Marshall

 Technically speaking, that is true. But silver hydroxide is a very weak
 base, and in fact it's pH is lower than many
 'genuine' salts. The point is, it exists in solution as dissociated ions
 - not as colloidal silver particles. Owing to claims
 made on the list that nitrate ion was the main problem with silver
 nitrate (even though argyria is a deposited silver
 condition), the question arises as to what species or combination of
 species potentially does the damage, and under
 what conditions. I think there is general agreement that unrealistically
 massive doses are required before home brew
 'CS' would be a problem. But I got the impression that silver nitrate
 could cause trouble at much lower relative dosage -
 ie it's not just a matter of silver ion dosage alone. Nitrate is a known
 carcinogen (less so than nitrite), and an irritant at
 high enough concentration. But is argyria solely due to the high silver
 dosage possible with silver nitrate (very high
 solubility), or is there some kind of synergy effect as well?  Anything
 taken to excess will cause trouble. The gist is
 this - do certain anions, innocuous of themselves (eg, acetate, citrate)
 somehow significantly and selectively 'promote'
 toxicity of silver ion relative to say, silver hydroxide at the same
 concentration? Maybe there is simply no definitive
 data on that, but if there is it would be a good thing to know.

 regards, Kevin Nolan

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Re: CSRe: concentrated CS and 'aglomeration'

2001-12-12 Thread Ode Coyote
  ##  Somehow my name got attached to something I didn't write.
But, hydroxide or not, salt or not...at 10 to 30 PPM, you'd drown long
before you get anything like a toxic dose. At a 94% elimination rate per 48
hrs, you'd drown for years before you'd get argryia. [note below that
ingest per day might need to be changed to retain over all to get that
total load of 3 grams...so 94x300? Or is that 6x 300 litres?]
 Note also that the 94% elimination rate was for an inhaled or ingested by
mouth dose of silver 'dust' eliminated via the feces.

 It has been postulated that the blue bloods got that name because they
ate off silver a lot and turned blue.  It seems they ate their dinnerware
for desert. A teaspoon a day keeps the bloodletter away?

  The requirement is very much higher than could possibly gained by
taking
  normal strength CS, Roger.

  To become blue you would need to ingest about 3 grams of silver per
day, 
  according to Alexander G. Schauss, Ph.D. Director, Life Sciences
  Division John Hopkins University. A litre of 10 ppm CS contains 10
  milligrams of silver, therefore one would need to ingest 300 litres
per
  day to reach this point.

  This is quite a bit higher than some of the references I have seen,
but
  even the most conservative estimate has one drinking tens of litres
per
  day.

  Ivan.


At 08:00 AM 12/13/01 +0600, you wrote:
Ken,
I never stopped to think that the silver may be the hydroxide, evedince
that my understanding of chemistry is limited. (Sometimes I stop and
think and sometimes I just stop.)  Can you fill me in on how it is that
the hydroxide is a salt? Does anybody else have an answer? I feel sure
that you're correct that the reason this is not harmful to us CS lovers
in the long run is that 10ppm is extremely dilute.  Of course this begs
a question about assertions here that it's not a problem to ingest ionic
silver at the really high concentrations.  If this sort of CS is
practically unattainable without the salts then responsible people
should talk about some kind of an upper ppm limit for ingesting.  On the
other hand finding that number may be difficult.

Also, back to my limited chemistry, I would like to know why it is that
one would have to isolate one element in a simple compound as the
culprit in a health problem.  For example with silver nitrate why does
the problem have to be either the silver or the nitrate.  I think the
problem is probably the compound.  Another example:  it's been stated
here that gold chloride is highly toxic, yet gold metal is beneficial
and chlorine is a lot less toxic.
Salaam,
Reid

Ode Coyote wrote:
Jason,
Thanks for the reference material. The conditions seem to be
pretty extreme - not many of us are going to be
using 1500 PPM solution of silver salts of any kind as drinking water.
Nevertheless it raises an interesting matter.
Most of the home made CS has the silver in the form of a silver salt -
namely silver hydroxide, more so than actual
colloid. So is it only the low concentration and/or dosage that prevents
poisoning? It has been stated previously on this
list that in the case of silver nitrate, nitrate is the actual culprit.
But acetate is from vinegar, so it seems safe to
assume that in the case of silver acetate, only the silver could be to
blame. Is the picture more complicated than this -
meaning the stuff exists in the body in part as undissociated complex
that acts differently to either ionic species?

regards, Kevin Nolan




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Re: CSRe: concentrated CS and 'aglomeration'

2001-12-12 Thread Marshall Dudley
Reid Harvey wrote:

 Ken,

 Also, back to my limited chemistry, I would like to know why it is that
 one would have to isolate one element in a simple compound as the
 culprit in a health problem.  For example with silver nitrate why does
 the problem have to be either the silver or the nitrate.  I think the
 problem is probably the compound.  Another example:  it's been stated
 here that gold chloride is highly toxic, yet gold metal is beneficial
 and chlorine is a lot less toxic.

You have a good point here, although gold chloride is not a good example
since chlorine is deadly toxic.A good example would be hydrogen cyanide,
HCN, which is deadly toxic, yet hydrogen, carbon and nitrogen alone are
totally harmless.

Marshall


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CSRe: concentrated CS and 'aglomeration'

2001-12-12 Thread Kevin Nolan
Reid Harvey, Ode Coyote wrote:

 Ken,
 I never stopped to think that the silver may be the hydroxide, evedince
 that my understanding of chemistry is limited. (Sometimes I stop and
 think and sometimes I just stop.)  Can you fill me in on how it is that
 the hydroxide is a salt?

Silver hydroxide would not be a salt, but rather a base.  IE sodium
hydroxide is lye. Marshall

Technically speaking, that is true. But silver hydroxide is a very weak base, 
and in fact it's pH is lower than many 'genuine' salts. The point is, it exists 
in solution as dissociated ions - not as colloidal silver particles. Owing to 
claims made on the list that nitrate ion was the main problem with silver 
nitrate (even though argyria is a deposited silver condition), the question 
arises as to what species or combination of species potentially does the 
damage, and under what conditions. I think there is general agreement that 
unrealistically massive doses are required before home brew 'CS' would be a 
problem. But I got the impression that silver nitrate could cause trouble at 
much lower relative dosage - ie it's not just a matter of silver ion dosage 
alone. Nitrate is a known carcinogen (less so than nitrite), and an irritant at 
high enough concentration. But is argyria solely due to the high silver dosage 
possible with silver nitrate (very high solubility), or is there some kind of 
synergy effect as well?  Anything taken to excess will cause trouble. The gist 
is this - do certain anions, innocuous of themselves (eg, acetate, citrate) 
somehow significantly and selectively 'promote' toxicity of silver ion relative 
to say, silver hydroxide at the same concentration? Maybe there is simply no 
definitive data on that, but if there is it would be a good thing to know.

regards, Kevin Nolan



Re: CSRe: concentrated CS and 'aglomeration'

2001-12-12 Thread Jason / AVRA




From: Kevin Nolan ken...@optusnet.com.au
But is argyria solely due to the high silver dosage possible with silver 
nitrate (very high solubility), or is there some kind of synergy effect as 
well?

regards, Kevin Nolan


Kevin:

The slate blue colour of argyria is not entirely due as one might suspect, 
to the deposition of metallic silver (Petering, 1976), but largely to an 
increased deposition of melanin.


Apparently, the issue of Argyria ( the two types ), is not as simple as it 
may first appear.


Silver, in any form, when it reaches a certain theoretical concentration 
in the body is a toxic substance.  Like most toxic substances, the effects 
will only last while the substance is active in the body.  The question is:  
When and why does silver accumulate in the body, and when and why does it 
not?


The body knows, and responds, to a toxic substance when detected in the 
body.  This is a toxic response...  It happens every time one eats garlic, 
for instance.  The body moves to remove the substance as quickly as 
possible.  This is a system-wide, metabolic response of the body.  This 
response is markedly different than the normal elimination responses in the 
body.


First, I believe that with the colloidal silver that most use, the body does 
NOT detect the silver as toxic.  I don't believe the concentration is great 
enough, even at say a liter per day.  I believe that isolated silver, 
whether in ion form or in colloidal particle form, is not deposited nor 
accumulated in organs or skin tissue, whether a toxic response is 
experienced or not.


I believe that some, if not many, silver salts and proteins DO deposit 
silver in the tissue, because the body does not handle these substances in 
the same manner as it does water.  This is the key concept.  What is the 
body trying to do with these salts and proteins?


I can induce a toxic reaction to isolated silver through heavy lung use.  If 
the silver accumulated in the organs, then the symptoms would continue as 
documented in many materia medicas.  However, the symptoms are temporary, 
corresponding very closely with the conclusions drawn in Roger Altman's 
informal study on silver elimination.


However, I believe that even a teaspoon of silver protein/salts can easily 
cause a toxic reaction in the body.  I believe this based upon the 
documented cases of Argyria from the old school medical documents and the 
EPA listed studies.  When a toxic reaction occurs to these substances, 
silver begins to accumulate in tissues.  Obviously, this doesn't ( or should 
say 'didn't ) occur in every case, or there would be thousands of more 
documented cases of Argyria back then.  Evidently, Argyria ( to the point 
where it was noticed and recorded ) only occured in a small percentage of 
the people using silver-based products.  These people may have accumulated 
silver more quickly through vitamin E and Selenium deficiencies.  There may 
be other factors involved as well.


The whole idea here:  One wants the silver product to be treated exactly 
like water, not like a food substance or a solid.  That is exactly what you 
get with a low PPM, fine particle/ionic isolated silver.  Luckily, there is 
every indication that silver oxide and silver chloride do not deposit silver 
into tissues.  Keep in mind:  just because silver comes in CONTACT with 
tissues, does not mean it will be DEPOSITED in tissues.


It certainly APPEARS as if the protein/salt combination CAUSES silver to be 
deposited in tissues.


Undoubtedly, there is a lot we don't know.  We don't know it, probably 
because nobody does!


Based on the information provided by Ken ( the lung study of metallic silver 
), it is becoming clearer that the issue of permanent silver toxicity is one 
of compounds and not silver itself.  However, it is good to keep in mind 
that it IS the silver that ultimately is responsible for the effects.






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Re: CSRe: concentrated CS and 'aglomeration'

2001-12-12 Thread Kevin Nolan
Not such a good example, either. The problem is the cyano anion: CN-, which
is deadly whether as HCN, NaCN, KCN etc. The only 'synergy' effect I can see
here is the solubility and hence dosage available between one cyanide
compound and another. The cation per se is of little consequence, unless
toxic in it's own right (eg, mercury).

Kevin Nolan

- Original Message -
From: Marshall Dudley mdud...@execonn.com
To: silver-list@eskimo.com
Sent: Thursday, December 13, 2001 2:20 AM
Subject: Re: CSRe: concentrated CS and 'aglomeration'


 Reid Harvey wrote:

  Ken,
 
  Also, back to my limited chemistry, I would like to know why it is that
  one would have to isolate one element in a simple compound as the
  culprit in a health problem.  For example with silver nitrate why does
  the problem have to be either the silver or the nitrate.  I think the
  problem is probably the compound.  Another example:  it's been stated
  here that gold chloride is highly toxic, yet gold metal is beneficial
  and chlorine is a lot less toxic.

 You have a good point here, although gold chloride is not a good example
 since chlorine is deadly toxic.A good example would be hydrogen cyanide,
 HCN, which is deadly toxic, yet hydrogen, carbon and nitrogen alone are
 totally harmless.

 Marshall


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Re: CSRe: concentrated CS and 'aglomeration'

2001-12-12 Thread Robb Allen


Greetings All..
since my last update about my arthritisI have severely relapsedI am 
personally 99% sure that what I am dealing with is none other than leaky gut 
syndrome...I experience alergic-like reactions whenever I eat..the 
main symptom besides the arthritis is watery eyes.this is just about to 
drive me nuts!!!My arthritis pain comes and goesand thus far is 
still avoiding my hands{Thank God}..My worst spot is my 
shoulders.the attacks jump around...from place to place...right now 
it is very very bad in my left shoulder..Feels like it has eate all the 
way through the soft tissues...and is on raw boneI had no choice but 
to take vioxx...nothing else I know of will take the edge off of the 
pain.I'm about to go nuts from all of this stuffif anyone can 
offer any advice.it would be much appreciated..I'm still hell bent 
against letting the medical proffesion do its thing...here's what 
I'm doing right nowI'm on the water cure..have been for a long 
time..since I weigh about 200.give or take.I'm drinking about 
100 ounces.I'm also on MSM6000 mg per day..b-12..6000 mcg per 
day...I just started liquid alo vera.3 ounces per dayand 
also just started PDA.these last two supplements were given by the 
advice of my local health food store guru.I've also stopped 
swallowing CS..I am doing the holding it in my mouth for 4 minute 
thingI'm afraid of further damaging my flora
If I fast for 2 or 3 days..the pain almost tottally 
disappears..but I gotsta eat.other than the hunger...I feel 
great while doing thisperhaps a marathon fast is in order??..I 
don't know if I can stand doing that or not..Many thanks to those of you 
who have offered help in the past.and many thanks in advance for what I 
know will be good advice in the future.Robb



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Re: CSRe: concentrated CS and 'aglomeration'

2001-12-12 Thread Jonathan B. Britten
R.e. bluebloods -- I have heard this anecdote many times.  I have also
heard that the popular glass harmonica,  invented I think by Benjamin
Franklin,  or at least enjoyed by him,  caused big problems because
players got led poisioning by rubbing away at their crystal glasses!   
That is hard to imagine,  but from what I read,  it is true. Some
people advise never to drink from crystalware.This seems pertinent
when considering the possibility of argyria from silverware.  It could
well be true. 

JBB

 

Ode Coyote wrote:
 
   ##  Somehow my name got attached to something I didn't write.
 But, hydroxide or not, salt or not...at 10 to 30 PPM, you'd drown long
 before you get anything like a toxic dose. At a 94% elimination rate per 48
 hrs, you'd drown for years before you'd get argryia. [note below that
 ingest per day might need to be changed to retain over all to get that
 total load of 3 grams...so 94x300? Or is that 6x 300 litres?]
  Note also that the 94% elimination rate was for an inhaled or ingested by
 mouth dose of silver 'dust' eliminated via the feces.
 
  It has been postulated that the blue bloods got that name because they
 ate off silver a lot and turned blue.  It seems they ate their dinnerware
 for desert. A teaspoon a day keeps the bloodletter away?
 
   The requirement is very much higher than could possibly gained by
 taking
   normal strength CS, Roger.
 
   To become blue you would need to ingest about 3 grams of silver per
 day,
   according to Alexander G. Schauss, Ph.D. Director, Life Sciences
   Division John Hopkins University. A litre of 10 ppm CS contains 10
   milligrams of silver, therefore one would need to ingest 300 litres
 per
   day to reach this point.
 
   This is quite a bit higher than some of the references I have seen,
 but
   even the most conservative estimate has one drinking tens of litres
 per
   day.
 
   Ivan.
 
 At 08:00 AM 12/13/01 +0600, you wrote:
 Ken,
 I never stopped to think that the silver may be the hydroxide, evedince
 that my understanding of chemistry is limited. (Sometimes I stop and
 think and sometimes I just stop.)  Can you fill me in on how it is that
 the hydroxide is a salt? Does anybody else have an answer? I feel sure
 that you're correct that the reason this is not harmful to us CS lovers
 in the long run is that 10ppm is extremely dilute.  Of course this begs
 a question about assertions here that it's not a problem to ingest ionic
 silver at the really high concentrations.  If this sort of CS is
 practically unattainable without the salts then responsible people
 should talk about some kind of an upper ppm limit for ingesting.  On the
 other hand finding that number may be difficult.
 
 Also, back to my limited chemistry, I would like to know why it is that
 one would have to isolate one element in a simple compound as the
 culprit in a health problem.  For example with silver nitrate why does
 the problem have to be either the silver or the nitrate.  I think the
 problem is probably the compound.  Another example:  it's been stated
 here that gold chloride is highly toxic, yet gold metal is beneficial
 and chlorine is a lot less toxic.
 Salaam,
 Reid
 
 Ode Coyote wrote:
 Jason,
 Thanks for the reference material. The conditions seem to be
 pretty extreme - not many of us are going to be
 using 1500 PPM solution of silver salts of any kind as drinking water.
 Nevertheless it raises an interesting matter.
 Most of the home made CS has the silver in the form of a silver salt -
 namely silver hydroxide, more so than actual
 colloid. So is it only the low concentration and/or dosage that prevents
 poisoning? It has been stated previously on this
 list that in the case of silver nitrate, nitrate is the actual culprit.
 But acetate is from vinegar, so it seems safe to
 assume that in the case of silver acetate, only the silver could be to
 blame. Is the picture more complicated than this -
 meaning the stuff exists in the body in part as undissociated complex
 that acts differently to either ionic species?
 
 regards, Kevin Nolan
 
 
 
 
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 List maintainer: Mike Devour mdev...@eskimo.com
 
 


CSRe: concentrated CS and 'aglomeration'

2001-12-12 Thread Reid Harvey
Kevin,
First off I should say that we produce concentrated CS using de-ionized
water, of TDS less than 10.  I am unclear as to just where the
dissociated hydroxide ions are coming from.
Reid

Kevin Nolan wrote:
Silver hydroxide would not be a salt, but rather a base.  IE sodium
hydroxide is lye. Marshall

Technically speaking, that is true. But silver hydroxide is a very weak
base, and in fact it's pH is lower than many
'genuine' salts. The point is, it exists in solution as dissociated ions
- not as colloidal silver particles. Owing to claims
made on the list that nitrate ion was the main problem with silver
nitrate (even though argyria is a deposited silver
condition), the question arises as to what species or combination of
species potentially does the damage, and under
what conditions. I think there is general agreement that unrealistically
massive doses are required before home brew
'CS' would be a problem. But I got the impression that silver nitrate
could cause trouble at much lower relative dosage -
ie it's not just a matter of silver ion dosage alone. Nitrate is a known
carcinogen (less so than nitrite), and an irritant at
high enough concentration. But is argyria solely due to the high silver
dosage possible with silver nitrate (very high
solubility), or is there some kind of synergy effect as well?  Anything
taken to excess will cause trouble. The gist is
this - do certain anions, innocuous of themselves (eg, acetate, citrate)
somehow significantly and selectively 'promote'
toxicity of silver ion relative to say, silver hydroxide at the same
concentration? Maybe there is simply no definitive
data on that, but if there is it would be a good thing to know.

regards, Kevin Nolan




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CSRe: concentrated CS and 'aglomeration'

2001-12-12 Thread Reid Harvey
Ken,
This is all very interesting, a new twist I haven't seen here before.
So an indication of a majority of particles of small size is to be found
where there is pale yellow with good TE.  If this is a large majority I
would imagine there would be an argument to suggest that this CS is no
less beneficial than the 10ppm CS.  In this scenario the fine particles
would be penetrating where the large particles could not go, but the
large particles would be beneficial at the outer surfaces, which also
need CS.
Reid

Ode Coyote wrote:
  Can you get any color CS without a TE?  I haven't found it yet.
I believe that having a color requires a particle and a particles are
what
makes a TE visible.
 If the TE is weak and the color is ,say, pale yellow... That would
indicate to me that the majority of the particles are fairly largish.
But
getting a pale yellow with a strong TE would indicate that the minority
of
particles are that of the yellow color producing size and the majority
are
of the smaller colorless variety.

 Something i noticed the other day..
A gallon of 25 +/- PPM crystal clear CS that's returned to the milk jug
the
distilled water came out of is significantly darker than an identical
jug
of distilled water.
...some sort of refraction effect i suppose.
Humm, I wonder if my 300PPM tap water does the same?
Ken






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CSRe: concentrated CS and 'aglomeration'

2001-12-11 Thread Kevin Nolan
Jason,
Thanks for the reference material. The conditions seem to be pretty 
extreme - not many of us are going to be using 1500 PPM solution of silver 
salts of any kind as drinking water. Nevertheless it raises an interesting 
matter. Most of the home made CS has the silver in the form of a silver salt 
- namely silver hydroxide, more so than actual colloid. So is it only the low 
concentration and/or dosage that prevents poisoning? It has been stated 
previously on this list that in the case of silver nitrate, nitrate is the 
actual culprit. But acetate is from vinegar, so it seems safe to assume that in 
the case of silver acetate, only the silver could be to blame. Is the picture 
more complicated than this - meaning the stuff exists in the body in part as 
undissociated complex that acts differently to either ionic species?

regards, Kevin Nolan ken...@optusnet.com.au

--Original Message-

Kevin:

Addition of silver acetate to the diet (130-1000 ppm) or drinking water 
(1500 ppm) of weaning rats fed a vitamin E deficient diet, precipitated a 
rapidly fatal hepatocellular necrosis and muscular dystrophy on day 14 of 
the treatment or subsequently. ( Grasso P. et al, 1967 Exp. Mol. Pathol., 
11,186-199 ). According to the study, the silver acetate ( NOT isolated 
silver! ) depletes the body of Vitamin E and Selenium.  If the body is 
deficient in either ( as in the above reference ) than severe symptoms of 
silver poisoining manifest, including increased silver retention in the 
kidneys and liver.

This was taken from a stock manufacturer's datasheet on silver acetate:



POTENTIAL HEALTH EFFECTS

Inhalation:
Mild irritant and nuisance dust. Coughing, sneezing or possibly 
breathing difficulty upon the inhalation of large amounts.

Ingestion:
Effect will be similar to but normally less severe than with silver 
nitrate. Acute poisoning can cause pain or burning sensation in mouth and
throat, salivation, vomiting, diarrhea, methemoglobinemia, anuria and, 
in extreme cases, shock, coma and death.

Skin Contact:
Causes irritation to skin. Symptoms include redness, itching, and pain.

Eye Contact:
Possibly some chemical or mechanical irritation of the conjunctiva.

Chronic Exposure: Prolonged or repeated inhalation, application or ingestion 
of silver compounds may cause a permanent bluish discoloration of the skin 
and mucous membranes. Aggravation of Pre-existing Conditions: Persons with 
heavy metals accumulation should cease contact with silver compounds.

Label Hazard Warning:
WARNING! HARMFUL IF SWALLOWED OR INHALED. CAUSES IRRITATION TO SKIN, EYES 
AND RESPIRATORY TRACT.



Now this datasheet is for pure silver acetate.  Still, silver acetate is 
silver acetate.

Documented cases of Argyria exist for six months of regular silver acetate 
usage.  In one case, 6.4 grams was the estimated silver accumulation in the 
individual from taking silver acetate lozenges.

I don't have any information on silver citrate!


Re: CSRe: concentrated CS and 'aglomeration'

2001-12-11 Thread Marshall Dudley
Reid Harvey wrote:

 Ken,
 I never stopped to think that the silver may be the hydroxide, evedince
 that my understanding of chemistry is limited. (Sometimes I stop and
 think and sometimes I just stop.)  Can you fill me in on how it is that
 the hydroxide is a salt?

Silver hydroxide would not be a salt, but rather a base.  IE sodium
hydroxide is lye.

Marshall


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CSRe: concentrated CS and 'aglomeration'

2001-12-11 Thread Reid Harvey
Ken,
I never stopped to think that the silver may be the hydroxide, evedince
that my understanding of chemistry is limited. (Sometimes I stop and
think and sometimes I just stop.)  Can you fill me in on how it is that
the hydroxide is a salt? Does anybody else have an answer? I feel sure
that you're correct that the reason this is not harmful to us CS lovers
in the long run is that 10ppm is extremely dilute.  Of course this begs
a question about assertions here that it's not a problem to ingest ionic
silver at the really high concentrations.  If this sort of CS is
practically unattainable without the salts then responsible people
should talk about some kind of an upper ppm limit for ingesting.  On the
other hand finding that number may be difficult.

Also, back to my limited chemistry, I would like to know why it is that
one would have to isolate one element in a simple compound as the
culprit in a health problem.  For example with silver nitrate why does
the problem have to be either the silver or the nitrate.  I think the
problem is probably the compound.  Another example:  it's been stated
here that gold chloride is highly toxic, yet gold metal is beneficial
and chlorine is a lot less toxic.
Salaam,
Reid

Ode Coyote wrote:
Jason,
Thanks for the reference material. The conditions seem to be
pretty extreme - not many of us are going to be
using 1500 PPM solution of silver salts of any kind as drinking water.
Nevertheless it raises an interesting matter.
Most of the home made CS has the silver in the form of a silver salt -
namely silver hydroxide, more so than actual
colloid. So is it only the low concentration and/or dosage that prevents
poisoning? It has been stated previously on this
list that in the case of silver nitrate, nitrate is the actual culprit.
But acetate is from vinegar, so it seems safe to
assume that in the case of silver acetate, only the silver could be to
blame. Is the picture more complicated than this -
meaning the stuff exists in the body in part as undissociated complex
that acts differently to either ionic species?

regards, Kevin Nolan




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CSRe: concentrated CS and 'aglomeration'

2001-12-10 Thread Kevin Nolan
In digest #1177, Jason/AVRA wrote:

When you tell someone a stabilizer is safe, you are risking their health 
and 
well-being on very serious level.  You are giving them the impression that 
silver is not a toxic substance in the human body.  While your own 
understanding may be great, theirs may not.  They just might see something 
like silver acetate, a stabilized silver product ( though of course not a 
protein )and assume it is safe to use..

Jason - would you mind filling in a bit on the danger with silver acetate? What 
about silver citrate?

regards, Kevin Nolan ken...@optusnet.com.au



Re: CSRe: concentrated CS and 'aglomeration'

2001-12-10 Thread Jason / AVRA

Kevin:

Addition of silver acetate to the diet (130-1000 ppm) or drinking water 
(1500 ppm) of weaning rats fed a vitamin E deficient diet, precipitated a 
rapidly fatal hepatocellular necrosis and muscular dystrophy on day 14 of 
the treatment or subsequently. ( Grasso P. et al, 1967 Exp. Mol. Pathol., 
11,186-199 ). According to the study, the silver acetate ( NOT isolated 
silver! ) depletes the body of Vitamin E and Selenium.  If the body is 
deficient in either ( as in the above reference ) than severe symptoms of 
silver poisoining manifest, including increased silver retention in the 
kidneys and liver.


This was taken from a stock manufacturer's datasheet on silver acetate:



POTENTIAL HEALTH EFFECTS

Inhalation:
   Mild irritant and nuisance dust. Coughing, sneezing or possibly 
breathing difficulty upon the inhalation of large amounts.


Ingestion:
   Effect will be similar to but normally less severe than with silver 
nitrate. Acute poisoning can cause pain or burning sensation in mouth and
   throat, salivation, vomiting, diarrhea, methemoglobinemia, anuria and, 
in extreme cases, shock, coma and death.


Skin Contact:
   Causes irritation to skin. Symptoms include redness, itching, and pain.

Eye Contact:
   Possibly some chemical or mechanical irritation of the conjunctiva.

Chronic Exposure: Prolonged or repeated inhalation, application or ingestion 
of silver compounds may cause a permanent bluish discoloration of the skin 
and mucous membranes. Aggravation of Pre-existing Conditions: Persons with 
heavy metals accumulation should cease contact with silver compounds.


Label Hazard Warning:
WARNING! HARMFUL IF SWALLOWED OR INHALED. CAUSES IRRITATION TO SKIN, EYES 
AND RESPIRATORY TRACT.




Now this datasheet is for pure silver acetate.  Still, silver acetate is 
silver acetate.


Documented cases of Argyria exist for six months of regular silver acetate 
usage.  In one case, 6.4 grams was the estimated silver accumulation in the 
individual from taking silver acetate lozenges.


I don't have any information on silver citrate!


From: Kevin Nolan ken...@optusnet.com.au
Reply-To: silver-list@eskimo.com
To: silver-list@eskimo.com
Subject: CSRe: concentrated CS and 'aglomeration'
Date: Tue, 11 Dec 2001 01:03:37 +1100

In digest #1177, Jason/AVRA wrote:

When you tell someone a stabilizer is safe, you are risking their 
health and

well-being on very serious level.  You are giving them the impression that
silver is not a toxic substance in the human body.  While your own
understanding may be great, theirs may not.  They just might see something
like silver acetate, a stabilized silver product ( though of course not a
protein )and assume it is safe to use..

Jason - would you mind filling in a bit on the danger with silver acetate? 
What about silver citrate?


regards, Kevin Nolan ken...@optusnet.com.au




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Re: CSRe: concentrated CS and 'aglomeration'

2001-12-10 Thread FHLew
Greetings all silverlist members,

Jason/AVRA wrote:

  According to the study, the silver acetate ( NOT isolated silver! )
depletes the body of Vitamin E and Selenium.  If the body is deficient in
either ( as in the above reference ) than severe symptoms of silver
poisoining manifest, including increased silver retention in the kidneys and
liver.

The rationale of advocating wholesome raw food is based on the
preservation of the complete complement of the enzyme systems and whole
nutrients present in the food to drive the Kreb Cycle in maintaining
homeostatic
metabolism. Denatured food leads to robbing Peter to
pay Paul in the body in order to complete the Kreb Cycle , resulting in
drainage and defiencies of concomitant components [ Zinc, Copper, Magnesium,
Selenium,
Vitamins,Amino-acids etc ]. Ill-health subsequently results. This applies to
all patho-physiological processes in the human system.

  In radonics, I  advise colloidal  mineral supplementation
to maintain at least a minimal basal
 supportive level for those on radonic treatment. There has been reported
instances of deaths when the radonic beam was suddenly switched off. In
radonics, distance is not the criterion but the resonance of vibrational
signatures.The latter may be from homeopathics, flower essences, nutrients,
trace minerals, sounds and colours. Each element has its specific frequency
and this is its vibrational signature. These are beamed at the subject from
afar.The reported fatalities may be attributed to the sudden interruption in
the biological transmutational processes [ Professor Louis C. Kevran ] under
cold nuclear fusion [  Professor Panos Pappas ] due to low supportive
mineral reserve in the body and other geomagnetic factors. Minerals in the
form of cell-salts regulate the vitality and viability of tissue
cells,electromagnetically - The  Autonomous Nervous System , The
Cardio-Vascular System, namely . Any interference to electromagnetic
transmission in these vital systems may  lead to their dysfunction and
eventual death. These systems are regulated by the geomagnetic  rhythmicity
of 7.83 Hz conforming to Schumann's Resonance. There is still the mootable
cloud over the silver ion. What determines  the proverbial properties of
colloidal silver: in the biologically transmutated form, after absorption in
the body or in the form generated from my Robey Deluxe Mark II generator ?
The topic on Biological Transmutation by Prof  Louis C Kevran was posted in
the recent past in the Silverlist.

 I practise Isopathy, Homeopathy, Biochemic
Therapy [ 12 tissue salts ], Orthostatic Nutrition and
Naturotherapy  in the  morning cup of mid-stream urine I partake daily with
skin scrubbing using my own urine. I recycle my mineral reserves,
phytonutrients, immuno-endocrine secretions [ Melantonin, DHEA ] from my own
cistern. Those with uro-genital tract infection, please abstain fron
urinetherapy,until such time when the infection is under control. The
psychological block to this mode of treatment is inevitable. There is a lot
of information on this
topic in cyberspace. I recycle what my body has given back to me,
transmutated but good for the next cycle,with
minimal energy expenditure under cold nuclear fusion.
Scientists have narrowed down to minerals from outer space which provide
vestigial signatures to substantiate that life on Earth may indeed
originated from outerspace. The linking evidence is found in  bacteria
containing  minerals,on earth. Why silver is bactericidal to some microbes
and not to others ? And why Argyria
manifests in some and not in others? I share Prof
Louis C Kevran 's view  in accepting mainstream Chemistry for what it is and
that there is more to it in life that does not conform to exact sciences.

With regards
 Lew






- Original Message -
From: Jason / AVRA silverd...@hotmail.com
To: silver-list@eskimo.com
Sent: Tuesday, December 11, 2001 5:22 AM
Subject: Re: CSRe: concentrated CS and 'aglomeration'


 Kevin:

 Addition of silver acetate to the diet (130-1000 ppm) or drinking water
 (1500 ppm) of weaning rats fed a vitamin E deficient diet, precipitated a
 rapidly fatal hepatocellular necrosis and muscular dystrophy on day 14 of
 the treatment or subsequently. ( Grasso P. et al, 1967 Exp. Mol. Pathol.,
 11,186-199 ). According to the study, the silver acetate ( NOT isolated
 silver! ) depletes the body of Vitamin E and Selenium.  If the body is
 deficient in either ( as in the above reference ) than severe symptoms of
 silver poisoining manifest, including increased silver retention in the
 kidneys and liver.

 This was taken from a stock manufacturer's datasheet on silver acetate:

 

 POTENTIAL HEALTH EFFECTS

 Inhalation:
 Mild irritant and nuisance dust. Coughing, sneezing or possibly
 breathing difficulty upon the inhalation of large amounts.

 Ingestion:
 Effect will be similar to but normally less severe than